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Human WNT1 ELISA Kit für Sandwich ELISA - ABIN572409
Lii, Chang, Chen, Chen, Liu, Yeh, Wang, Liu, Tsai, Li: Docosahexaenoic acid inhibits 12-O-tetradecanoylphorbol-13- acetate-induced fascin-1-dependent breast cancer cell migration by suppressing the PKCδ- and Wnt-1/β-catenin-mediated pathways. in Oncotarget 2016
maternal Wnt (zeige WNT2 ELISA Kits)/STOP signaling, but not beta-catenin (zeige CTNNB1 ELISA Kits) signaling, has a role in cleavage after fertilization and cell cycle progression
sfrp1 (zeige SFRP1 ELISA Kits) promotes cardiomyocyte differentiation in Xenopus via negative-feedback regulation of Wnt (zeige WNT2 ELISA Kits) signalling.
analysis of differential role of Axin (zeige AXIN1 ELISA Kits) RGS (zeige PITX2 ELISA Kits) domain function in Wnt (zeige WNT2 ELISA Kits) signaling during anteroposterior patterning and maternal axis formation
Data show taht combined Wnt (zeige WNT2 ELISA Kits) and Nodal signaling synergistically activates transcription of Spemann organizer genes.
The authors propose that these dual functions of DP1 (zeige TFDP1 ELISA Kits) can promote and stabilize biphasic Wnt (zeige WNT2 ELISA Kits)-on and Wnt (zeige WNT2 ELISA Kits)-off states in response to a gradual gradient of Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signalling to determine differential cell fates.
Pax9 (zeige PAX9 ELISA Kits)-dependent Wnt (zeige WNT2 ELISA Kits) signaling has a role in palatogenesis and cleft palates
Data show that autocrine Wnt (zeige WNT2 ELISA Kits) secretion is important for the survival, chromosomal stability, differentiation, and tumorigenic potential of embryonic stem cells (ESCs (zeige NR2E3 ELISA Kits)).
Results demonstrated functional differences in the molecular mechanisms downstream of Wnt1 function in the diencephalon, in relation to the spinal cord. Wnt1 signal determines the patterning of the diencephalic dorso-ventral axis
Data show that both transgenic Wnt1-cre and P0-cre are similarly effective in deleting beta-catenin (zeige CTNNB1 ELISA Kits) in the neural crest.
data suggest that WNT1-related osteogenesis imperfecta (zeige COL1A2 ELISA Kits) and osteoporosis are caused in part by decreased mTORC1-dependent osteoblast function resulting from loss of WNT1 signaling in osteocytes.
Administration of EET alters Wnt1, NOV (zeige NOV ELISA Kits), and HO-1 (zeige HMOX1 ELISA Kits) signaling to prevent obesity-induced cardiomyopathy in obese mice.
Data indicate that Wnt1 proto-oncogene (zeige RAB1A ELISA Kits) protein (WNT1) is the direct target of microRNA miR (zeige MLXIP ELISA Kits)-34a in dendritic cell (DC).
In order to evaluate the function of IFT88 (zeige IFT88 ELISA Kits) in regulating craniofacial development, we generated Wnt1-Cre;Ift88fl/fl mice to eliminate Ift88 (zeige IFT88 ELISA Kits) specifically in cranial neural crest (CNC) cells. Wnt1-Cre;Ift88fl/flpups died at birth due to severe craniofacial defects including bilateral cleft lip and palate and tongue agenesis, following the loss of the primary cilia in the CNC-derived palatal mesenchyme
Wnt (zeige WNT2 ELISA Kits), Eda (zeige EDA ELISA Kits), and Shh (zeige SHH ELISA Kits) have roles in touch dome Merkel cell development
The data obtained from the 14-3-3epsilon/14-3-3zeta (zeige YWHAZ ELISA Kits)/Wnt1-Cre mice strongly indicate the importance of 14-3-3 (zeige YWHAQ ELISA Kits) proteins in the development of melanocyte lineages.
High WNT1 expression in stromal cells is associated with myelodysplastic syndrome.
High WNT1 expression is associated with gastric cancer.
Novel compound heterozygous WNT1 missense mutations, p.Glu123Asp and p.Cys153Gly, identified in a patient with osteogenesis imperfecta (zeige COL1A2 ELISA Kits).
Osteocyte protein expression is altered in patients with osteoporosis caused by WNT1 mutation.
High WNT1 expression is associated with glioma cell invasion.
bone formation is under the control of WNT1 produced by osteocytes, the cells that reside deep in the bone matrix and form dendritic networks.
Knocking down of Wnt-1 by siRNA had the similar effect of miRNA-148a overexpression on cell migration and invasion in lung cancer cells.
High WNT1 expression is associated with Lung Adenocarcinoma Progression.
WNT1 overexpression is associated with Breast Cancer Invasion and Metastasis.
PKA signaling is pivotal in pigmentation process itself in cultured melanoma cells, while the importance of Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling should be emphasized in the context of development and differentiation
Data indicate that Wnt-1 protein is present in postdevelopmental endothelial cells where it associates with cytoskeletal elements and may retain function as a tissue polarity gene.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
wnt1 and wnt10b (zeige WNT10B ELISA Kits) are required to maintain threshold levels of Pax2.1 and Fgf8 (zeige FGF8 ELISA Kits) at the midbrain-hindbrain boundary.
novel role for Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signalling in determining endocardial cell fate
In zebrafish embryos lacking Wnt3a (zeige WNT3A ELISA Kits), Wnt1 and Wnt10b (zeige WNT10B ELISA Kits), the expression of engrailed orthologs, pax2a and fgf8 (zeige FGF8 ELISA Kits) is not maintained after mid-somitogenesis
two Dvl (zeige DVL2 ELISA Kits)-associated paralogs, Dpr1 (zeige DACT1 ELISA Kits) and Dpr2, participate in distinct Wnt (zeige WNT2 ELISA Kits)-dependent developmental processes
Wnt (zeige WNT2 ELISA Kits)/Axin (zeige AXIN1 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) pathway has a role in ventral CNS development
The boundary and roof plate expression of wnt1 each contribute to upregulation of proneural and delta gene expression and neurogenesis in non-boundary regions.
Epistatic analyses suggest a possible genetic interaction between Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) and Myostatin (zeige MSTN ELISA Kits) in regulation of slow and fast twitch muscle myofibrillogenesis
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It is very conserved in evolution, and the protein encoded by this gene is known to be 98% identical to the mouse Wnt1 protein at the amino acid level. The studies in mouse indicate that the Wnt1 protein functions in the induction of the mesencephalon and cerebellum. This gene was originally considered as a candidate gene for Joubert syndrome, an autosomal recessive disorder with cerebellar hypoplasia as a leading feature. However, further studies suggested that the gene mutations might not have a significant role in Joubert syndrome. This gene is clustered with another family member, WNT10B, in the chromosome 12q13 region.
, protein Wnt-1
, proto-oncogene Int-1
, proto-oncogene Wnt-1
, proto-oncogene protein Wnt-1
, proto-oncogene Int-1 homolog
, wingless-type MMTV integration site family, member 1 (oncogene INT1)
, Wingless-type MMTV integration site 1 homolog
, Wingless-type MMTV integration site 1, homolog
, wingless-related MMTV integration site 1
, murine mammary tumor virus integration site
, wingless-type MMTV integration site family member 1