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anti-Mouse (Murine) BTG2 Antikörper:
anti-Human BTG2 Antikörper:
anti-Rat (Rattus) BTG2 Antikörper:
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Cow (Bovine) Polyclonal BTG2 Primary Antibody für WB - ABIN2778007
Park, Kim, Baek, Park, Lim, Seo, Chun: B-cell translocation gene 2: expression in the rat ovary and potential association with adenine nucleotide translocase 2 in mitochondria. in Molecular and cellular endocrinology 2013
Dog (Canine) Polyclonal BTG2 Primary Antibody für WB - ABIN2778006
Tsui, Hsieh, Lin, Chang, Juang: Triiodothyronine modulates cell proliferation of human prostatic carcinoma cells by downregulation of the B-cell translocation gene 2. in The Prostate 2008
Human Polyclonal BTG2 Primary Antibody für IHC (p), WB - ABIN654080
Zhang, Huang, Wu, Wang, Wu: Impact of BTG2 expression on proliferation and invasion of gastric cancer cells in vitro. in Molecular biology reports 2010
Show all 2 Pubmed References
Tumor suppressors BTG1 (zeige BTG1 Antikörper) and BTG2 regulate early mouse B-cell development.
Results identify a key molecular mechanism by which the BTG2-PRMT1 (zeige PRMT1 Antikörper) module regulates pre-B cell differentiation and inhibits pre-B cell leukemogenesis.
Results suggest that Btg2 is an anti-adipogenic factor and its expression is controlled by the Stat3 (zeige STAT3 Antikörper) signaling pathway.
these data show that both Btg1 (zeige BTG1 Antikörper) and Btg2 are required for normal vertebral patterning of the axial skeleton, but each gene contributes differently in specifying the identity along the anterior-posterior axis of the skeleton.
Btg2 overexpression in vivo and in vitro induced all the observed changes during interdigit regression, including oxidative stress, arrest of cell cycle progression, transcriptional regulation of senescence markers, and caspase (zeige CASP3 Antikörper)-mediated apoptosis.
study provides a novel molecular mechanism of BTG2-mediated induction of hepcidin (zeige HAMP Antikörper) gene expression, thereby contributing to a better understanding of the hepatic hepcidin (zeige HAMP Antikörper) production involved in iron homeostasis
TIS21 is required for development of spiral ganglion cells
we show that the putative tumor suppressor gene Btg2 is consistently downregulated in high grade gliomas and that its downregulation is necessary for a cell to maintain the malignant phenotype.
Mice lacking the Tis21 3' UTR (zeige UTS2R Antikörper) develop a microcephalic neocortex with fewer neurons.
BTG2 regulates hepatic glucose homeostasis via induction of orphan nuclear receptor (zeige NR1D1 Antikörper) Nur77 (zeige NR4A1 Antikörper) in diabetic mouse model.
we found that miR (zeige MLXIP Antikörper)-27-3p was overexpressed in gastric cancer tissues and cell lines and that BTG2 was a direct and functional target of miR (zeige MLXIP Antikörper)-27a-3p in gastric cancer
Effect of TIS21 on the regulation of p53 (zeige TP53 Antikörper) activity was confirmed by knockdown of TIS21 expression by RNA interference.
TIS21 attenuated Doxorubicin-induced cancer cell senescence by inhibiting linear actin nucleation via Nox4 (zeige NOX4 Antikörper)-ROS (zeige ROS1 Antikörper)-ABI2 (zeige ABI2 Antikörper)-DRF (zeige MPO Antikörper) signal cascade
Data indicate that BTG2, MAP3K11 (zeige MAP3K11 Antikörper), RPS6KA1 (zeige RPS6KA1 Antikörper) and PRDM1 (zeige PRDM1 Antikörper) as putative targets of microRNA miR (zeige MLXIP Antikörper)-125b.
The results indicated that BTG2 expression was downregulated in skin cancer cell lines. Overexpression of BTG2 significantly inhibited cell proliferation, cell cycle progression, and the invasion and migration of skin cancer cells.
Overexpression of BTG2 inhibited the proliferation and migration/invasion of human osteosarcoma cells in vitro
clinical data support conclusions generated from patient-derived xenograft models and indicate that BTG2 expression is a candidate prognostic biomarker for TNBC.
miR (zeige MLXIP Antikörper)-21 was found to be overexpressed and BTG2 was found to to be downregulated in HepG2 liver cancer cells.
BTG2 stimulates mRNA deadenylation via CAF1 (zeige CHAF1B Antikörper) activation through interaction with PABPC1 (zeige PABPC1 Antikörper). Interaction of BTG2 with the first RRM domain of PABPC1 (zeige PABPC1 Antikörper) is required for BTG2 to control cell proliferation.
SETD1A (zeige SETD1A Antikörper) suppresses BTG2 expression through its induction of several BTG2-targeting miRNAs.
The full length sequences of cDNA and genomic DNA of BTG2 gene from the porcine skeletal muscle, was cloned.
The protein encoded by this gene is a member of the BTG/Tob family. This family has structurally related proteins that appear to have antiproliferative properties. This encoded protein is involved in the regulation of the G1/S transition of the cell cycle.
B-cell translocation gene 2, anti-proliferative
, BTG family, member 2
, protein BTG2
, B-cell translocation gene 2
, B cell translocation protein 2
, BTG family member 2
, NGF-inducible protein TIS21
, NGF-inducible anti-proliferative protein PC3
, nerve growth factor-inducible anti-proliferative
, pheochromacytoma cell-3
, B-cell translocation protein 2
, Early induced gene B-cell translocation gene 2
, NGF-inducible anti-proliferative putative secreted protein
, cell surface alloantigen