SLC16A2/MCT8 Antikörper

Produktnummer ABIN6569545

Produktdetails anti-SLC16A2/MCT8 Antikörper

Target: SLC16A2/MCT8 (SLC16A2) (Solute Carrier Family 16 Member 2 (SLC16A2))

Reaktivität: Human, Maus, Ratte

Wirt: Kaninchen

Klonalität: Polyklonal

Konjugat: Dieser SLC16A2/MCT8 Antikörper ist unkonjugiert

Applikation: Western Blotting (WB)

Aufreinigung: Affinity purification

Immunogen: Recombinant fusion protein of human SLC16A2 (NP_006508.2).

Isotyp: IgG

Anwendungsinformationen

Applikationshinweise: WB 1:500-1:2000

Beschränkungen: Nur für Forschungszwecke einsetzbar

Handhabung

Konzentration: 1 mg/mL

Buffer: PBS with 0.02 % sodium azide, 50 % glycerol, pH 7.3

Konservierungsmittel: Sodium azide

Vorsichtsmaßnahmen: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Lagerung: -20 °C

Informationen zur Lagerung: Store at -20°C. Avoid freeze / thaw cycles.

Details zu SLC16A2/MCT8

Target: SLC16A2/MCT8 (SLC16A2) (Solute Carrier Family 16 Member 2 (SLC16A2))

Andere Bezeichnung: SLC16A2 (SLC16A2 Produkte)

Synonyme: MCT8 antikoerper, AW105741 antikoerper, Mct8 antikoerper, Xpct antikoerper, AHDS antikoerper, DXS128 antikoerper, DXS128E antikoerper, MCT 7 antikoerper, MCT 8 antikoerper, MCT7 antikoerper, MRX22 antikoerper, XPCT antikoerper, solute carrier family 16 member 2 antikoerper, solute carrier family 16 (monocarboxylic acid transporters), member 2 antikoerper, SLC16A2 antikoerper, Slc16a2 antikoerper

Hintergrund: SLC16A2,AHDS,DXS128,DXS128E,MCT 7,MCT 8,MCT7,MCT8,MRX22,XPCT,This gene encodes an integral membrane protein that functions as a transporter of thyroid hormone. The encoded protein facilitates the cellular importation of thyroxine (T4), triiodothyronine (T3), reverse triiodothyronine (rT3) and diidothyronine (T2). This gene is expressed in many tissues and likely plays an important role in the development of the central nervous system. Loss of function mutations in this gene are associated with psychomotor retardation in males while females exhibit no neurological defects and more moderate thyroid-deficient phenotypes. This gene is subject to X-chromosome inactivation. Mutations in this gene are the cause of Allan-Herndon-Dudley syndrome.

Molekulargewicht:

Observed_MW: 70kDa

Calculated_MW: 59kDa

Gen-ID: 6567

UniProt: P36021

Pathways: Hormone Transport