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anti-Mouse (Murine) Apelin Antikörper:
anti-Human Apelin Antikörper:
anti-Rat (Rattus) Apelin Antikörper:
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Human Polyclonal Apelin Primary Antibody für ELISA, IF - ABIN347115
Daviaud, Boucher, Gesta, Dray, Guigne, Quilliot, Ayav, Ziegler, Carpene, Saulnier-Blache, Valet, Castan-Laurell: TNFalpha up-regulates apelin expression in human and mouse adipose tissue. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006
Show all 4 Pubmed References
Human Polyclonal Apelin Primary Antibody für WB - ABIN522328
Li, Takai, Yuge, Furukawa, Tsuno, Tsukamoto, Kong, Moriyama, Narahara: Novel target genes responsive to the anti-growth activity of triptolide in endometrial and ovarian cancer cells. in Cancer letters 2010
Apelin signaling regulates lymphatic development by promoting serine-threonine kinase Akt/protein kinase B (zeige AKT1 Antikörper) activity in a VEGF-C (zeige VEGFC Antikörper)/VEGF receptor (zeige FLT1 Antikörper) 3-independent manner during zebrafish embryogenesis.
Apelin knockdown inhibits both hypoxia-induced endothelial cell proliferation in vitro and hypoxia-induced vessel regeneration in the caudal (zeige CAD Antikörper) fin regeneration of zebrafish.
Results revealed that apelin-13 attenuated brain edema and reduced cellular death by suppressing apoptosis after intracerebral hemorrhage in mice.
Apelin-36-[L28C(30kDa (zeige TAF10 Antikörper)-PEG (zeige PAEP Antikörper))] provides a starting point for the development of diabetes therapeutics that are devoid of the blood pressure effects associated with canonical APJ (zeige APLNR Antikörper) activation
miR (zeige MLXIP Antikörper)-503 promotes cardiac fibrosis via miR (zeige MLXIP Antikörper)-503-Apelin-13-TGF-beta (zeige TGFB1 Antikörper)-CTGF (zeige CTGF Antikörper)-collagen production pathway.
Apelin directly interferes with thrombin-mediated signaling pathways and platelet activation, secretion, and aggregation, but not with ADP and thromboxane A2-mediated pathways.
Apelin regulates inflammatory response, diminishes inflammatory remote organ damage and improves survival in an experimental model of burn sepsis, which is at least partly mediated by a phosphatidylinositol 3-kinase/protein kinase B (zeige AKT1 Antikörper) dependent pathway.
apelin is a potential activator of inflammation factors through the PI3K/Akt (zeige AKT1 Antikörper) and Erk (zeige EPHB2 Antikörper) signaling pathway and is potential therapeutically relevant to inflammatory responses of microglia cells
we showed the effect of Apelin on RAW264.7 macrophage under normal and hypoxic condition, which could further influence the angiogenesis and inflammation process that promoted by macrophages.
PDL- and Cer (zeige CBLN1 Antikörper)-induced CP resulted in increased production of the pancreatic BMP2 (zeige BMP2 Antikörper), apelin, and PTHrP (zeige PTHLH Antikörper) signaling systems and that significant cross talk occurred among pancreatic BMP2 (zeige BMP2 Antikörper), apelin, and PTHrP (zeige PTHLH Antikörper)
Therapeutic potential of apelin to prevent myocardial metabolic abnormalities in heart failure paired with obesity.
Data suggest that CTRP3 (cartducin) up-regulates expression and secretion of adipokines (adiponectin, leptin, visfatin, and apelin) in adipocytes; these responses to CTRP3 are down-regulated by insulin resistance or inhibition of AMPK signaling.
APJ receptor (zeige APLNR Antikörper) in granulosa cells and both apelin and the APJ receptor (zeige APLNR Antikörper) in theca tissues are expressed in bovine ovary.
The data suggest that apelin/APJ (zeige APLNR Antikörper) system is involved in the mechanism regulating angiogenesis during follicle maturation as well as during corpora lutea formation and function in the bovine ovary.
Apelin concentrations are associated with altered plaque stability mediator levels and atherosclerosis in patients with rheumatoid arthritis. These relations are partially dependent on population origin and systemic inflammatory status.
APLN and APLNR (zeige APLNR Antikörper) are present in human ovarian cells and APLN increases IGF1 (zeige IGF1 Antikörper)-induced steroidogenesis in granulosa cells through an increase in HSD3B (zeige HSD3B1 Antikörper) protein expression and activation of the MAPK3 (zeige MAPK3 Antikörper)/1 and Akt (zeige AKT1 Antikörper) pathways. Therefore, APLN and APLNR (zeige APLNR Antikörper) may play a role in human follicular development and the pathogenesis of PCOS.
Apelin levels mirror the cardiovascular changes seen in pregnancy. Serum and placental apelin levels are decreased in intrauterine growth restriction .
Reduced apelin levels may enhance vasoconstriction to influence high BP and heart's workload in EH and ACS.
Our data suggest that tumor Apelin can be used as a marker to evaluate clinical characteristics and predict prognosis in GC patients.
The apelin/APLNR (zeige APLNR Antikörper) axis regulates cholangiocarcinoma cell proliferation and tumor angiogenesis.
While plasma apelin increases in obesity, it decreases with increasing age and pubertal stage
Increased plasma concentrations of apelin are associated with ST-segment elevation acute myocardial infarction severity.
Data suggest that the differential expression of Apelin and Apelin receptor APJ (zeige APLNR Antikörper) yields a "self-generated" gradient mechanisms that accelerates the extension of the sprout.
Postmenopausal breast cancer patients had high levels of apelin and high BMI scores.
Pig Apelin mRNA was sequenced.
This gene encodes a peptide that functions as an endogenous ligand for the G protein coupled receptor APJ. The encoded protein is synthesized as a prepropeptide that is processed into biologically active C-terminal fragments. The peptide fragments activate different tissue specific signaling pathways that regulate diverse biological functions including fluid homeostasis, cardiovascular function and insulin secretion. This protein also functions as a coreceptor for the human immunodeficiency virus 1.
, apelin, AGTRL1 ligand
, APJ endogenous ligand
, apelin; peptide ligand for APJ receptor
, AGTRL1 ligand