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anti-Human CFP Antikörper:
anti-Mouse (Murine) CFP Antikörper:
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Human Monoclonal CFP Primary Antibody für IHC (fro), ELISA - ABIN2473074
Shafa: Visually elicited response of the frog cerebellum. in Brain research 1977
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Human Monoclonal CFP Primary Antibody für Func, IHC (fro) - ABIN2473073
Hourcade: The role of properdin in the assembly of the alternative pathway C3 convertases of complement. in The Journal of biological chemistry 2006
Show all 2 Pubmed References
All Species Polyclonal CFP Primary Antibody für WB - ABIN411450
Bhattacharya, Ansari, Hamatake, Walker, Kazmierski, Striker: Pharmacological disruption of hepatitis C NS5A protein intra- and intermolecular conformations. in The Journal of general virology 2014
data indicate that properdin enhances platelet/granulocyte aggregates (PGAs) formation via increased production of C5a, and that inhibition of properdin function has therapeutic potential to limit thromboinflammation in diseases characterized by increased PGA formation
this study shows that RNA interference of properdin in dendritic cells decreased alloantigen-specific T-cell proliferation
studies demonstrate an essential role of properdin oligomerization in vivo while our monomers enable detailed structural insight paving the way for novel modulators of complement.
can directly interact with neutrophil myeloperoxidase (zeige MPO Antikörper) resulting in activation of alternative pathway of complement
Our data show that physiological forms of human properdin bind directly to human platelets after activation by strong agonists in the absence of C3
Factor h (zeige CFH Antikörper) and properdin recognize different epitopes on renal tubular epithelial heparan sulfate.
Properdin released from human polymorphonuclear cells does not bind to zymosan or E. coli, but when incubated in properdin-depleted serum this form of properdin binds efficiently to both substrates in a strictly complement C3 (zeige C3 Antikörper)-dependent manner.
Properdin and SC5b-9 may be novel biomarkers for future risk of type 2 diabetes in this high-risk population and warrant further investigation.
Immune human serum that contained bactericidal Abs directed against the 2C (zeige C4BPA Antikörper)7 lipooligosaccharide epitope (zeige C4BPA Antikörper)required functional properdin to kill C4BP-binding strains, but not C4BP-nonbinding strains.
report a large Finnish family with a novel mutation in the properdin gene. The mutation is located in exon 9 and changes guanine to adenine at nucleotide 1164 (c.1164G>A) that causes tryptophan to change to a premature stop codon (W388X).
Properdin (zeige CFB Antikörper) deficiency protects from 5-fluorouracil-induced small intestinal mucositis in a complement activation-independent, interleukin-10 (zeige IL10 Antikörper)-dependent mechanism
We induced anti-myeloperoxidase vasculitis in mice and showed that neither MASP-2- nor properdin-deficient mice were protected, suggesting that alternative pathway activation does not require properdin or the lectin pathway.
The results show that Properdin (zeige CFB Antikörper) plays a key role in allergen-induced airway inflammation and represents a potential therapeutic target for human asthma.
Authors conclude that properdin (zeige CFB Antikörper) controls the strength of immune responses by affecting humoral as well as cellular phenotypes during acute bacterial infection and ensuing inflammation.
Properdin (zeige CFB Antikörper) deficiency exacerbated renal injury in mice lacking complement factor H (zeige CFH Antikörper).
deficiency of properdin (zeige CFB Antikörper) in mice with factor H (zeige CFH Antikörper) mutation exacerbates C3 glomerulonephritis
Murine antigonococcal antiserum required functional properdin (zeige CFB Antikörper) to kill C4BP-binding strains, but not C4BP-nonbinding strains.
properdin (zeige CFB Antikörper) may be added to the list of complement proteins which influence lipid metabolism, energy storage and insulin (zeige INS Antikörper) resistance, and further support the hypothesis of a dual role of complement in adipose tissue
At the initial phase of local, zymosan-induced inflammation, properdin (zeige CFB Antikörper)-deficient and wild-type mice showed bone erosion, proteoglycan (zeige Vcan Antikörper) loss and cell infiltration.
Properdin (zeige CFB Antikörper) localizes to mast cells, has the ability to directly associate with Escherichia coli DH5alpha, and plays a significant role in the outcome of polymicrobial sepsis.
This gene encodes a plasma glycoprotein that positively regulates the alternative complement pathway of the innate immune system. This protein binds to many microbial surfaces and apoptotic cells and stabilizes the C3- and C5-convertase enzyme complexes in a feedback loop that ultimately leads to formation of the membrane attack complex and lysis of the target cell. Mutations in this gene result in two forms of properdin deficiency, which results in high susceptibility to meningococcal infections. Multiple alternatively spliced variants, encoding the same protein, have been identified.
complement factor P
, properdin P factor, complement
, properdin factor, complement
, Properdin P factor, complement