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anti-Human REST Antikörper:
anti-Mouse (Murine) REST Antikörper:
anti-Rat (Rattus) REST Antikörper:
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Human Polyclonal REST Primary Antibody für ICC, IF - ABIN152175
Kashiwagi, Ishii, Sakaeda, Arimasu, Shimoyamada, Sato, Miyata, Kamma, Aoki, Yazawa: Differences of molecular expression mechanisms among neural cell adhesion molecule 1, synaptophysin, and chromogranin A in lung cancer cells. in Pathology international 2012
Findings indicate that RE1 (zeige NPC2 Antikörper) silencing transcriptional factor (REST) regulates its miR (zeige MLXIP Antikörper)-124 and mir (zeige MLXIP Antikörper)-203 gene and suggest how REST maintains oncogenic competence in glioblastoma (GBM)stem cells. These mechanisms could potentially be utilized to block REST-mediated GBM tumorigenesis.
findings indicate that SCYL1 (zeige CCL13 Antikörper) does not contribute to REST turnover and thus do not support a previous study suggesting a role for SCYL1 (zeige CCL13 Antikörper) in mediating REST degradation
the mechanisms of inhibition of the small-molecule drugs 4SC-202 and SP2509 were analyzed. These drugs interfered with the viability of medulloblastoma cells, where REST/NRSF has been implicated in cancer pathogenesis. Thus, a resource for molecular REST/NRSF studies and drug development has been established.
Expressions of REST and RCOR1 (zeige RCOR1 Antikörper) genes may downregulate SYN1 (zeige SYN1 Antikörper) expression in gliomas.
REST represses transcription of HIF-1alpha (zeige HIF1A Antikörper) in prolonged hypoxia, thus contributing to the resolution of the HIF-1alpha (zeige HIF1A Antikörper) response.
A role was determined for the NRSF-BDNF (zeige BDNF Antikörper) pathway in the modulation of cognitive function in patients with newly diagnosed epilepsy.
The splicing of REST by SRRM4 (zeige SRRM4 Antikörper) could promote the neuroendocrine phenotype in CRPC.
TSPYL2 (zeige TSPYL2 Antikörper) is an essential component of the REST/NRSF transcriptional complex for TGFbeta (zeige TGFB1 Antikörper) signaling activation
Cultured astrocytes gradually became refractory to reprogramming, in part by the repressor REST preventing Neurog2 (zeige NEUROG2 Antikörper) from binding to the NeuroD4 (zeige NEUROD4 Antikörper) promoter.
identified and validated a novel promoter of the MIR137 gene adjacent to miR (zeige MLXIP Antikörper)-137 itself which can direct the expression of distinct mRNA isoforms encoding miR (zeige MLXIP Antikörper)-137; internal promoter is regulated by repressor element-1 silencing transcription factor (REST)
Findings provide evidence for the loss of globin-linked neuroprotection to be linked to Alzheimer's disease angiopathogenesis, while raising the possibility of REST/ NRSF being an upstream regulator.
a new paradigm of transcriptional regulation critical for cardiac development and maturation that is controlled by the interaction of REST, DNMT3B (zeige DNMT3B Antikörper) and non-CpG methylation.
Data indicate that levels of repressor element-1 silencing transcription factor (REST) are elevated in polyubiquitin-B (zeige UBB Antikörper) knockout (Ubb (zeige UBA52 Antikörper) -/-) cells.
Results identified REST as a novel target gene that was negatively regulated by miR (zeige MLXIP Antikörper)-218 in cardiomyocytes hypertrophy process.
Detailed analysis of specific lineage markers expression showed selective downregulation of endoderm markers in REST-null cells, thus contributing to a loss of cardiogenic signals. REST regulates cardiac differentiation of ESCs (zeige NR2E3 Antikörper) by negatively regulating the Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signaling pathway and positively regulating the cardiogenic TF Gata4 (zeige GATA4 Antikörper)
The transcriptional repressor REST represses miR (zeige MLXIP Antikörper)-21 and, thus, regulates self-renewal in E14Tg2a.4 mESCs cultured in the absence of mouse embryonic fibroblast feeder cell effects.
Data suggest that Rest regulates early osteoblast differentiation via modulating Rest expression that is independent of Osx (zeige SP7 Antikörper) expression.
REST expression in nerve injury is decreased following alternative splicing which in turn controls PACAP (zeige ADCYAP1 Antikörper) expression.
We propose a mechanism for transcriptional activation in neurons that involves REST-guided targeting of TET3 (zeige TET3 Antikörper) to the DNA for directed 5hmC generation and NSD3 (zeige WHSC1L1 Antikörper)-mediated H3K36 trimethylation
This study demostrated that maternal rest plays a long-term role in regulation of gene expression through snap25a/b and behavior during development.
The transcriptional repressor rest modulates hedgehog (zeige SHH Antikörper) signaling.
This gene encodes a transcriptional repressor that represses neuronal genes in non-neuronal tissues. It is a member of the Kruppel-type zinc finger transcription factor family. It represses transcription by binding a DNA sequence element called the neuron-restrictive silencer element. The protein is also found in undifferentiated neuronal progenitor cells and it is thought that this repressor may act as a master negative regular of neurogenesis. Alternatively spliced transcript variants have been described
neural-restrictive silencer factor
, neuron restrictive silencer factor
, repressor binding to the X2 box
, zinc finger transcription factor REST protein
, RE1-silencing transcription factor
, RE-1 silencer of transcription