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Involved in transcriptional regulation. Zusätzlich bieten wir Ihnen Zinc Finger Protein 148 Proteine (4) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal ZNF148 Primary Antibody für WB - ABIN1882015
Udelhoven, Pasieka, Leeser, Krone, Schubert: Neuronal insulin receptor substrate 2 (IRS2) expression is regulated by ZBP89 and SP1 binding to the IRS2 promoter. in The Journal of endocrinology 2010
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Human Polyclonal ZNF148 Primary Antibody für ICC, IF - ABIN4367002
Sayin, Nilton, Ibrahim, Ågren, Larsson, Petit, Hultén, Ståhlman, Johansson, Bergo, Lindahl: Zfp148 deficiency causes lung maturation defects and lethality in newborn mice that are rescued by deletion of p53 or antioxidant treatment. in PLoS ONE 2013
Cow (Bovine) Polyclonal ZNF148 Primary Antibody für WB - ABIN2775856
Chupreta, Brevig, Bai, Merchant, Iñiguez-Lluhí: Sumoylation-dependent control of homotypic and heterotypic synergy by the Kruppel-type zinc finger protein ZBP-89. in The Journal of biological chemistry 2007
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ZNF148 as a gene involved in a newly described intellectual disability syndrome with a recurrent phenotype and postulate that the ZNF148 is a hitherto unrecognized but crucial transcription factor in the development of the corpus callosum.
A larger role ZBP-89 plays in gene regulation during inflammation.ZBP-89 regulates of MMP-1 (zeige MMP1 Antikörper) expression.ZBP-89 and NF-kappaB (zeige NFKB1 Antikörper) appear to bind cooperatively on both promoters.
ZBP-89 attenuates HDAC3 (zeige HDAC3 Antikörper) by increasing IkappaB degradation, dependent on Pin1 (zeige PIN1 Antikörper) but independent of NF-Kappab (zeige NFKB1 Antikörper)
zinc-binding protein (zeige PTMS Antikörper)-89 upregulates the expression of Bak (zeige BAK1 Antikörper) by targeting multiple components of the epigenetic pathway in hepatocellular carcinoma.
lower expression of ZNF148 in colorectal cancer was significantly associated with worse clinicopathologic variables, including lymph node metastases, poor differentiation, and a higher rate of disease recurrence
this study provides vigorous evidence that ZBP-89 was significantly downregulated in clear cell renal cell carcinoma (zeige MOK Antikörper)(CCRCC) and could be served as a promising biomarker for prediction of distal metastasis and prognosis of patient with CCRCC.
Colony formation was reduced dramatically in those hepatocellular carcinoma cell lines in which ZBP-89 overexpression was demonstrated; this appeared to correlate with increased apoptosis.
We found that the TP53 (zeige TP53 Antikörper)-G245D variant but not TP53 (zeige TP53 Antikörper)-R249S abrogated HDAC (zeige HDAC3 Antikörper) inhibitor p21 (zeige CDKN1A Antikörper) induction by binding to ZBP-89 and retaining it in the cytoplasm.
ZBP-89 is a regulator of Pdcd4 (zeige PDCD4 Antikörper) gene, binding to the basal promoter either alone or by interacting with Sp family members.
An activating role for ZBP-89 in human globin gene regulation and erythroid differentiation.
Study demonstrated that miR (zeige MLXIP Antikörper)-20b suppressed ZFP-148 expression by directly binding to the 3'-UTR (zeige UTS2R Antikörper) of target mRNA and repressed translation at posttranscriptional level during virus myocarditis.
Zfp148 deficiency increases p53 (zeige TP53 Antikörper) activity and protects against atherosclerosis by causing proliferation arrest of lesional macrophages.
These data identify an important role for ZBP-89 in regulating stress hematopoiesis in adult mouse bone marrow.
Results indicate that Zfp148-deficient mice generate normal mature hematopoietic populations.
Zfp148 deficiency causes lung maturation defects and lethality.
ZBP-89 is required for expression of the Tph1 (zeige TPH1 Antikörper) gene and subsequent production of 5HT (zeige DDC Antikörper) in response to bacterial infection in mice. Reductions in epithelial ZBP-89 increase susceptibility to colitis and sepsis after infection with S. typhimurium.
MiR (zeige MLXIP Antikörper)-203 expression downregulated ZFP-148 mRNA translation by specifically targeting its 3' UTR (zeige UTS2R Antikörper), which resulted in differential expression of its downstream target genes, which in turn promote cell survival to foster Coxsackievirus B3 replication
ataxia telangiectasia mutated (zeige ATM Antikörper) protein phosphorylation of ZBP-89 contributes to Histone deacetylase (zeige HDAC1 Antikörper) inhibitors induction of p21(waf1 (zeige CDKN1A Antikörper)) gene expression
identify ZBP-89 as being a novel transcription factor involved in erythroid and megakaryocytic development and suggest that it serves a cooperative function with GATA-1 (zeige GATA1 Antikörper) and/or FOG-1 (zeige ZFPM1 Antikörper) in a developmental stage-specific manner
The transcriptional repressor ZBP-89 and the lack of Sp1 (zeige SP1 Antikörper)/Sp3 (zeige SP3 Antikörper), c-jun (zeige JUN Antikörper) and Stat3 (zeige STAT3 Antikörper) are important for the down-regulation of the vimentin (zeige VIM Antikörper) gene during C2C12 myogenesis.
Involved in transcriptional regulation. Represses the transcription of a number of genes including gastrin, stromelysin and enolase. Binds to the G-rich box in the enhancer region of these genes.
CACCC box-binding protein
, CLL-associated antigen KW-10
, transcription factor ZBP-89
, zinc finger DNA-binding protein 89
, G-rich box-binding protein
, beta enolase repressor factor 1
, transcription factor BFCOL1
, zinc finger binding protein 89
, zinc finger DNA binding protein 89
, solute carrier family 12 (potassium/chloride transporters), member 8
, zinc finger protein 148 (pHZ-52)