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B7H6 belongs to the B7 family (see MIM 605402) and is selectively expressed on tumor cells. Zusätzlich bieten wir Ihnen Natural Killer Cell Cytotoxicity Receptor 3 Ligand 1 Antikörper (80) und Natural Killer Cell Cytotoxicity Receptor 3 Ligand 1 Proteine (17) und viele weitere Produktgruppen zu diesem Protein an.
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B7-H6 was expressed in gliomas and correlated with glioma progression. Knockdown of B7-H6 inhibited glioma cell proliferation, colony formation, migration, and invasion via upregulation of E-cadherin and Bax, and downregulation of vimentin, N-cadherin, MMP-2, MMP-9 and survivin.
these studies find that soluble B7H6 is constitutively expressed during pregnancy
Data suggest that B7-H6 play an important role in the regulation of the biological behavior of glioma cells.
Data indicate a sequence of events driven by tumour-derived prostaglandin D2 (PGD2 (zeige PTGDS ELISA Kits)) associated with engagement of the natural cytotoxicity triggering receptor 3 (NKp30 (zeige NCR3 ELISA Kits))-B7H6 antigen (B7H6) pathway leading to significant group 2 innate lymphoid cells (ILC2s) activation and expansion.
B7H6-derived peptides trigger TNF-alpha (zeige TNF ELISA Kits)-dependent immunostimulatory activity of lymphocytic NK92-MI cells.
Study confirms that B7-H6 is widely expressed in B-cell lymphomas and demonstrates its important role in the pathogenesis and chemosensitivity of lymphoma.
our present data revealed that higher B7-H6 expression in ovarian cancer tissues was positively correlated with tumor metastasis and cancer progression
NKp30 (zeige NCR3 ELISA Kits)-B7-H6 interaction is a novel cell contact mechanism that mediates activation of Group 2 innate lymphoid cells and identifies a potential target for the development of novel therapeutics for atopic dermatitis and other atopic diseases.
study not only reveals the possibility that tumor therapeutics work as stress inducers to enhance tumor sensitivity to NK cell cytolysis but also suggests that B7-H6 could be a potential target for tumor therapy in the future
Our results suggest that NKP30 (zeige NCR3 ELISA Kits)-B7-H6 interaction can aggravate hepatocyte damage, probably through up-regulation of IL-32 (zeige IL32 ELISA Kits) expression in hepatitis B virus-related acute-on-chronic liver failure
B7H6 belongs to the B7 family (see MIM 605402) and is selectively expressed on tumor cells. Interaction of B7H6 with NKp30 (NCR3\; MIM 611550) results in natural killer (NK) cell activation and cytotoxicity (Brandt et al., 2009
B7 homolog 6
, natural cytotoxicity triggering receptor 3 ligand 1
, putative Ig-like domain-containing protein DKFZp686O24166/DKFZp686I21167