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anti-Mouse (Murine) WNT3A Antikörper:
anti-Human WNT3A Antikörper:
anti-Rat (Rattus) WNT3A Antikörper:
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Human Polyclonal WNT3A Primary Antibody für IF (p), IHC (p) - ABIN733178
Gao, Yang, Gao, Xu, Niu, Li, Wen: Salvianolic acid B improves bone marrow-derived mesenchymal stem cell differentiation into alveolar epithelial cells type I via Wnt signaling. in Molecular medicine reports 2015
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Human Polyclonal WNT3A Primary Antibody für IHC (p), ELISA - ABIN2477144
Skalská: [Use of linear logistic regression in classification of observations]. in Sborník v?deckých prací Léka?ské fakulty Karlovy univerzity v Hradci Králové. Supplementum 1991
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Human Polyclonal WNT3A Primary Antibody für ELISA, WB - ABIN2477145
Lai, Cheng, Cheng, Feasel, Beste, Peng, Nusrat, Moreno: SOX4 interacts with plakoglobin in a Wnt3a-dependent manner in prostate cancer cells. in BMC cell biology 2011
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Cow (Bovine) Polyclonal WNT3A Primary Antibody für IHC, WB - ABIN2787841
Chiquet, Blanton, Burt, Ma, Stal, Mulliken, Hecht: Variation in WNT genes is associated with non-syndromic cleft lip with or without cleft palate. in Human molecular genetics 2008
Human Monoclonal WNT3A Primary Antibody für ELISA, ICC - ABIN449669
Denysenko, Annovazzi, Cassoni, Melcarne, Mellai, Schiffer: WNT/β-catenin Signaling Pathway and Downstream Modulators in Low- and High-grade Glioma. in Cancer genomics & proteomics 2015
These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (zeige CTNNB1 Antikörper) and link eNOS (zeige NOS3 Antikörper)-derived NO to the modulation by VEGF (zeige VEGFA Antikörper) of Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper)-induced endothelial cell proliferation.
These results indicate that PEDF (zeige SERPINF1 Antikörper) counters Wnt (zeige WNT2 Antikörper) signaling to allow for osteoblast differentiation and provides a mechanistic insight into how the PEDF (zeige SERPINF1 Antikörper) null state results in OI type VI.
These results suggest novel mechanisms for Wnt3a-induced osteoblast proliferation and cell survival via Npnt (zeige NPNT Antikörper) gene expression
this study shows that the Wnt3a expression levels in dendritic cells influences the generation of memory T cells after 5 days in co-culture with naive T cells through activation of the Wnt (zeige WNT2 Antikörper) canonical pathway
Wnt3a induces Osx (zeige SP7 Antikörper) expression via p38 MAPK (zeige MAPK14 Antikörper) signaling in dental follicle cells. Wnt3a-induced Osx (zeige SP7 Antikörper) expression was inhibited in the presence of p38 mitogen-activated protein kinase (zeige MAPK14 Antikörper) (MAPK (zeige MAPK1 Antikörper)) inhibitors (SB203580 and SB202190) at gene and protein levels, as assessed by real-time PCR and immunocytohistochemistry, respectively.
Furthermore, pigment epithelium-derived factor (PEDF (zeige SERPINF1 Antikörper)), a secreted glycoprotein known for its anti-tumor properties, blocked Wnt3a-directed induction of autophagy proteins. Autophagy inhibition was complemented by reciprocal regulation of the oxidative stress enzymes, superoxide dismutase 2 (SOD2 (zeige SOD2 Antikörper)) and catalase (zeige CAT Antikörper).
Ginkgo biloba exocarp extract inhibits tumor angiogenesis, which may be closely relevant to its effect in blockage of Wnt /beta-catenin-VEGF signaling pathway in Lewis lung carcinoma.
Western blot analysis demonstrated that following BMP2 (zeige BMP2 Antikörper) and BMP7 (zeige BMP7 Antikörper) cotransfection of MC3T3E1 cells, the protein expression levels of BMP2 (zeige BMP2 Antikörper), BMP4 (zeige BMP4 Antikörper), BMP6 (zeige BMP6 Antikörper), BMP7 (zeige BMP7 Antikörper), BMP9 (zeige GDF2 Antikörper) and Wnt3a were increased compared with control cells
Wnt3a promotes macrophage-mediated bacterial killing by elevating CRAMP and BD1 (zeige DEFB1 Antikörper) levels.
Wnt3a acutely reduces nuclear acetyl-CoA (zeige LPCAT1 Antikörper), the necessary substrate for histone acetyltransferases, resulting in a global decrease in histone acetylation.
study reveals that Foxi1 (zeige FOXI1 Antikörper)/miR (zeige MLXIP Antikörper)-491-5p/Wnt3a/beta-catenin (zeige CTNNB1 Antikörper) signaling is critical in the progression of GC. Targeting the pathway described in this study may open up new prospects to restrict the progression of gastric cancer
CHIR suppressed the hypertrophic propensity of the MSC (zeige MSC Antikörper)-derived cartilage after in vivo implantation to an extent approaching that of WNT3A protein. These results indicate that CHIR may be a promising alternative for WNT3A protein for certain applications of human bone marrow-derived MSCs.
PEGylated Wnt3A liposomes associated with skeletal stem cell populations in human bone marrow and promoted osteogenesis.
The AChE plays a role in osteoblastic differentiation and is regulated by both Wnt3a and Runx2 (zeige RUNX2 Antikörper).
In promoting the self-renewal symmetric division of hTERT(high) prostate cancer cells, WNT3a dramatically decreased the ratio of hTERT(high) prostate cancer cells undergoing asymmetric division. Increased WNT (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signal activation was also detected in hTERT(high) prostate cancer cells. hTERT-mediated CSC properties were at least partially dependent on beta-catenin (zeige CTNNB1 Antikörper).
the results indicate neighboring structural elements within full-length Wnt3a affect saposin-like subdomain (SLD) conformational stability. Moreover, SLD function(s) in Wnt (zeige WNT2 Antikörper) proteins appear to have evolved away from those commonly attributed to SAPLIP family members.
Data suggest that PORCN (zeige PORCN Antikörper) exhibits substrate specificity that includes a Wnt3a peptide fragment (residues 199-219, with disulfide bonds); recombinant PORCN (zeige PORCN Antikörper) containing a point mutation (R228C) associated with focal dermal hypoplasia exhibits impaired acylation activity toward Wnt3a peptide fragment. (PORCN (zeige PORCN Antikörper) = porcupine (zeige PORCN Antikörper) O-acyltransferase; Wnt3a = Wnt (zeige WNT2 Antikörper) family member 3A)
CPE (zeige CPE Antikörper) through its N'-terminal sequence, forms aggregates with Wnt3a and possible endoplasmic reticulum (ER) stress leading to its loss of function.
Our findings suggest that Pyk2 (zeige PTK2B Antikörper) plays an important role in the coordination of stabilization of beta-catenin (zeige CTNNB1 Antikörper) in the crosstalk between Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) and Wnt (zeige WNT2 Antikörper)/Ca(2 (zeige CA2 Antikörper)+) signaling pathways upon Wnt3a stimulation in differentiating hNPCs.
Macrophage polarization seems to have a key role in the progression of pediatric non-alcoholic fatty liver disease; the modulation of macrophage polarization could drive hepatic progenitor cell response by Wnt3a production and beta-catenin (zeige CTNNB1 Antikörper) phosphorylation.
Data suggest that Wnt3, Wnt3a, and Wnt8a (zeige WNT8A Antikörper) bind to their respective receptors (Fz8 (zeige FZD2 Antikörper), Lrp6 (zeige LRP5 Antikörper), and Lypd6 (zeige LYPD6 Antikörper)) in ordered plasma membrane environments; ordered plasma membrane environments appear to be essential for binding of Wnt (zeige WNT2 Antikörper) proteins to their receptor complexes and stimulation of downstream signaling activity.
It was shown that Wnt3a-Wnt8a (zeige WNT8A Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signaling directly regulates ciliogenic transcription factor foxj1a expression and ciliogenesis in zebrafish Kupffer's vesicle.
In zebrafish embryos lacking Wnt3a, Wnt1 (zeige WNT1 Antikörper) and Wnt10b (zeige WNT10B Antikörper), the expression of engrailed orthologs, pax2a and fgf8 (zeige FGF8 Antikörper) is not maintained after mid-somitogenesis
data suggest a specific role for Wnt3a in the development of cardiac NCCs; propose that this function of wnt3a in r6 is partially mediated by crip2 (zeige CRIP2 Antikörper) expression in the premigratory cardiac NCCs, which subsequently affects cardiac function and PA patterning
Sulf1 (zeige SULF1 Antikörper) does not affect Wnt3a-mediated activation of canonical Wnt (zeige WNT2 Antikörper) signaling.
Wnt3a protein alone is sufficient to rescue the severe loss of inner ear structures resulting from dorsal but not ventral half ablations.
hindbrain-repressive Wnt3a/Meis3/Tsh1 circuit promotes neuronal differentiation and coordinates tissue maturation
Wnt3a thus acts downstream of FAK (zeige PTK2 Antikörper) to balance anterior-posterior cell fate specification in the developing neural plate
Data suggest a new model for neural anteroposterior patterning, in which Wnt3a from the paraxial mesoderm induces posterior cell fates via direct activation of a crucial transcription factor in the overlying neural plate.
Wnt3A secretion from tectal cells along with ephrin-B1 (zeige EFNB1 Antikörper) signaling are specifically responsible for enhanced neural responses in the developing optic tectum.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It encodes a protein which shows 96% amino acid identity to mouse Wnt3A protein, and 84% to human WNT3 protein, another WNT gene product. This gene is clustered with WNT14 gene, another family member, in chromosome 1q42 region.
wingless-type MMTV integration site family, member 3A
, protein Wnt-3a-like
, protein Wnt-3a
, vestigial tail
, wingless-type MMTV integration site family, member 3 like
, wnt3 like
, wingless-type MMTV integration site family, member 3
, Wnt-3a homolog
, Wnt3a variant 3
, wingless-type MMTV integration site family member 3a
, wingless-related MMTV integration site 3A