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Dog (Canine) Polyclonal MYH9 Primary Antibody für WB - ABIN2783216
Sabbir, Dillon, Mowat: Dlc1 interaction with non-muscle myosin heavy chain II-A (Myh9) and Rac1 activation. in Biology open 2016
Dog (Canine) Polyclonal MYH9 Primary Antibody für ICC, IF - ABIN153126
Klingner, Cherian, Fels, Diesinger, Aufschnaiter, Maghelli, Keil, Beck, Tolić-Nørrelykke, Bathe, Wedlich-Soldner: Isotropic actomyosin dynamics promote organization of the apical cell cortex in epithelial cells. in The Journal of cell biology 2014
demonstrated that though the targeted integration site for exogenous genes is shifted to MYH9 intron 2 (about 500bp downstream exon 2), the high HR efficiency and the endogenous MYH9 gene integrity are not only preserved, but the expected expression of the inserted gene(s) is observed in a pre-designed set of experiments conducted in mouse ES cells
ALPK1 (zeige ALPK1 Antikörper) is a kinase that participates in the regulation of Golgi-derived TNF-alpha (zeige TNF Antikörper) trafficking through myosin IIA phosphorylation in the inflammation of gout.
we present high MYH9 expression as a reliable and powerful prognostic factor for patients with AML (zeige RUNX1 Antikörper).
Mutations of MYH9 cause MYH9-RD, a form of congenital thrombocytopenia characterized by giant platelets and no platelet functional abnormalities.
Heterozygous deletional mutation (c.5803delG) in exon 41 of the MYH9 gene is associated with inherited thrombocytopenia.
cytokinesis failure in megakaryocytes is the consequence of both the absence of NMIIB and a low RhoA activity that impairs NMIIA localization at the cleavage furrow through increased actin turnover.
The loss of junctional NMIIA, upon Coronin 1B (zeige CORO1B Antikörper) knockdown, perturbed RhoA (zeige RHOA Antikörper) signaling.
Results identified nonsynonymous variants in MYH9 and ABCA4 (zeige ABCA4 Antikörper) to be the most frequent risk loci in nonsyndromic orofacial clefts in the Taiwanese population.
Our study is the first to identify genetic predisposition to transplant renal artery stenosis in patients with MYH9 risk allelic variants (rs4821480, rs4821481, rs3752462, rs11089788, rs136211, rs5756168, rs2032487, and rs2239784)
The hearing impairment associated with MYH9 disorder in this family was characterized as adult onset, progressive, and high-frequency dominant. Hematological manifestations of MYH9 disorder show complete penetrance, whereas extra-hematological manifestations show incomplete penetrance and variable expressivity in this family.
Fluorescence resonance energy transfer experiments showed that FliI (zeige FLII Antikörper)-NMMIIA interactions require Ca(2 (zeige CA2 Antikörper)+) influx. We conclude that Ca(2 (zeige CA2 Antikörper)+) influx through the TRPV4 (zeige TRPV4 Antikörper) channel regulates FliI (zeige FLII Antikörper)-NMMIIA interaction, which in turn enables generation of the cell extensions essential for collagen remodeling
Collectively, these results implicate both moesin (zeige MSN Antikörper) and myosin IIA in the regulation of phagolysosome biogenesis and in host defense against infections.
APPL1 (zeige APPL1 Antikörper) enhances glucose uptake by modulating the activation and localization of PKCzeta (zeige PRKCZ Antikörper), as well as its functional interaction with both PP2A (zeige PPP2R2B Antikörper) and myosin IIa.
Data show that deletion of myosin heavy chain II-A (Myh9)/myosin heavy chain II-B (Myh10 (zeige MYH10 Antikörper)) caused severe hydroureter/hydronephrosis at birth.
our data indicated that PGDHC is a Pg-derived, cell-permeable ceramide that possesses a unique property of promoting osteoclastogenesis via interaction with Myh9 which, in turn, activates a Rac1/DC-STAMP (zeige TM7SF4 Antikörper) pathway for upregulation of osteoclast cell fusion.
Conditional deletion of nonmuscle myosin II-A in mouse tongue epithelium results in squamous cell carcinoma
Inhibition of NMMHC IIA impedes TF expression and venous thrombosis via Akt (zeige AKT1 Antikörper)/GSK3beta-NF-kappaB (zeige NFKB1 Antikörper) signalling pathways in the endothelium both in vitro and in vivo.
Myh9 mediates colitis-induced epithelium injury by restricting Lgr5 (zeige LGR5 Antikörper)+ stem cells
Mst1 (zeige MST1 Antikörper) regulates Myosin IIa dynamics to organize high and low affinity LFA-1 (zeige ITGAL Antikörper) to the anterior and posterior membrane during T cell migration.
DDR1 malfunction causes outer hair cells deformation and the separation of the lateral wall, the location of the cellular motor responsible for the electromotile property, explicitly in those regions showing DDR1 and NM-IIA co-localization.
Results demonstrate an important role of NMHC-IIA for the proper formation and function of the glomerulus in zebrafish.
Data show that the locomotion and blebbing of the primordial germ cells (PGCs) required F-actin, myosin II activity and RhoA/Rho-associated protein kinase (ROCK) signaling.
MYH9 physically interacts with the porcine reproductive and respiratory syndrome virus GP5 (zeige GP5 Antikörper) protein via its C-terminal domain and confers susceptibility of cells to virus infection.
This gene encodes a conventional non-muscle myosin\; this protein should not be confused with the unconventional myosin-9a or 9b (MYO9A or MYO9B). The encoded protein is a myosin IIA heavy chain that contains an IQ domain and a myosin head-like domain which is involved in several important functions, including cytokinesis, cell motility and maintenance of cell shape. Defects in this gene have been associated with non-syndromic sensorineural deafness autosomal dominant type 17, Epstein syndrome, Alport syndrome with macrothrombocytopenia, Sebastian syndrome, Fechtner syndrome and macrothrombocytopenia with progressive sensorineural deafness.
cellular myosin heavy chain, type A
, myosin heavy chain 9
, myosin heavy chain, non-muscle IIa
, non-muscle myosin heavy chain A
, non-muscle myosin heavy chain IIa
, non-muscle myosin heavy polypeptide 9
, nonmuscle myosin heavy chain II-A
, myosin IIA
, myosin heavy chain IX
, myosin, heavy chain 9, non-muscle, like-2
, myosin, heavy polypeptide 9, non-muscle
, NMMHC II-a
, heavy polypeptide 9
, myosin heavy chain, nonmuscle
, non-muscle IIa
, type A
, nonmuscle myosin II heavy chain A