NOG Antikörper
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- Target Alle NOG Antikörper anzeigen
- NOG (Noggin (NOG))
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Reaktivität
- Human, Maus, Ratte
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Wirt
- Kaninchen
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Klonalität
- Polyklonal
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Konjugat
- Dieser NOG Antikörper ist unkonjugiert
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Applikation
- Western Blotting (WB)
- Spezifität
- Reacts with human Noggin
- Kreuzreaktivität
- Maus
- Kreuzreaktivität (Details)
- Cross reacts with mouse and rat.
- Aufreinigung
- Antigen affinity purified
- Immunogen
- Synthetic peptide comprising an internal sequence of the human Noggin protein. Sequence is 100% conserved in rat and mouse.
- Isotyp
- IgG
- Top Product
- Discover our top product NOG Primärantikörper
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- Applikationshinweise
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Working dilution: Optimal dilution should be determined by the end user.
The following are guidelines only:
- WB :1/1 000 to 1/2 000 - Beschränkungen
- Nur für Forschungszwecke einsetzbar
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- Format
- Liquid
- Buffer
- PBS, Sodium azide 0.02 %
- Konservierungsmittel
- Sodium azide
- Vorsichtsmaßnahmen
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Lagerung
- 4 °C/-20 °C
- Informationen zur Lagerung
- Short term storage at +4°C. For extended periods store in aliquots at -20°C. Antibodies are guaranteed for 6 month from date of receipt.
- Haltbarkeit
- 6 months
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- Target
- NOG (Noggin (NOG))
- Andere Bezeichnung
- Noggin (NOG Produkte)
- Synonyme
- SYM1 antikoerper, SYNS1 antikoerper, nog-A antikoerper, nog1 antikoerper, noggin-1 antikoerper, noggin antikoerper, noggin antikoerper, noggin L homeolog antikoerper, noggin protein antikoerper, NOG antikoerper, Nog antikoerper, nog.L antikoerper, noggin antikoerper
- Hintergrund
- Noggin is involved in numerous developmental processes, such as neural tube fusion and joint formation. The morphogenesis of organs is initiated by a downgrowth from a layer of epithelial stem cells. This process is achieved through the receipt of signals from 1) a WNT protein (WNT3A) to stabilize beta-catenin, and 2) Noggin, which is a bone morphogenetic protein inhibitor. Noggin mutations in unrelated families with proximal symphalangism (SYM1) and multiple synostoses syndrome (SYNS1) have been identified, which have multiple joint fusion as their principal defect.
- Pathways
- Stem Cell Maintenance, Tube Formation
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