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Genetic variability in DNM3 modifies age of onset for LRRK2 (zeige LRRK2 ELISA Kits) Gly2019Ser parkinsonism.
DNM3 may be involved in risk of obsessive-compulsive disorder.
Microdeletion of DNM3 harboring miR199 and miR214 is associated with skeletal abnormalities.
DNM3 attenuates the proliferation and induces apoptosis of gastric cancer cells.
Data show that the classical dynamin DNM1 (zeige DNM1 ELISA Kits) and DNM3 genes reach their maximum expression levels (100% of maximal expression) in all normal central nervous system tissues studied.
Data indicate that dynamin 3 (DNM3) harbors MEIS1 (zeige MEIS1 ELISA Kits) binding sites and is associated with differences in mean platelet volume (MPV).
DNM3 not only participates in megakaryocyte progenitor amplification, but is also involved in cytoplasmic enlargement and the formation of the demarcation membrane system.
Dynamin (zeige DNM1 ELISA Kits) GTPase (zeige RACGAP1 ELISA Kits) regulation is altered by PH domain mutations found in centronuclear myopathy patients.
dynamin-3 and the postsynaptic adaptor Homer (zeige HOMER1 ELISA Kits) positions the endocytic zones near the postsynaptic density
Upon comparison of PBMC and skin samples of Sezary syndrome versus mycosis fungoides, CDO1 (zeige CDO1 ELISA Kits) and DNM3 were found upregulated only in Sezary syndrome.
The data of this study demonstrated that dynamin 3 is required for the formation, functional maturation, and subsequent survival of the calyx of Held.
In the developing neurons, there is a dynamin 1-, dynamin 3- and clathrin-independent pathway of synaptic vesicle recycling mediated by bulk endocytosis.
As low-level expression of the dynamin 3 gene in these cells could not be excluded, we have now engineered dynamin 1 (zeige DNM1 ELISA Kits), 2 and 3 triple KO (TKO (zeige MRPS12 ELISA Kits)) fibroblasts.
Data indicate that coxsackievirus and adenovirus receptor (zeige CXADR ELISA Kits) (CAR) internalization was lipid microdomain-, actin-, and dynamin (zeige DNM1 ELISA Kits)-dependent, and subsequently followed by CAR degradation in lysosomes.
Data indicate that dynamin (zeige DNM1 ELISA Kits), clathrin, adaptor protein complex-2 (AP2 (zeige TFAP2A ELISA Kits)), and amphiphysin (zeige AMPH ELISA Kits) contribute to the depolarization-evoked entry in endocytosis.
These findings reveal a mechanism aimed at preventing synaptic transmission failure due to vesicle depletion when recycling vesicle traffic is backed up by a defect in dynamin (zeige DNM1 ELISA Kits)-dependent endocytosis.
Lack of dynamin 3 does not produce an overt phenotype in mice; however, it worsens the dynamin 1 (zeige DNM1 ELISA Kits) KO phenotype, leading to perinatal lethality and a more severe defect in activity-dependent synaptic vesicle endocytosis.
Dynamin 3 participates in the growth and development of megakaryocytes.
Members of the dynamin family, such as DNM3, possess mechanochemical properties involved in actin-membrane processes, predominantly in membrane budding (Orth and McNiven, 2003
, dynamin family member
, dynamin, testicular
, Dyna III
, Dynamin III
, testicular dynamin