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Human TLR9 ELISA Kit für Sandwich ELISA - ABIN414673
Yang, Xie, Deng, Qin: Expression of soluble Toll-like receptors in pleural effusions. in Chinese medical journal 2010
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Rat (Rattus) TLR9 ELISA Kit - ABIN368321
Tunc, Aydemir, Karaoglu, Cekmez, Kul, Aydinoz, Babacan, Yaman, Sarici: Toll-like receptor levels and caffeine responsiveness in rat pups during perinatal period. in Regulatory peptides 2013
The present study investigated the effects of vitamin D3 on the expression of TLR3 (zeige TLR3 ELISA Kits), TLR7 (zeige TLR7 ELISA Kits), and TLR9 in Systemic lupus erythematosus patients.
There appears to be a defect of TLR 9 signaling in patients with tuberculosis, the nature of which needs to be further explored.
sickle red blood cell induced TLR9, NLRP3 (zeige NLRP3 ELISA Kits), Caspase-1 (zeige CASP1 ELISA Kits), IL-1beta (zeige IL1B ELISA Kits) and IL-18 (zeige IL18 ELISA Kits) expression and induced IL-1beta (zeige IL1B ELISA Kits), LTB4 (zeige PTGR1 ELISA Kits) and nitrite production in PBMC cultures.
Findings indicate that genotype TT in the -1486 locus of the TLR9 gene could comprise a risk genotype for HPV infection, squamous intraepithelial cervical lesion, and uterine cervical neoplasm in Mexican female population.
data show that TLR-5 (zeige TLR5 ELISA Kits) and TLR-9 are susceptible genes to lupus nephritis (LN) and that their expression is dysregulated in LN patients' kidneys, supporting a role of these mediators in the pathogenesis of LN.
By CRISPR/Cas9-induced inactivation of TLR9, MyD88 (zeige MYD88 ELISA Kits), IRAK4 (zeige IRAK4 ELISA Kits) and IRAK1 (zeige IRAK1 ELISA Kits) we confirm that BZLF1 repression is dependent on functional TLR9 and MyD88 (zeige MYD88 ELISA Kits) signaling, and identify IRAK4 (zeige IRAK4 ELISA Kits) as an essential element for TLR9-induced repression of BZLF1 expression upon BCR (zeige BCR ELISA Kits) cross-linking
results imply that TLR9-mediated activation of B-cells not only promotes cell survival, but may via p53 (zeige TP53 ELISA Kits) provide cells with a barrier against harmful consequences of enhanced activation and proliferation
mutually exclusive transcriptional regulation by AP-1 (cjun (zeige JUN ELISA Kits)/cfos) and non-canonical NF-kappaB (zeige NFKB1 ELISA Kits) (RelB (zeige RELB ELISA Kits)/p52 (zeige FKBP4 ELISA Kits)) downstream of MEK (zeige MAP2K1 ELISA Kits)-ERK (zeige EPHB2 ELISA Kits) and NIK (zeige MAP3K14 ELISA Kits)-IKK-alpha (zeige CHUK ELISA Kits)-NF-kappaB2 (p100 (zeige CUX1 ELISA Kits)) phosphorylation, respectively was responsible for persistent Ccl20 (zeige CCL20 ELISA Kits) expression in the colonic cells.
This study evaluation the roles of SOCS1 (zeige SOCS1 ELISA Kits), the regulator of TLR9, RIG-I (zeige DDX58 ELISA Kits), and CD152 (zeige CTLA4 ELISA Kits) in patients with liver fibrosis/cirrhosis; the use of polymorphisms as markers for genetic risk is reported.
cathelicidin selectively modulated synthesis of TLR4 (zeige TLR4 ELISA Kits) and 9 in intestinal epithelium, but only when cells were exposed to virulence factors, mostly from apical surfaces.
TLR9 expression on B1a cells is not critical for their IgM-dependent atheroprotection.
This study establishes a correlation between TLR-3 (zeige TLR3 ELISA Kits) and TLR-9 expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
these data demonstrate that TLR7 (zeige TLR7 ELISA Kits)/TLR9 ligands push the macrophage into a phagocytic long-lived cell, with a decreased capacity of antigen presentation and reminiscent of the M2 polarized state.
TLR9/MyD88 (zeige MYD88 ELISA Kits) signaling selectively in CD11c (zeige ITGAX ELISA Kits)(+) dendritic cells (DCs) strongly enhances murine cytomegalovirus clearance.
High-glucose induces tau hyperphosphorylation through activation of TLR9-P38 MAPK (zeige MAPK14 ELISA Kits) pathway.
proposed that TLR9 regulates the NF-kappaB (zeige NFKB1 ELISA Kits)-NLRP3 (zeige NLRP3 ELISA Kits)-IL-1beta (zeige IL1B ELISA Kits) pathway negatively in Salmonella-induced NKG2D (zeige KLRK1 ELISA Kits)-mediated intestinal inflammation and plays a critical role in defense against S. typhimurium infection and in the protection of intestinal integrity.
mechanism integrating BCR, TLR9, and cytokine signals provides a peripheral checkpoint for DNA-containing antigens that, if circumvented by survival and differentiative cues, yields B cells with the autoimmune-associated T-bet+ phenotype.
we demonstrated for the first time that the cross-talk of TLR2 (zeige TLR2 ELISA Kits) and TLR9 triggered Th1 (zeige HAND1 ELISA Kits) activation collaboratively
the cooperative role of TLR9 with TLR2 (zeige TLR2 ELISA Kits) or TLR6 (zeige TLR6 ELISA Kits) receptors in sensing Brucella, was determined.
accelerating effects of Tlr9 deficiency
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (zeige TLR2 ELISA Kits), TLR3 (zeige TLR3 ELISA Kits) and TLR9 contribute to NF-kappaB (zeige NFKB1 ELISA Kits) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (zeige DDX58 ELISA Kits).
expression of TLR3 (zeige TLR3 ELISA Kits), TLR7 (zeige TLR7 ELISA Kits) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (zeige TLR1 ELISA Kits) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2 (zeige TLR2 ELISA Kits), and TLR4 (zeige TLR4 ELISA Kits) together with those of beta-defensin 5 (BNBD5 (zeige DEFB105A ELISA Kits)), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein