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MaxiK channels are large conductance, voltage and calcium-sensitive potassium channels which are fundamental to the control of smooth muscle tone and neuronal excitability. Zusätzlich bieten wir Ihnen Potassium Large Conductance Calcium-Activated Channel, Subfamily M, beta Member 2 Proteine (5) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 79 products:
Human Polyclonal KCNMB2 Primary Antibody für WB - ABIN1881476
Zarei, Song, Wilson, Cox, Colom, Knaus, Stefani, Toro: Endocytic trafficking signals in KCNMB2 regulate surface expression of a large conductance voltage and Ca(2+)-activated K+ channel. in Neuroscience 2007
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Mammalian Monoclonal KCNMB2 Primary Antibody für ISt, IHC - ABIN1304557
Zhang, Mok, Lee, Charbonnet, Gold: Inflammation-induced changes in BK(Ca) currents in cutaneous dorsal root ganglion neurons from the adult rat. in Molecular pain 2012
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The study verified that the BK(Ca) channel alpha-subunit (zeige POLG Antikörper) is located to smooth muscle cells of porcine basilar and middle cerebral arteries.
IFN-gamma (zeige IFNG Antikörper) changed mRNA levels of the BK beta-modulatory proteins KCNMB2 (increased) and KCNMB4 (zeige KCNMB4 Antikörper) (decreased) as well as leucine-rich repeat-containing protein (LRRC)26 (zeige LRRC26 Antikörper) (decreased).
results reveal the molecular regions in the beta1 and beta2 subunits that determine their differential functional coupling with the pore-forming alpha-subunit (zeige POLG Antikörper)
significance of inactivation in BK channels mediated by beta subunits arises not from inactivation, but from differences in the amplitude and duration of repolarizing currents arising from the beta-subunit (zeige POLG Antikörper)-specific energetics of recovery from inactivation
reports that coexpression of beta2 subunit (KCNMB2) with hSlo (zeige KCNMA1 Antikörper) can also modulate hSlo (zeige KCNMA1 Antikörper) surface expression levels in HEK293T cells
Report beta subunit (zeige POLG Antikörper) (KNMB1-4)-specific modulations of BK channel (zeige KCNMA1 Antikörper) function by a Slo1 (zeige KCNMA1 Antikörper) mutation associated with epilepsy and dyskinesia.
MaxiK channels are large conductance, voltage and calcium-sensitive potassium channels which are fundamental to the control of smooth muscle tone and neuronal excitability. MaxiK channels can be formed by 2 subunits: the pore-forming alpha subunit and the modulatory beta subunit. The protein encoded by this gene is an auxiliary beta subunit which decreases the activation time of MaxiK alpha subunit currents. Two variants encoding the same protein have been found for this gene.
calcium-activated potassium channel beta 2 subunit
, potassium large conductance calcium-activated channel, subfamily M beta member 3
, calcium-activated potassium channel subunit beta-2
, calcium-activated potassium channel subunit beta-3
, potassium large conductance calcium-activated channel, subfamily M, beta member 2
, BK channel subunit beta-2
, MaxiK channel beta 2 subunit
, charybdotoxin receptor subunit beta-2
, large conductance calcium-activated potassium channel beta 2 subunit
, large-conductance Ca2+-activated K+ channel beta2 subunit
, calcium-activated potassium channel, subfamily M subunit beta-2
, maxi K channel subunit beta-2
, calcium-activated potassium channel beta 2