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TIMP4 belongs to the TIMP gene family. Zusätzlich bieten wir Ihnen TIMP4 Kits (69) und TIMP4 Proteine (30) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 185 products:
Human Monoclonal TIMP4 Primary Antibody für IHC (fro), IF - ABIN371660
Greene, Wang, Liu, Raymond, Rosen, Shi: Molecular cloning and characterization of human tissue inhibitor of metalloproteinase 4. in The Journal of biological chemistry 1997
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Human Polyclonal TIMP4 Primary Antibody für EIA, IHC (p) - ABIN955223
Liss, Sreedhar, Keshgegian, Sauter, Chernick, Prendergast, Wallon: Tissue inhibitor of metalloproteinase-4 is elevated in early-stage breast cancers with accelerated progression and poor clinical course. in The American journal of pathology 2009
Zeige alle 2 Referenzen für 955223
Mouse (Murine) Monoclonal TIMP4 Primary Antibody für IHC, ELISA - ABIN1043744
Donover, Wojciechowski, Thirumaran, Zemba-Palko, Prendergast, Wallon: Development of a monoclonal antibody that specifically detects tissue inhibitor of metalloproteinase-4 (TIMP-4) in formalin-fixed, paraffin-embedded human tissues. in Journal of cellular biochemistry 2010
Human Polyclonal TIMP4 Primary Antibody für ICC, IF - ABIN4359834
Silva, Nascimento, Pereira, Siqueira, Brum, Jaeger, Miyabara: β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9. in Cell and tissue research 2016
This report provides the first example that TIMP-4 regulates carcinogenesis through enriching the tumor progenitor cell population in cervical cancer cells.
Study evaluated MMP-12 (zeige MMP12 Antikörper) and TIMP-1 (zeige TIMP1 Antikörper), TIMP-2 (zeige TIMP2 Antikörper), TIMP-3 (zeige TIMP3 Antikörper), and TIMP-4 levels in 40 patients with asymptomatic and symptomatic critical carotid artery stenosis (CAS (zeige CSE1L Antikörper)) with neurologic symptoms onset within the preceding 12 hours; results suggest that MMP-12 (zeige MMP12 Antikörper) is related to critical CAS (zeige CSE1L Antikörper) independently on symptoms, moreover, TIMP-3 (zeige TIMP3 Antikörper) and TIMP-4 seem to be specifically related to stroke
Concluding, miR (zeige MLXIP Antikörper)-200b-3p mediates regulation of TIMP4 expression in prostate cancer but exact mechanism needs to be investigated.
TIMP1 (zeige TIMP1 Antikörper), TIMP2 (zeige TIMP2 Antikörper), and TIMP4 are increased in aqueous humor from primary open angle glaucoma patients.
ncreased TGFB1 (zeige TGFB1 Antikörper) expression and decreased TIMP-4 expression correlated with atrial fibrosis and extracellular matrix changes in the atria of rheumatic heart disease patients with atrial fibrillation.
Upregulation of plasma TIMP-4 might contribute to PIH [pregnancy-induced hypertension] processes
Expression levels of TIMP-1 (zeige TIMP1 Antikörper), TIMP-3 (zeige TIMP3 Antikörper) and TIMP-4 were negligible (<10% of cells) in primary spontaneous pneumothorax lesions.
No evidence was found for any associations between the TIMP-1,-2,-3, or -4 gene single nucleotide polymorphisms with unexplained recurrent spontaneous abortions in this Han Chinese Han population.
This study provides evidence that the promoter TIMP4 rs3755724 is a new focal epilepsy susceptibility variant that is plausibly involved in inflammation-induced seizures in Malaysian Chinese.
Data show that gene polymorphisms of TIMP metallopeptidase (zeige ECEL1 Antikörper) inhibitors TIMP-3 (zeige TIMP3 Antikörper) -1296 T>C (rs9619311) and TIMP-4 -55 T>C (rs3755724) were associated with the susceptibility of hepatocellular carcinoma among Taiwan women.
there are 7 CpG islands in the TIMP4 promoter which get methylated during the progression of heart failure which leads to its epigenetic silencing.
High fat diet-induced obesity downregulates adipocyte MMP3 (zeige MMP3 Antikörper) expression to trigger adipogenesis, and adipocyte TIMP4 may modulate this process to regulate hyperplastic vs. hypertrophic adipose tissue expansion, fat distribution, and metabolic health in a sex- and depot-dependent manner.
Our findings highlight a protective mechanism induced by PPARgamma (zeige PPARG Antikörper) in deoxycorticosterone acetate-salt treatment, establishing a novel mechanistic link between PPARgamma (zeige PPARG Antikörper) and TIMP-4.
TIMP4 augments contractility and induces differentiation of embryonic stem cells into cardiac phenotype.
TIMP-4 might act as a negative regulator of adipogenesis through NFkB cascade modulation.
Replenishment of myocardial TIMP4 could serve as an effective therapy in post-I/R recovery for patients with reduced TIMP4.
TIMP-4 protein was localized to corpus luteum and theca-intera cells in various periods throughout the pregnancy and postpartum day 1.
Timp4(-/-) mice are more susceptible to MI but not to pressure overload, and TIMP4 functions in its capacity as a metalloproteinase inhibitor after myocardial infarction
Identification of an initiator-like element essential for the expression of the tissue inhibitor of metalloproteinases-4 (Timp-4) gene
molecular cloning and localization of the gene within intron 5 of the synapsin 2 (zeige SYN2 Antikörper) gene
This gene belongs to the TIMP gene family. The proteins encoded by this gene family are inhibitors of the matrix metalloproteinases, a group of peptidases involved in degradation of the extracellular matrix. The secreted, netrin domain-containing protein encoded by this gene is involved in regulation of platelet aggregation and recruitment and may play role in hormonal regulation and endometrial tissue remodeling.
TIMP metallopeptidase inhibitor 4
, tissue inhibitor of metalloproteinase 4
, metalloproteinase inhibitor 4
, tissue inhibitor of metalloproteinases 4
, tissue inhibitor of metalloproteinase
, tissue inhibitor of metalloproteinase-4