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anti-Human ACP5 Antikörper:
anti-Rat (Rattus) ACP5 Antikörper:
anti-Mouse (Murine) ACP5 Antikörper:
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Human Polyclonal ACP5 Primary Antibody für WB - ABIN513079
Lozano, Fernández-de-Castro, Portal-Núñez, López-Herradón, Dapía, Gómez-Barrena, Esbrit: The C-terminal fragment of parathyroid hormone-related peptide promotes bone formation in diabetic mice with low-turnover osteopaenia. in British journal of pharmacology 2011
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Human Polyclonal ACP5 Primary Antibody für WB - ABIN1881042
Shih, Janckila, Kwok, Ho, Chou, Chao: Effects of exercise on insulin sensitivity, inflammatory cytokines, and serum tartrate-resistant acid phosphatase 5a in obese Chinese male adolescents. in Metabolism: clinical and experimental 2009
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Human Polyclonal ACP5 Primary Antibody für WB - ABIN513078
Leung, Pickarski, Zhuo, Masarachia, Duong: The effects of the cathepsin K inhibitor odanacatib on osteoclastic bone resorption and vesicular trafficking. in Bone 2011
Human Monoclonal ACP5 Primary Antibody für FACS, ELISA - ABIN1724847
Janckila, Lin, Wu, Ku, Yang, Lin, Lee, Yam, Chao: Serum tartrate-resistant acid phosphatase isoform 5a (TRACP5a) as a potential risk marker in cardiovascular disease. in Clinica chimica acta; international journal of clinical chemistry 2011
Nidogen-2, a prote (zeige NID2 Antikörper)in shown to be expressed intracellularly and for secretion by pre-adipocytes, was shown to interact, through its globular G3 domain, with TRAP 5a in vitro.
Tartrate-resistant acid phosphatase deficiency in plasmacytoid dendritic cells leads to increased IFNalpha production, providing at least a partial explanation for how ACP5 mutations cause lupus in the context of spondyloenchondrodysplasia. Detection of ACP5 missense variants in a lupus cohort suggests that impaired tartrate-resistant acid phosphatase functioning may increase susceptibility to sporadic lupus
Biallelic ACP5 mutations are associated with autoimmune cytopenias.
Severe short stature can be the only presenting sign of ACP5 deficiency and the latter could therefore be considered as a rare cause in the differential diagnosis of severe, proportionate growth failure.
Spondyloenchondrodysplasia (SPENCD) is a rare autosomal recessive skeletal dysplasia caused by recessive mutations in the ACP5 gene, and it is characterized by the persistence of chondroid tissue islands within the bone. [review]
Data show that the levels of serum vascular endothelial growth factor (VEGF) and tartrate resistant acid phosphatase (TRacp-5b) are higher in multiple myeloma with cytogenetic abnormalities.
A diverse spectrum of spondyloenchondrodysplasia phenotypes due to mutations in ACP5 has been presented.
We identified five new biomarkers: GDF15 (zeige GDF15 Antikörper), osteonectin (zeige SPARC Antikörper), TRAP5, TWEAK (zeige TNFSF12 Antikörper), and YKL40 (zeige CHI3L1 Antikörper) as being promising markers for monitoring bone metastases.
Trap-5b is overexpressed in renal cell carcinoma (zeige MOK Antikörper) patients with bone metastasis and bone resorption.
Serum levels of NTx and TRACP5b are sensitive and simple biomarkers to indicate aberrant bone metabolism in giant cell tumor of bone, and they may have a clinical significance in GCT (zeige QPCT Antikörper) diagnosis.
TRAP and nidogen-2 (zeige NID2 Antikörper) co-localized in adipose tissue cells in situ.
Uteroferrin is a cytokine secreted by uterine glands in response to progesterone that promotes fetal erythropoiesis at various stages of pregnancy.
Supraphysiologic oscillatory fluid shear induces upregulation of osteoclastic activity, measured by tartrate-resistant acid phosphatase activity and formation of mineral resorption pits.
Pax6 (zeige PAX6 Antikörper) binds endogenously to the proximal region of the tartrate acid phosphatase (TRAP) gene promoter and suppresses nuclear factor of activated T cells c1 (NFATc1 (zeige NFATC1 Antikörper))-induced TRAP gene expression.
tartrate resistant acid phosphatase
dephosphorylation of Man6P-containing lysosomal proteins requires the concerted action of Acp2 (zeige ACP2 Antikörper) and Acp5 and is needed for hydrolysis and removal of degradation products
Accumulation of vesicles in the cytoplasm of mutant osteoclasts indicates a novel function for tartrate-resistant acid phosphatase in modulating intracellular vesicular transport in osteoclasts
USF1 (zeige USF1 Antikörper) and USF2 (zeige USF2 Antikörper) play a functional role in RANKL (zeige TNFSF11 Antikörper)-dependent TRAP expression during osteoclast differentiation.
TRACP expression is a target for regulation by the macrophage/osteoclast transcription factor PU.1 and the osteoclast commitment factor MiTF (zeige MITF Antikörper)
YY1 (zeige YY1 Antikörper) plays a functional role in RANKL (zeige TNFSF11 Antikörper)-mediated TRAP gene expression during osteoclast differentiation.
characterization and proteolytic analysis of uteroferrin
Cows with a low tartrate-resistant acid phosphatase (TRAP) activity are at risk of developing milk fever in comparison to cows with high TRAP activity.
This study reported a fluorometric method for measuring tartrate-resistant acid phosphatase isoform 5b and reported on the activity in the blood of cattle through development.
This gene encodes an iron containing glycoprotein which catalyzes the conversion of orthophosphoric monoester to alcohol and orthophosphate. It is the most basic of the acid phosphatases and is the only form not inhibited by L(+)-tartrate.
acid phosphatase 5, tartrate resistant
, tartrate-resistant acid phosphatase type 5
, acp5 protein
, tartrate resistant acid phosphatase
, Tartrate-resistant acid phosphatase type 5
, tartrate-resistant acid ATPase
, acid phosphatase type V
, type 5 acid phosphatase
, acid phosphatase type 5
, tartrate-resistant acid phosphatase