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ERG encodes a member of the erythroblast transformation-specific (ETS) family of transcriptions factors. Zusätzlich bieten wir Ihnen V-Ets erythroblastosis Virus E26 Oncogene Homolog (Avian) Proteine (17) und V-Ets erythroblastosis Virus E26 Oncogene Homolog (Avian) Kits (1) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 114 products:
Cow (Bovine) Polyclonal ERG Primary Antibody für WB - ABIN2787485
Uhl, Liu, Drgon, Johnson, Walther, Rose, David, Niaura, Lerman: Molecular genetics of successful smoking cessation: convergent genome-wide association study results. in Archives of general psychiatry 2008
Human Polyclonal ERG Primary Antibody für WB - ABIN871680
Jønson, Vikesaa, Krogh, Nielsen, Hansen, Borup, Johnsen, Christiansen, Nielsen: Molecular composition of IMP1 ribonucleoprotein granules. in Molecular & cellular proteomics : MCP 2007
Human Polyclonal ERG Primary Antibody für IHC (p) - ABIN374642
Tomlins, Rhodes, Perner, Dhanasekaran, Mehra, Sun, Varambally, Cao, Tchinda, Kuefer, Lee, Montie, Shah, Pienta, Rubin, Chinnaiyan: Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer. in Science (New York, N.Y.) 2005
Cow (Bovine) Polyclonal ERG Primary Antibody für WB - ABIN2792539
Matsui, Chansky, Barahmand-Pour, Zielinska-Kwiatkowska, Tsumaki, Myoui, Yoshikawa, Yang, Eyre: COL11A2 collagen gene transcription is differentially regulated by EWS/ERG sarcoma fusion protein and wild-type ERG. in The Journal of biological chemistry 2003
KLF2 (zeige KLF2 Antikörper) and ERG associate in a physical complex and the two proteins synergistically activate transcription of Flk1 (zeige KDR Antikörper).
loss of ERG and FLI1 (zeige FLI1 Antikörper) might contribute to the pathogenesis of vascular lung complications through the induction of inflammation.
Combined, these data suggest a possible pathway linking GDF5 (zeige GDF5 Antikörper), ERG and downstream factors in the processes of chondrocyte differentiation during articular joint formation.
Our study reveals that ERG has essential and cell autonomous roles in endothelial cell development and blood vessel maintenance.
We show that the gene expression signature specifically attributable to trisomy of Erg in the murine model is strongly correlated with gene expression changes in human Down syndrome hematopoietic cells.
Articular cartilage endurance and resistance to osteoarthritic changes require transcription factor Erg.
ERG plays a critical role in coordinating the balance between self-renewal and differentiation of HSCs.
ERG activates the transcriptional program regulated by YAP1 (zeige YAP1 Antikörper) of the Hippo signaling pathway and found that prostate-specific (zeige KLK3 Antikörper) activation of either ERG or YAP1 (zeige YAP1 Antikörper) in mice induces similar transcriptional changes and results in age-related prostate tumors.
ERG and APLNR (zeige APLNR Antikörper) are essential for endothelial homeostasis in venules in the lung and that perturbation in ERG-APLNR (zeige APLNR Antikörper) signaling is crucial for the development of pulmonary veno-occlusive disease.
Regulation of Erg by TGF-beta (zeige TGFB1 Antikörper) was confirmed in these cells.
Ablation of the oncogenic transcription factor ERG by deubiquitinase inhibition in prostate cancer.
Data show that homogeneous ERG positivity is very rare in prostate cancer, especially in elderly patients. However, development of subpopulations with ERG fusions may be a much more frequent event in ERG negative cancer foci as previously believed.
Genome-wide gene expression profiling identified a network of VEGF (zeige VEGFA Antikörper)-responsive and ERG-dependent genes.
ERG deletion is associated with acute lymphoblastic leukemia.
Study provide evidence that PTEN (zeige PTEN Antikörper) deletion and TMPRSS2 (zeige TMPRSS2 Antikörper)-ERG gene fusion were mutually exclusive in patients with prostate neoplasm. TMPRSS2 (zeige TMPRSS2 Antikörper)-ERG gene fusion was rare compared to peripheral zone tumors and to PTEN (zeige PTEN Antikörper) inactivation in T1a (zeige PDPN Antikörper) transition zone tumors.
ERG rearrangement, but not PTEN (zeige PTEN Antikörper) loss, is present in a minority of potential neoplastic precursor lesions in the prostate.
Patients with Prostate cancer had significantly higher levels of the prostate cancer-associated gene ERG in post-digital rectal examination urine extracellular vesicles. Continued analysis of urine EVs offers the potential discovery of novel biomarkers for pre-biopsy Prostate cancer detection.
Neither the expression of aberrant proteins from internally deleted allele nor the reduced expression of wild type ERG seem to provide a plausible explanation of the specific biology of ERG -related leukemia subgroup.
Our results suggest that the immune response may differ between ERG negative and ERG positive prostatic carcinomas
identified five phosphorylated serines on endogenous ERG in T-ALL and AML (zeige RUNX1 Antikörper) cells.
fli1 (zeige FLI1 Antikörper), and etsrp (zeige ETV2 Antikörper), demonstrated that erg (zeige KCNH2 Antikörper) and fli1 (zeige FLI1 Antikörper) act cooperatively and are required for angiogenesis possibly via direct regulation of an endothelial cell junction molecule, VE-cadherin (zeige CDH5 Antikörper)
A combination of gain-of-function and loss-of-function studies suggested a redundant yet specific role for erg (zeige KCNH2 Antikörper) in both angioblast specification/proliferation and early angiogenesis, and a synergistic interaction with the critical ETS (zeige ETS1 Antikörper) factor etsrp (zeige ETV2 Antikörper).
This gene encodes a member of the erythroblast transformation-specific (ETS) family of transcriptions factors. All members of this family are key regulators of embryonic development, cell proliferation, differentiation, angiogenesis, inflammation, and apoptosis. The protein encoded by this gene is mainly expressed in the nucleus. It contains an ETS DNA-binding domain and a PNT (pointed) domain which is implicated in the self-association of chimeric oncoproteins. This protein is required for platelet adhesion to the subendothelium, inducing vascular cell remodeling. It also regulates hematopoesis, and the differentiation and maturation of megakaryocytic cells. This gene is involved in chromosomal translocations, resulting in different fusion gene products, such as TMPSSR2-ERG and NDRG1-ERG in prostate cancer, EWS-ERG in Ewing's sarcoma and FUS-ERG in acute myeloid leukemia. Multiple alternatively spliced transcript variants encoding different isoforms have been identified.
v-ets erythroblastosis virus E26 oncogene like
, Ether-a-go-go-related gene potassium channel 1
, Potassium voltage-gated channel subfamily H member 2
, v-ets erythroblastosis virus E26 oncogene homolog (avian)
, v-ets erythroblastosis virus E26 oncogene homolog
, erg gene (erg_E)
, vascular ETS factor
, transcriptional regulator ERG
, avian erythroblastosis virus E-26 (v-ets) oncogene related
, TMPRSS2/ERG fusion
, transcriptional regulator ERG (transforming protein ERG)
, v-ets avian erythroblastosis virus E26 oncogene related
, transcriptional regulator Erg