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The protein encoded by TSG101 belongs to a group of apparently inactive homologs of ubiquitin-conjugating enzymes. Zusätzlich bieten wir Ihnen TSG101 Proteine (14) und TSG101 Kits (5) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 121 products:
Hamster Monoclonal TSG101 Primary Antibody für ELISA, FACS - ABIN152256
Garrus, von Schwedler, Pornillos, Morham, Zavitz, Wang, Wettstein, Stray, Côté, Rich, Myszka, Sundquist: Tsg101 and the vacuolar protein sorting pathway are essential for HIV-1 budding. in Cell 2001
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Human Polyclonal TSG101 Primary Antibody für EIA, IHC (p) - ABIN357572
Favre, Butticaz, Stevenson, Jongeneel, Telenti: High frequency of alternative splicing of human genes participating in the HIV-1 life cycle: a model using TSG101, betaTrCP, PPIA, INI1, NAF1, and PML. in Journal of acquired immune deficiency syndromes (1999) 2003
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Human Polyclonal TSG101 Primary Antibody für EIA, WB - ABIN357573
Lu, Hope, Brasch, Reinhard, Cohen: TSG101 interaction with HRS mediates endosomal trafficking and receptor down-regulation. in Proceedings of the National Academy of Sciences of the United States of America 2003
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Dog (Canine) Monoclonal TSG101 Primary Antibody für WB - ABIN968912
Li, Cohen: Tsg101: a novel tumor susceptibility gene isolated by controlled homozygous functional knockout of allelic loci in mammalian cells. in Cell 1996
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Cow (Bovine) Polyclonal TSG101 Primary Antibody für IHC, WB - ABIN2780037
Stefan, Claiborn, Stasiek, Chai, Ohta, Longnecker, Greally, Nicholls: Genetic mapping of putative Chrna7 and Luzp2 neuronal transcriptional enhancers due to impact of a transgene-insertion and 6.8 Mb deletion in a mouse model of Prader-Willi and Angelman syndromes. in BMC genomics 2005
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Mouse (Murine) Polyclonal TSG101 Primary Antibody für IF, IHC - ABIN3023223
Yu, Yan, Yang, Wu, Ling, Chen, Tang, Tan, Cao, Wu, Yan, Wang: Platelets promote tumour metastasis via interaction between TLR4 and tumour cell-released high-mobility group box1 protein. in Nature communications 2014
Cow (Bovine) Polyclonal TSG101 Primary Antibody für WB - ABIN2780762
Surjit, Oberoi, Kumar, Lal: Enhanced alpha1 microglobulin secretion from Hepatitis E virus ORF3-expressing human hepatoma cells is mediated by the tumor susceptibility gene 101. in The Journal of biological chemistry 2006
loss of ept alters nuclear cell cycle control in developing imaginal discs and the DaPKC, presenilin, and rbf1 genes as modifiers of molecular and cellular phenotypes that result from loss of ept
analysis of genetic interactions between the Drosophila tumor suppressor gene ept and the stat92E transcription factor
Results indicate that Vpr overcomes the effects of TSG101 overexpression to support viral production by competing with TSG101 to bind Gag.
our studies reveal that Tsg101 plays a role in the trafficking of macropinocytosed Kaposi's sarcoma-associated herpesvirus in the endothelial cells
Knockdown of LAMP2A, a CMA-related protein, and TSG101, an mA-related protein, significantly but only partially decreased the punctate accumulation of GAPDH (zeige GAPDH Antikörper)-HT in AD293 cells and primary cultured rat cortical neurons.
the variant alleles of TSG101 rs2292179 and ATF2 (zeige ATF2 Antikörper) rs3845744 were associated with a reduced risk of breast cancer, particularly for subjects with BMI <24 (kg/m(2)) and postmenopausal women, respectively
TSGDelta154-1054 splice variant increases TSG101 oncogenicity by inhibiting its E3-ligase-mediated proteasomal degradation.
Results show that TSG101 bidirectionally modulates cell invasion through regulating MMP-9 (zeige MMP9 Antikörper) mRNA expression in different cell types.
TSG101 plays an important role in the development of hepatocellular carcinoma
Expression of tsg101 mRNA and of TSG101 protein were significantly higher in the oxaliplatin resistant cell line than parent HT-29 cels.
PSAP (zeige PSAP Antikörper) motif of OFR3 is required for hepatitis E virus exit and interaction with host TSG101.
Stress-internalized EGFR (zeige EGFR Antikörper) is retained intracellularly by continued p38 (zeige CRK Antikörper) activity in a mechanism involving ubiquitin-independent, ESCRT/ALIX (zeige PDCD6IP Antikörper)-dependent incorporation onto intraluminal vesicles (ILVs (zeige IARS Antikörper)) of MVBs
Tsg101 was deleted from mature oligodendroglia in vivo. This resulted in severe spongiform encephalopathy, histopathologically similar to that observed in Mgrn1 (zeige MGRN1 Antikörper) null mutant mice but with a more rapid onset.
BLOS1 (zeige BLOC1S1 Antikörper) interacts with sorting nexin 2 (zeige SNX2 Antikörper) and the ESCRT-I (zeige VPS28 Antikörper) component TSG101 to mediate the sorting of EGFR (zeige EGFR Antikörper) into endosomal compartments
pigment-type switching likely requires MGRN1's ubiquitin ligase activity but not its ability to bind TSG101 or NEDD4.
Tsg101 has a role in expression of EGFR (zeige EGFR Antikörper) and ErbB2 (zeige ERBB2 Antikörper) and preventing induction of autophagy prior to cell death
show that ubiquitin recognition by TSG101 is required for cSMAC formation, T cell receptor (TCR) microcluster signal termination, TCR downregulation.
Negative regulation of cell growth and differentiation by TSG101 through association with p21(Cip1/WAF1 (zeige CDKN1A Antikörper)).
Targeted deletion results in cell cycle arrest and p53 (zeige TP53 Antikörper)-independent cell death
our results suggest that Tsg101 is required for normal cell function of embryonic and adult tissues but that this gene is not a tumor suppressor for sporadic forms of breast cancer.
the TSG101 interaction with HRS is a crucial step in endocytic down-regulation of mitogenic signaling and this interaction may have a role in linking the functions of early and late endosomes
the Tsg101 protein has only weak oncogenic properties
The protein encoded by this gene belongs to a group of apparently inactive homologs of ubiquitin-conjugating enzymes. The gene product contains a coiled-coil domain that interacts with stathmin, a cytosolic phosphoprotein implicated in tumorigenesis. The protein may play a role in cell growth and differentiation and act as a negative growth regulator. In vitro steady-state expression of this tumor susceptibility gene appears to be important for maintenance of genomic stability and cell cycle regulation. Mutations and alternative splicing in this gene occur in high frequency in breast cancer and suggest that defects occur during breast cancer tumorigenesis and/or progression.
, tumor susceptibility Gene-101
, Tumor Susceptibility Gene homolog family member (tsg-101)
, ESCRT-I complex subunit TSG101
, tumor susceptibility gene 101 protein
, tumor susceptibility protein 101
, tumor susceptibility gene 101
, tumor susceptibility gene 10
, tumor susceptibility protein
, Tumor supressor