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ovarian BAD-interacting protein\; may inhibit apoptosis [RGD, Feb 2006].. Zusätzlich bieten wir Ihnen MCL1 Antikörper (5) und viele weitere Produktgruppen zu diesem Protein an.
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The findings support a model in which survival is determined by quantitative participation of multiple anti-apoptotic proteins, BCL2 (zeige BCL2 ELISA Kits), Mcl1, and BCL2A1 (zeige BCL2A1 ELISA Kits), rather than by a single anti-apoptotic protein.
This dynamic change in B cell survival mechanisms is unique to Epstein-Barr virus-infected cells and relies on regulation of MCL-1 mitochondrial localization and BFL-1 transcription by the viral EBNA3A protein.
Overexpression of Mcl-1 via the hemopoietic cell specific vavP-Mcl-1 transgene markedly exacerbates and accelerates the lpr (zeige FAS ELISA Kits) phenotype. The progressive splenomegaly and lymphadenopathy displayed by lpr (zeige FAS ELISA Kits) mice was far more severe in Mcl (zeige CLEC4D ELISA Kits)-1tg/lpr (zeige FAS ELISA Kits) littermates
Endothelial cell -specific deletion of Mcl1 resulted in a dose-dependent increase in endothelial cell apoptosis in the angiogenic vasculature and a corresponding decline in vessel density.
Our findings indicate that MCL-1 expression is an important biomarker of TEC (zeige NR4A3 ELISA Kits) survival
These results identify MCL-1 as a critical prosurvival protein for preventing beta-cell death and clarify the mechanisms behind its downregulation by proinflammatory cytokines.
These data show that Mcl-1 is dispensable for the regulation of apoptosis during infection with different large DNA viruses.Bcl-XL, on the other hand, can be important to maintain survival of virus-infected cells
BCL-XL (zeige BCL2L1 ELISA Kits) expression promotes survival of immature B cells, expression of BCL-2 (zeige BCL2 ELISA Kits) is important for survival of mature B cells and long-lived plasma cells (PC), and expression of MCL-1 is important for survival throughout B-cell development.
miR (zeige MLXIP ELISA Kits)-32/MCL-1 pathway members were identified as key early genetic events driving melanoma progression.
GSK3B-MCL1 signaling to regulate axonal autophagy might be important for the successful completion of Wallerian degeneration.
ovarian BAD-interacting protein\; may inhibit apoptosis
bcl-2-related protein EAT/mcl1
, induced myeloid leukemia cell differentiation protein Mcl-1 homolog