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increased expression of NF-YA may promote a malignant phenotype in OS cells via modulation of FASN (zeige FASN ELISA Kits) expression.
The findings indicate that overexpression of NF-YA contributes to tumor angiogenesis through EZH2 (zeige EZH2 ELISA Kits)-STAT3 (zeige STAT3 ELISA Kits) signaling in human melanoma cells, highlighting NF-YA as a potential therapeutic target in human melanoma.
Data indicate that specific cancer-driving nodes are generally under NF-YA/B control.
our results indicate that NF-YA alternative splicing is an influential muscle cell determinant, through direct regulation of selected cell cycle blocking genes, and, directly and indirectly, of muscle-specific (zeige EIF3K ELISA Kits) transcription factors
enhanced CDCA8 (zeige CDCA8 ELISA Kits) promoter activities by NF-Y overexpression and reduced CDCA8 (zeige CDCA8 ELISA Kits) transcription by NF-Y knockdown further verified that NF-Y is a positive regulator of CDCA8 (zeige CDCA8 ELISA Kits) transcription.
Data show that Zinc-fingers and homeoboxes 2 (ZHX2 (zeige ZHX2 ELISA Kits)) represses nuclear transcription factor Y alpha (NF-Y)-mediated activation of multidrug resistance 1 (MDR1) transcription.
NF-Y inhibits NT2/D1 cell growth in p53 (zeige TP53 ELISA Kits)-independent manner and decreases SOX2 (zeige SOX2 ELISA Kits) expression.
Stimulation of the lymphocytes with phytohemagglutinin, restores normal OGG1 (zeige OGG1 ELISA Kits) levels and repair rates. MAP kinase (zeige MAPK1 ELISA Kits) c-Jun N-terminal kinase (JNK) and the recruitment of the transcription factor NFYA to the promoter region of OGG1 (zeige OGG1 ELISA Kits) are shown to be involved.
Association of p21 (zeige CDKN1A ELISA Kits) with NF-YA suppresses the expression of PLK1 (zeige PLK1 ELISA Kits) and prevents mitotic death in response to DNA damage.
NF-YA binds directly over the CCAAT sequence.
Interaction of C/EBP-beta (zeige CEBPB ELISA Kits) and NF-Y factors constrains activity levels of the nutritionally controlled promoter IA expressing the acetyl-CoA carboxylase-alpha (zeige ACACA ELISA Kits) gene in cattle.
Data show that nuclear factor Y (NF-Y) binds to the inverted CCAAT element (ICE) in the Fatty acid synthase (Fasn (zeige FASN ELISA Kits)) promoter specifically in refeeding states.
findings may point to a possible role of NF-YA in stress conditions that occur in chronic obesity, ER stress might be involved in the pathogenesis of obesity through NF-YA depletion.
we conclude that NF-Y and YY1 (zeige YY1 ELISA Kits) are important for the basal transcription of Pcyt2 (zeige PCYT2 ELISA Kits) and that NF-Y is involved in the inhibitory effects of 25-HC on Pcyt2 (zeige PCYT2 ELISA Kits) transcription.
these results indicate that the transcription factor NF-Y regulates the proximal promoter activity of mouse Col11a1 (zeige COL11A1 ELISA Kits) gene in chondrocytes
these results raise an exciting possibility that targeting CDK1 (zeige CDK1 ELISA Kits) or NF-Y in the diseased heart may inhibit fibrosis and subsequently confer cardioprotection.
NF-Ya is a functional activator of Bmal1 (zeige ARNTL ELISA Kits) transcription and it cooperates with Sp1 (zeige SP1 ELISA Kits) and Rev-Erbalpha (zeige NR1D1 ELISA Kits) to generate the daily cycle of Bmal1 (zeige ARNTL ELISA Kits) expression.
Sp1 (zeige SP1 ELISA Kits), CREB (zeige CREB1 ELISA Kits), HNF-1 (zeige HNF1A ELISA Kits), and NF-Y, known to be responsive to hormones and diet, regulate NAGS (zeige NAGS ELISA Kits) transcription
results indicate nuclear transcription factor NF-Y as a pivotal upstream participant in a regulatory network necessary for the preservation of cycling hematopoietic stem cell.
Findings establish that NF-Y acts upstream of E2F1 (zeige E2F1 ELISA Kits) in p53 (zeige TP53 ELISA Kits)-mediated apoptosis.
Transcriptional regulation of myeloid differentiation primary response (MyD) genes during myeloid differentiation is mediated by nuclear factor Y.
The protein encoded by this gene is one subunit of a trimeric complex, forming a highly conserved transcription factor that binds to CCAAT motifs in the promoter regions in a variety of genes. Subunit A associates with a tight dimer composed of the B and C subunits, resulting in a trimer that binds to DNA with high specificity and affinity. The sequence specific interactions of the complex are made by the A subunit, suggesting a role as the regulatory subunit. In addition, there is evidence of post-transcriptional regulation in this gene product, either by protein degradation or control of translation. Further regulation is represented by alternative splicing in the glutamine-rich activation domain, with clear tissue-specific preferences for the two isoforms.
, nuclear transcription factor Y, alpha
, CAAT box DNA-binding protein subunit A
, CAAT-box DNA binding protein subunit A
, CCAAT-binding transcription factor subunit B
, HAP2 CCAAT-binding protein
, Transcription factor NF-Y, A subunit
, nuclear transcription factor Y subunit A
, nuclear transcription factor Y subunit alpha
, CAAT-box DNA-binding protein subunit A
, NF-Y protein chain A
, nuclear transcription factor-Y alpha