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RETNLB Antikörper

RETNLB Reaktivität: Human ELISA, IHC Wirt: Kaninchen Polyclonal unconjugated
Produktnummer ABIN7235503
  • Target Alle RETNLB Antikörper anzeigen
    RETNLB (Resistin Like beta (RETNLB))
    Reaktivität
    • 49
    • 18
    Human
    Wirt
    • 63
    • 2
    • 1
    • 1
    Kaninchen
    Klonalität
    • 64
    • 3
    Polyklonal
    Konjugat
    • 29
    • 11
    • 5
    • 3
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    Dieser RETNLB Antikörper ist unkonjugiert
    Applikation
    • 51
    • 32
    • 13
    • 13
    • 7
    • 4
    • 4
    • 4
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    ELISA, Immunohistochemistry (IHC)
    Produktmerkmale
    Polyclonal Antibody
    Aufreinigung
    Affinity purification
    Immunogen
    Recombinant protein of human RETNLB
    Isotyp
    IgG
    Top Product
    Discover our top product RETNLB Primärantikörper
  • Applikationshinweise
    IHC 1:50-1:200
    Beschränkungen
    Nur für Forschungszwecke einsetzbar
  • Format
    Liquid
    Konzentration
    0.3 mg/mL
    Buffer
    PBS with 0.05 % sodium azide and 50 % glycerol, PH7.4
    Konservierungsmittel
    Sodium azide
    Vorsichtsmaßnahmen
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Lagerung
    -20 °C
    Informationen zur Lagerung
    Store at -20°C. Avoid freeze / thaw cycles.
  • Target
    RETNLB (Resistin Like beta (RETNLB))
    Andere Bezeichnung
    RETNLB (RETNLB Produkte)
    Synonyme
    FIZZ1 antikoerper, FIZZ2 antikoerper, HXCP2 antikoerper, RELM-beta antikoerper, RELMb antikoerper, RELMbeta antikoerper, XCP2 antikoerper, RETNLB antikoerper, 9030012B21Rik antikoerper, Fizz2 antikoerper, Relmb antikoerper, Xcp3 antikoerper, resistin like beta antikoerper, RETNLB antikoerper, Retnlb antikoerper
    Hintergrund
    Resistin-like betais aproteinthat in humans is encoded by theRETNLBgene. An infusion of either resistin or RELMB in rats rapidly induced severe hepatic but not peripheral insulin resistance. Increases in circulating resistin or RELMB levels markedly stimulated hepatic glucose production despite the presence of fixed physiologic insulin levels. This enhanced rate of glucose output was due to increased flux through glucose-6-phosphatase. The results supported the notion that a novel family of fat- and gut-derived circulating proteins modulates hepatic insulin action.
    UniProt
    Q9BQ08
    Pathways
    Hormone Activity
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