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The Fos gene family consists of 4 members FOSL1, and FOSL2. Zusätzlich bieten wir Ihnen FOSL1 Kits (22) und FOSL1 Proteine (10) und viele weitere Produktgruppen zu diesem Protein an.
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Cow (Bovine) Polyclonal FOSL1 Primary Antibody für IHC, WB - ABIN2777265
Debinski, Gibo: Fos-related antigen 1 modulates malignant features of glioma cells. in Molecular cancer research : MCR 2005
Show all 3 Pubmed References
Human Polyclonal FOSL1 Primary Antibody für ICC, IHC - ABIN1858911
Witusik-Perkowska, Zakrzewska, Sikorska, Papierz, Jaskolski, Szemraj, Liberski: Glioblastoma-derived cellsin vitrounveil the spectrum of drug resistance capability - comparative study of tumour chemosensitivity in different culture systems. in Bioscience reports 2017
Human Polyclonal FOSL1 Primary Antibody für ELISA, WB - ABIN251492
Belguise, Kersual, Galtier, Chalbos: FRA-1 expression level regulates proliferation and invasiveness of breast cancer cells. in Oncogene 2005
Fra1 inhibited the protein levels of Bcl2 (zeige BCL2 Antikörper), cMyc (zeige MYC Antikörper), Survivin (zeige BIRC5 Antikörper), and BclxL (zeige BCL2L1 Antikörper). Fra1 overexpression in intestinal mucosal epithelial cells may restrain damage repair after intestinal mucosal injury in inflammatory bowel disease remittent period through weakening the protective effect of intestinal mucosa, thus increasing the risk of recurrence.
High FRA1 expression is associated with Esophageal Squamous Cell Carcinoma.
constitutive activation and non-regulation of Fra-1 by bicalutamide in PC-3 (zeige PCSK1 Antikörper) cells suggested that Fra-1, probably a key component, involved in transition of aggressive androgen-independent PC-3 (zeige PCSK1 Antikörper) cells with poor prognosis.
the overexpression of Fra1 disrupted inflammatory cytokine secretion by medullary thymic epithelial cells in the myasthenia gravis thymus
Taken together, our results indicated that miR (zeige MLXIP Antikörper)-449 overexpression inhibited osteogenic differentiation of HG-FFA-treated hBMSCs through the Sirt1 (zeige SIRT1 Antikörper)/Fra-1 signal pathway.
These findings indicate that an aberrant Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signalling leads to the epithelial-mesenchymal transition and drug resistance of glioma via Fra1 induction, which suggests novel therapeutic strategies for the malignant disease.
MUC1 (zeige MUC1 Antikörper) enhancement of ERK (zeige EPHB2 Antikörper) activation influences FRA-1 activity to modulate tumor migration, invasion and metastasis in a subset of pancreatic cancer cases
the results indicate that FRA1 promotes cancer growth through AKT (zeige AKT1 Antikörper), and enhances cancer cell migration through JNK/c-Jun, pinpointing FRA1 as a key integrator of JNK (zeige MAPK8 Antikörper) and AKT (zeige AKT1 Antikörper) signaling pathways and a potential therapeutic target for squamous cell carcinoma of the skin and head and neck squamous cell carcinoma.
Study shows that FOSL1 is the main immediate early AP-1 (zeige JUN Antikörper) member induced by melanoma oncogenes; found that FOSL1 is involved in melanoma cell migration as well as cell proliferation and anoikis-independent growth.
a Fra-1-miR (zeige MLXIP Antikörper)-134 axis drives a positive feedback loop that amplifies ERK (zeige EPHB2 Antikörper)/JNK (zeige MAPK8 Antikörper) signaling and reduces chemosensitivity in ovarian cancer cells.
NF-kappaB (zeige NFKB1 Antikörper) and c-Jun (zeige JUN Antikörper) coregulate lipopolysaccharide-induced Fra-1 transcription.
Upon stimulation with poly(I:C), malaria parasite-infected red blood cells (iRBCs), or vesicular stomatitis virus (VSV), FOSL1 "translocated" from the nucleus to the cytoplasm, where it inhibited the interactions between TNF receptor-associated factor 3 (TRAF3 (zeige TRAF3 Antikörper)), TIR domain-containing adapter inducing IFN-beta (zeige IFNB1 Antikörper) (TRIF (zeige RNF138 Antikörper)), and Tank-binding kinase 1 (TBK1 (zeige TBK1 Antikörper)) via impairing K63-linked polyubiquitination of TRAF3 (zeige TRAF3 Antikörper) and TRIF (zeige RNF138 Antikörper).
Adipose-derived stromal cells overexpressing Fra-1 effectively protect against osteoarthritis.
Overexpression of Fosl1, which encodes a component of the AP-1 (zeige JUN Antikörper) transcription factor complex, in osteoblasts significantly blocked malleal capillary narrowing.
Fra-1 gene expression is activated in response lipopolysaccharide induced lung-injury in mice.
Data suggest that expression of Fra1 (fos-related antigen 1) in preadipocytes is up-regulated by arachidonic acid (AA) during late stages of adipogenesis; however, in early stage of adipogenesis, AA inhibits adipogenesis via Fra1-dependent mechanism.
Fra-1 plays a key role in promoting chronic CS-induced lung macrophagic inflammation in vivo
we demonstrate that Fra1 negatively controls plasma cell differentiation by repressing Blimp1 (zeige PRDM1 Antikörper) expression.
observations suggest that MyoD (zeige MYOD1 Antikörper) and pRb (zeige PGR Antikörper) work together cooperatively at the level of the intronic enhancer of Fra-1 during terminal cell cycle arrest
findings, based on a definitive genetic mouse model provide fundamental insights into mammary duct maturation and homeostasis and reveal that Scrib (zeige SCRIB Antikörper) loss activates a MAPK (zeige MAPK1 Antikörper)/Fra1 pathway that alters mammary progenitor activity
The Fos gene family consists of 4 members FOSL1, and FOSL2. These genes encode leucine zipper proteins that can dimerize with proteins of the JUN family, thereby forming the transcription factor complex AP-1. As such, the FOS proteins have been implicated as regulators of cell proliferation, differentiation, and transformation.
, fos-related antigen 1
, FOS-like antigen 1