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KHSV miR (zeige MLXIP ELISA Kits)-K3 activates the GRK2 (zeige ADRBK1 ELISA Kits)/CXCR2/AKT (zeige AKT1 ELISA Kits) axis inducing KSHV-induced angiogenesis and promoting KSHV latency.
CXCR2 mRNA and protein expression levels were significantly decreased in preeclamptic placentas than normal control. The invasive abilities of the two trophoblast cell lines were significantly inhibited when CXCR2 was silenced, but that CXCR2 overexpression promoted trophoblast cells invasion.
CXCR2 promotes breast cancer metastasis and chemoresistance via suppressing AKT1 (zeige AKT1 ELISA Kits) and activating COX2 (zeige COX2 ELISA Kits).
we conclude that CXCR2 is required for the recruitment of TANs, which in turn can suppress antitumor T-cell responses. We showed that CXCR2 ligands, particularly CXCL5 (zeige CXCL5 ELISA Kits), are elevated in both human and mouse PDA.
CXCR4 (zeige CXCR4 ELISA Kits) and CXCR2 were highly expressed in a high invasive gastric cancer cell model and in gastric cancer tissues; crosstalk between CXCR4 (zeige CXCR4 ELISA Kits) and CXCR2 contributed to EMT (zeige ITK ELISA Kits), migration and invasion of gastric cancer.
A unique viral protein, vCXCL1, which targets three chemokine (zeige CCL1 ELISA Kits) receptors: CXCR1 (zeige CXCR1 ELISA Kits) and CXCR2 expressed on neutrophils and CXCR1 (zeige CXCR1 ELISA Kits) and CX3CR1 (zeige CX3CR1 ELISA Kits) expressed on Natural killer cells.
The expressions of CXCL1 (zeige CXCL1 ELISA Kits) in cancer cells and CXCR2 in stromal cells are useful prognostic factors for gastric cancer patients
CXCR2 preferentially supports the maintenance of human pluripotent stem cell characteristics as well as facilitates ectodermal differentiation after the commitment to differentiation, and the mechanism might be associated with mTOR (zeige FRAP1 ELISA Kits), beta-catenin (zeige CTNNB1 ELISA Kits), and hTERT activities.
Our result showed that CXCR2 expression was correlated with high grade (P = 0.024), advanced stage (P = 0.029) and metastasis (P = 0.018). The log-rank test revealed that high CXCR2 and CXCR3 (zeige CXCR3 ELISA Kits) expressions are related to poorer overall survival (P < 0.001; P < 0.001).
These data demonstrate that the CXCR2 network and CXCL4 (zeige PF4 ELISA Kits) play a role in the maintenance of normal HSC (zeige FUT1 ELISA Kits)/HPC cell fates, including survival and self-renewal.
Results also demonstrated that in CXCR2 (zeige CXCR1 ELISA Kits), genotypes BC, CC and FF were probably relevant with mastitis and the genotypes AA, AB and EE may have better milk quality.
Combining CSF1R (zeige CSF1R ELISA Kits) inhibitor with a CXCR2 antagonist blocked granulocyte infiltration of tumors and showed strong anti-tumor effects.
Results demonstrated that complete Freund's adjuvant increased CXCL1 (zeige CXCL1 ELISA Kits) and CXCR2 expression in the dorsal root ganglion, with the cellular distribution in all sizes neurons. In addition, specific inhibition of CXCR2 in the dorsal root ganglion attenuated established inflammatory pain.
postnatal development of the intestinal microbiota is an important susceptibility factor for experimental biliary atresia, which involves Cxcr2 signaling
this study demonstrates CXCR2-driven activation of NLRP3 (zeige NLRP3 ELISA Kits) inflammasome in macrophages, and indicates a potential host-directed therapeutic target to limit the damaging inflammation associated with overt production of proinflammatory IL-1beta (zeige IL1B ELISA Kits)
upregulation of CCRL2 (zeige CCRL2 ELISA Kits) observed under inflammatory conditions is functional to finely tune CXCR2-mediated neutrophil recruitment at sites of inflammation.
RelA (zeige NFkBP65 ELISA Kits) has a role in regulating OIS in preneoplastic lesions; the RelA (zeige NFkBP65 ELISA Kits)/CXCL1 (zeige CXCL1 ELISA Kits)/CXCR2 axis is an essential mechanism of tumor surveillance in pancreatic ductal adenocarcinoma
TNFalpha (zeige TNF ELISA Kits)-activated mesenchymal stromal cells promote breast cancer metastasis by recruiting
the results of this study indicate a new potential use for gastrin-releasing peptide receptor (zeige GRPR ELISA Kits) antagonist for treatment of drug-induced liver injury through a mechanism involving adhesion molecule (zeige NCAM1 ELISA Kits) modulation and possible direct binding to CXCR2
The protein encoded by this gene is a member of the G-protein-coupled receptor family. This protein is a receptor for interleukin 8 (IL8). It binds to IL8 with high affinity, and transduces the signal through a G-protein activated second messenger system. This receptor also binds to chemokine (C-X-C motif) ligand 1 (CXCL1/MGSA), a protein with melanoma growth stimulating activity, and has been shown to be a major component required for serum-dependent melanoma cell growth. This receptor mediates neutrophil migration to sites of inflammation. The angiogenic effects of IL8 in intestinal microvascular endothelial cells are found to be mediated by this receptor. Knockout studies in mice suggested that this receptor controls the positioning of oligodendrocyte precursors in developing spinal cord by arresting their migration. This gene, IL8RA, a gene encoding another high affinity IL8 receptor, as well as IL8RBP, a pseudogene of IL8RB, form a gene cluster in a region mapped to chromosome 2q33-q36. Alternatively spliced variants, encoding the same protein, have been identified.
C-X-C chemokine receptor type 2
, interleukin 8 receptor beta
, interleukin-8 receptor CXCR2
, CXCR2 gene for IL8 receptor type B
, GRO/MGSA receptor
, IL-8 receptor type 2
, IL-8R B
, chemokine (CXC) receptor 2
, high affinity interleukin-8 receptor B
, interleukin 8 receptor B
, interleukin 8 receptor type 2
, interleukin 8 receptor, beta
, interleukin-8 receptor type B
, chemokine receptor CXCR2
, interleukin 8 receptor, alpha
, IL-8 receptor alpha chain
, chemokine (C-X-C) receptor 2
, IL-8 receptor
, High affinity interleukin-8 receptor B
, interleukin-8 receptor, beta