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in the presence of Pax6(S), beta(3) is translocated from the cytoplasm to the nucleus;full-length Ca(v)beta may act directly as a transcription regulator independent of its role in regulating Ca(2+) channel activity
During development Cavbeta3 protein expression in the cerebellum peaks at postnatal days 15 and 21, and has diminished drastically by day 60, and in the hippocampus increases with age throughout all subfields.
In transgenic mice lacking the calcium channel beta3 subunit, there are compensatory changes of calcium channel abundance leading to adaptation of alternate calcium transport mechanisms.
Differential gene expression analysis identifies murine Cacnb3 as strongly upregulated in distinct dendritic cell populations upon stimulation
these results indicate the importance of the beta3 subunit in determining calcium channel characteristics and catecholamine release in adrenal chromaffin cells.
neither Ca(V)beta3 nor Ca(V)beta4 are indispensable for hair cell Ca(2+) currents but contribute to the overall current properties
Mice with cardiac-specific sequestration of the beta-subunit of the L-type calcium channel
Data show that the calcium channel beta3 subunit negatively modulates inositol triphosphate-induced calcium release.
Down-regulation of Cacnb3 expression does not rescue placental hyperplasia in a model of interspecies hybrid placentas, indicaating that upregulation is a downstream event.
the involvement of voltage-dependent Ca2+ channels and importance of the beta3 subunit in olfactory signal transduction
Ca(V)beta(3) has only subtle effects on L-type Ca(2+) currents in ileum smooth muscle.
This study showed the importance of voltage-dependent calcium channels and the key role of the beta3 subunit in memory formation, nociceptive sensory transduction, and various neurological signal transduction pathways.
These results suggest a possibility that, unrelated to voltage-dependent Ca(2+) channels regulation, Ca(v)beta3 negatively regulates the NMDAR activity in the hippocampus.
Modified sympathetic nerve system activity with overexpression of the voltage-dependent calcium channel beta3 subunit.(
Deficiency in beta3 regulatory subunit of voltage-gated calcium channels resulted in a profound survival defect of CD8(+) T cells.
This gene encodes a regulatory beta subunit of the voltage-dependent calcium channel. Beta subunits are composed of five domains, which contribute to the regulation of surface expression and gating of calcium channels and may also play a role in the regulation of transcription factors and calcium transport.
voltage-dependent calcium channel beta subunit
, calcium channel, voltage-dependent, beta 3 subunit
, voltage-dependent calcium channel beta 3b subunit
, voltage-dependent L-type calcium channel subunit beta-3
, voltage-dependent L-type calcium channel subunit beta-3-like
, calcium channel voltage-dependent subunit beta 3
, calcium channel beta 3 subunit
, Calcium channel subunit beta 3
, voltage-dependent calcium channel beta 3a