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Four SNPs in IFNGR2 (zeige IFNGR2 Proteine), IL12RB1 (zeige IL12RB1 Proteine), IL12RB2 (zeige IL12RB2 Proteine), and IL23R were found to be associated with the MAP infection status of the resource population.
Data indicate that the interleukin-23 receptor (IL-23R) SNPs rs11209026, p.Arg381Gln; rs41313262 p.Val362Ile were not associated with susceptibility to inflammatory bowel disease (IBD) in Chinese Han population.
Interleukin-23 receptor cytokine-binding homology region balances the ratio of Th17/Th9/Treg cells in collagen-induced arthritis.
our study provides the evidence that functional IL-23R rs1884444 G/T and IL-17F (zeige IL17F Proteine) rs763780 A/G polymorphisms may be a new genetic susceptibility factor to SLE, especially in the Polish population.
IL23R rs10889677 and IL17A (zeige IL17A Proteine) rs2275913 were not associated with the susceptibility to Necrotizing enterocolitis (NEC). In conclusion, data suggest that a variant of IL17F (zeige IL17F Proteine) (rs763780) may contribute to the development of NEC.
this meta-analysis suggests that each allele of IL-23R, including rs7519847, rs17375018 and rs11209032 was negatively associated with uveitis; however, homozygote models, including the rs17375018 GG genotype and rs11209032 AA genotype, were significantly associated with uveitis
an evaluation of what is currently known about the protective role of R381Q variant in IL-23R gene in immune-based diseases (Review).
Th17 cells expressed consistent high levels of the IL-12Rbeta1 subunit, which appeared a better predictor of responsiveness to IL-23 (zeige IL23A Proteine) than the expression of the IL-23R subunit.
We conclude that variants in IL-23A (zeige IL23A Proteine) and IL-23R genes were associated with the risk of multiple sclerosis or other inflammatory demyelinating diseases.
IL-23 (zeige IL23A Proteine) R (rs7517847) and LEP (zeige LEP Proteine) (rs7799039) polymorphisms were associated with an increased risk but not affecting the clinical presentation of HCC (zeige FAM126A Proteine) among Egyptian patients
In a Turkish population, IL23R polymorphism is a risk factor for UC and is protective against CD.
RBPJ (zeige RBPJ Proteine) binds and trans-activates the Il23r promoter and induces IL-23R expression and represses anti-inflammatory IL-10 (zeige IL10 Proteine) production in Th17 cells.
Epithelial IL-23R signaling enables protective IL-22 (zeige IL22 Proteine) responses in experimental colitis.
Study highlights the importance of IL-23R expression level and the instability of Foxp3 (zeige FOXP3 Proteine)+ regulatory T cells in the development of inflammatory bowel diseases.
Differential splicing generates antagonistic soluble IL-23R (sIL-23R) variants, which might limit IL-23-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL-23R.
Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 (zeige IL23A Proteine) receptor signaling and IL-17A (zeige IL17A Proteine) production in patients with severe asthma.
Data show the contribution of IL-23/IL-23 (zeige IL23A Proteine) receptor and IL-7/IL-7 (zeige IL7 Proteine) receptor signaling in Th17 and Th1 (zeige HAND1 Proteine) cell dynamics during experimental autoimmune encephalomyelitis (EAE).
Study provides evidence that IL-23 (zeige IL23A Proteine)/IL-23R plays a critical role in brain ischemic injury
IL-23 (zeige IL23A Proteine) plus T-cell receptor signalling results in significant upregulation of IL-23 receptor expressed predominantly on CD4 (zeige CD4 Proteine)(hi)CD8 (zeige CD8A Proteine)(hi)CD3 (zeige CD3E Proteine)(+)alphabetaTCR(+) thymocytes, and leads to RORgammat-dependent apoptosis.
Homeostatic IL-23 receptor signaling limits Th17 response through IL-22 (zeige IL22 Proteine)-mediated containment of commensal microbiota.
Characterization of potent IL-23R small-peptide modulator, 2305 (teeeqqly), that decreases inflammatory response.
The protein encoded by this gene is a subunit of the receptor for IL23A/IL23. This protein pairs with the receptor molecule IL12RB1/IL12Rbeta1, and both are required for IL23A signaling. This protein associates constitutively with Janus kinase 2 (JAK2), and also binds to transcription activator STAT3 in a ligand-dependent manner.
, interleukin 23 receptor
, interleukin-23 receptor-like
, IL-23 receptor
, interleukin 23 receptor-like