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anti-Human CTH Antikörper:
anti-Mouse (Murine) CTH Antikörper:
anti-Rat (Rattus) CTH Antikörper:
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Human Monoclonal CTH Primary Antibody für ELISA, WB - ABIN560521
Schicho, Krueger, Zeller, Von Weyhern, Frieling, Kimura, Ishii, De Giorgio, Campi, Schemann: Hydrogen sulfide is a novel prosecretory neuromodulator in the Guinea-pig and human colon. in Gastroenterology 2006
Show all 21 Pubmed References
Human Monoclonal CTH Primary Antibody für IHC (p), ELISA - ABIN560523
Siebert, Cantré, Eipel, Vollmar: H2S contributes to the hepatic arterial buffer response and mediates vasorelaxation of the hepatic artery via activation of K(ATP) channels. in American journal of physiology. Gastrointestinal and liver physiology 2008
Show all 7 Pubmed References
Human Monoclonal CTH Primary Antibody für ELISA, WB - ABIN560522
dEmmanuele di Villa Bianca, Sorrentino, Maffia, Mirone, Imbimbo, Fusco, De Palma, Ignarro, Cirino: Hydrogen sulfide as a mediator of human corpus cavernosum smooth-muscle relaxation. in Proceedings of the National Academy of Sciences of the United States of America 2009
Show all 5 Pubmed References
Cow (Bovine) Polyclonal CTH Primary Antibody für WB - ABIN2782463
Moore, Malats, Rothman, Real, Kogevinas, Karami, García-Closas, Silverman, Chanock, Welch, Tardón, Serra, Carrato, Dosemeci, García-Closas: Polymorphisms in one-carbon metabolism and trans-sulfuration pathway genes and susceptibility to bladder cancer. in International journal of cancer. Journal international du cancer 2007
Show all 4 Pubmed References
Human Monoclonal CTH Primary Antibody für IHC (p), WB - ABIN2473121
Fu, Liu, Geng, Fang, Tang: Hydrogen sulfide protects rat lung from ischemia-reperfusion injury. in Life sciences 2008
Human Polyclonal CTH Primary Antibody für IF (p), IHC (p) - ABIN1387469
Shiina, Shima, Horii, Naitou, Nakamori, Sano, Shimizu: Inhibitory action of hydrogen sulfide on esophageal striated muscle motility in rats. in European journal of pharmacology 2016
Cow (Bovine) Polyclonal CTH Primary Antibody für IHC, WB - ABIN2782464
Yang, Cao, Wu, Wang: Cystathionine gamma-lyase overexpression inhibits cell proliferation via a H2S-dependent modulation of ERK1/2 phosphorylation and p21Cip/WAK-1. in The Journal of biological chemistry 2004
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Inducible expression of CBS and CSE was found to be associated with a mineralizing phenotype in mesenchymal stem cells transitioning to mineralizing osteoblasts.
ur findings suggest that these two families of amines inhibit cystathionine-gamma-lyase (CSE) to varying extents, which directly results in altered levels of intracellular HCY and consequent changes in Human vascular smooth muscle cells proliferation.
Lipopolysaccharide increases the expression of CSE in human macrophages, increasing H2S synthesis, via ERK (zeige EPHB2 Antikörper)/NF-kappaB (zeige NFKB1 Antikörper) pathway.
Nox4 (zeige NOX4 Antikörper) is a positive transcriptional regulator of cystathionine-gamma-lyase in endothelial cells.
The expression of CSE was positively correlated with the severity of gastric ulcer
Our study demonstrates that CSE/H2S system is regulated by miR (zeige MLXIP Antikörper)-216a, and regulates ABCA1 (zeige ABCA1 Antikörper)-mediated cholesterol efflux and cholesterol levels through the PI3K (zeige PIK3CA Antikörper)/AKT (zeige AKT1 Antikörper) pathway.
Collectively, our findings indicate that radiation could promote HCC (zeige FAM126A Antikörper) cell invasion through EMT (zeige ITK Antikörper) mediated by endogenous H2S/CSE signaling via the p38MAPK (zeige MAPK14 Antikörper) pathway.
3-Mercaptopyruvate sulphurtransferase and not cystathionine gamma-lyase is the primary regulator of coronary artery hydrogen sulfide (zeige SQRDL Antikörper) production and function.
An increase of placental mRNA levels in the pre-eclampsia group was observed for methionine synthase (zeige MTR Antikörper) and cystathionine gamma-lyase.
GPBAR1 plays a role in secondary bile acid induced vasodilation via reglation of cystathionine gamma-lyase. The GPBAR1/CSE pathway might contribute to endothelial dysfunction and hyperdynamic circulation in liver cirrhosis.
Exogenous administration of CO exacerbated allergic symptoms, resulting in higher levels of both CO and heme oxygenase-1 expression, and a further reduction in H2S levels and CSE expression.
The antiatherosclerotic effect of estrogen is mediated by CSE-generated H2S
Cystathionine-Gamma-Lyase Gene Deletion is associated with reduced inflammation and liver damage.
this study reveals the molecular basis for the differential expression of Cth in mouse models of essential hypertension under basal and pathophysiological conditions.
the findings of this study indicate that a deficiency in 3MST does not significantly affect endotoxemia, while a deficiency in CBS (zeige CBS Antikörper) or CSE slightly ameliorates the outcome of LPS (zeige TLR4 Antikörper)-induced endotoxemia in vivo.
lung MPO activity increased following induction of sepsis with CLP while siRNA treatment significantly reduced MPO activity. Liver and lung cytokine and chemokine levels in CLP-induced sepsis reduced following treatment with siRNA. These findings show a crucial pro-inflammatory role for H2S synthesized by CSE in macrophages in sepsis and suggest CSE gene silencing with siRNA as a potential therapeutic approach for this co
H2S augmented the S-sulfhydration of the rate-limiting gluconeogenic enzymes and PGC-1alpha (zeige PPARGC1A Antikörper) and increased their activities, which were lower in untreated : To investigate the regulation of hepatic glucose production by cystathionine gamma-lyase KO hepatocytes
Endogenous cystathionine gamma-lyase/Hydrogen sulfide (zeige SQRDL Antikörper) regulates ischaemic vascular remodelling mediated during hind limb ischaemia through NO-dependent monocyte recruitment
The results of this study suggest that CSE is not critically involved in chronic pain signaling in mice and that sources different from CSE mediate the pain relevant effects of H2S.
CSE-H2S increased PPARgamma (zeige PPARG Antikörper) activity by direct sulfhydration at the C139 site, thereby changing glucose into triglyceride storage in adipocytes.
Therefore, our data suggest that DNA hypermethylation of CpG rich region in cse promoter might contribute to the decrease of cse transcription and H2S production in macrophages, and thus contribute to atherosclerosis development.
This gene encodes a cytoplasmic enzyme in the trans-sulfuration pathway that converts cystathione derived from methionine into cysteine. Glutathione synthesis in the liver is dependent upon the availability of cysteine. Mutations in this gene cause cystathioninuria. Alternative splicing of this gene results in three transcript variants encoding different isoforms.
, cystathionase (cystathionine gamma-lyase)
, cysteine desulfhydrase
, cysteine-protein sulfhydrase
, homoserine deaminase
, homoserine dehydratase
, CTL target antigen
, probasin-related antigen