Coagulation Factor V Proteine (F5)

F5 encodes an essential cofactor of the blood coagulation cascade. Zusätzlich bieten wir Ihnen Coagulation Factor V Antikörper (132) und Coagulation Factor V Kits (67) und viele weitere Produktgruppen zu diesem Protein an.

alle Proteine anzeigen Gen GeneID UniProt
F5 14067 O88783
F5 2153 P12259
F5 304929  
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Showing 10 out of 24 products:

Katalog Nr. Origin Quelle Konjugat Bilder Menge Anbieter Lieferzeit Preis Details
Insektenzellen Maus His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Anmelden zum Anzeigen 60 Days
$9,797.11
Details
Insektenzellen Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Anmelden zum Anzeigen 60 Days
$9,797.11
Details
Escherichia coli (E. coli) Maus His tag 100 μg Anmelden zum Anzeigen 15 bis 18 Tage
$720.00
Details
Escherichia coli (E. coli) Human His tag 100 μg Anmelden zum Anzeigen 15 bis 18 Tage
$742.00
Details
Escherichia coli (E. coli) Rind (Kuh) His tag,GST tag 100 μg Anmelden zum Anzeigen 15 bis 18 Tage
$781.00
Details
Escherichia coli (E. coli) Wildschwein His tag 100 μg Anmelden zum Anzeigen 15 bis 18 Tage
$448.00
Details
Wheat germ Human GST tag 10 μg Anmelden zum Anzeigen 11 bis 12 Tage
$414.29
Details
Escherichia coli (E. coli) Schwein Unkonjugiert SDS-PAGE analysis of Pig Coagulation Factor V Protein. 100 μg Anmelden zum Anzeigen 11 bis 18 Tage
$469.04
Details
Human Human Unkonjugiert   0.05 mg Anmelden zum Anzeigen 2 bis 3 Tage
$429.00
Details
Escherichia coli (E. coli) Ratte Unkonjugiert   100 μg Anmelden zum Anzeigen 11 bis 18 Tage
$582.75
Details

F5 Proteine nach Spezies und Herkunft

Origin Exprimiert in Konjugat
Mouse (Murine) ,

Human , , ,
,
Rat (Rattus)

Weitere Proteine zu Coagulation Factor V (F5) Interaktionspartnern

Mouse (Murine) Coagulation Factor V (F5) Interaktionspartner

  1. Mice with the FVL mutation do not have increased spermatogenesis as compared to wildtype mice.

  2. Platelet-derived FV contributes to the control of angiogenesis and is likely associated with thrombin (zeige F2 Proteine) generation in hind limb ischemia model.

  3. These findings reveal a novel biological function and mechanism of the protein C (zeige PROC Proteine) pathway in which protein S and the aPC (zeige APC Proteine)-cleaved form of fV are cofactors for anti-inflammatory cell signaling by aPC (zeige APC Proteine) in the context of endotoxemia and infection

  4. Mice deficient in LMAN1 exhibit FV and FVIII deficiencies and liver accumulation of alpha1-antitrypsin.

  5. The FVL mutation does not influence coagulation activation, lung inflammation or survival in lethal influenza A.

  6. It suggested that there could be a combination of GLA (zeige GLA Proteine) deficiency and FVL or other thrombosis-related gene defect in patients with genetic severe early-onset thrombosis.

  7. Data suggest that tissue factor (zeige F3 Proteine) and factor V induction by LPS (zeige TLR4 Proteine) may in part accelerate mesangioproliferative glomerulonephritis through activation of factor X and downstream proinflammatory and procoagulant mechanisms.

  8. The source of the FVL leading to accelerated thrombosis appears to be circulating, non-platelet-derived plasma FVL.

  9. FVL has the ability to improve the hemophilia A or B phenotype, but this effect is principally evident at the microcirculation level following a particular vascular injury.

  10. observations demonstrate a synergistic interaction between alpha-galactosidase A (zeige GLA Proteine) deficiency and Factor V Leiden toward tissue fibrin deposition; concomitant mutations in these genes may increase the penetrance of vascular thrombotic events in humans

Human Coagulation Factor V (F5) Interaktionspartner

  1. the prothrombotic activity of FII is the result of a polymorphism and of a missense mutation, whereas that of FV derives only from a polymorphism. The observation that a clotting factor (zeige F7 Proteine) defect may be associated with both bleeding or venous thrombosis depending on the site of the mutation has caused an extensive reevaluation of the blood clotting mechanism.

  2. study found the FVL A allele frequency and GA genotype are significantly more prevalent among patients with coronary artery disease (CAD) compared to controls and may be predisposing to CAD; further found that the FVL mutation is an independent risk factor whose effect is not modified by other risk factors; FV HR2 variation does not show any statistically significant association with CAD

  3. suggesting that the FVL paradox is related to the carriership of one wild type and one mutated factor V allele

  4. Review/Meta-analysis: Factor V G1691A single nucleotide gene polymorphism was associated with risk of ischemic stroke mainly in young adults.

  5. Factor V Leiden mutation is associated with venous thromboembolism in cancer.

  6. we were not able to confirm the association between the polymorphisms of f5, f2, and mthfr (zeige MTHFR Proteine) and pregnancy loss in Bosnian women

  7. Human FVL carriers have a higher total sperm count than non-carriers, with an adjusted mean difference of 31 x 106 (95%CI 0.2-61.7; P = 0.048).

  8. contribution of FVLeiden causing resistance to activated protein C in Indian population is not as strong as previously reported in Western countries

  9. The frequencies of GA and AA genotypes and A allele of coagulation factor V (FV) 1691G>A polymorphism significantly increased in the lower extremity deep venous thrombosis (LDVT) group. Patients with LDVT carrying A allele (GA + AA) had both higher patency and recurrence rates than those carrying GG genotype. Coagulation factor V (FV) 1691G>A polymorphism may be associated with both the risk and prognosis of LDVT.

  10. Factor V Leiden-mutations were found in 16.8% of patients with cerebral sinus venous thrombosis and in 17.8% of patients with arterial ischemic stroke, which was significantly more frequent than in controls at a rate of 4.95% (ORs: 3.89 and 4.16).

Cow (Bovine) Coagulation Factor V (F5) Interaktionspartner

  1. Data suggest factor Xa (FXa (zeige F10 Proteine)) and factor Va (FVa) compete to bind FXa (zeige F10 Proteine) on both PS model membranes and microparticles from activated platelets; this competition between dimerization/prothrombinase (zeige FGL2 Proteine) complex formation appears to regulate blood coagulation.

Coagulation Factor V (F5) Protein Überblick

Protein Überblick

This gene encodes an essential cofactor of the blood coagulation cascade. This factor circulates in plasma, and is converted to the active form by the release of the activation peptide by thrombin during coagulation. This generates a heavy chain and a light chain which are held together by calcium ions. The activated protein is a cofactor that participates with activated coagulation factor X to activate prothrombin to thrombin. Defects in this gene result in either an autosomal recessive hemorrhagic diathesis or an autosomal dominant form of thrombophilia, which is known as activated protein C resistance.

Genbezeichner und Symbole assoziert mit Coagulation Factor V Proteine (F5)

  • coagulation factor V (f5)
  • coagulation factor V (F5)
  • Ac2-120 Protein
  • AI173222 Protein
  • Cf-5 Protein
  • Cf5 Protein
  • FVL Protein
  • PCCF Protein
  • RPRGL1 Protein
  • THPH2 Protein

Bezeichner auf Proteinebene für Coagulation Factor V Proteine (F5)

coagulation factor V , activated protein C cofactor , activated protein c cofactor , coagulation factor V jinjiang A2 domain , factor V Leiden , proaccelerin, labile factor

GENE ID SPEZIES
100534566 Oreochromis niloticus
14067 Mus musculus
2153 Homo sapiens
304929 Rattus norvegicus
280687 Bos taurus
100715882 Cavia porcellus
397217 Sus scrofa
100685858 Canis lupus familiaris
101123649 Ovis aries
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