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KIF18A is a member of the kinesin superfamily of microtubule-associated molecular motors (see MIM 148760) that use hydrolysis of ATP to produce force and movement along microtubules (Luboshits and Benayahu, 2005 [PubMed 15878648]).[supplied by OMIM, Aug 2008].. Zusätzlich bieten wir Ihnen KIF18A Kits (4) und KIF18A Proteine (3) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal KIF18A Primary Antibody für ICC, IF - ABIN4328804
Stadler, Rexhepaj, Singan, Murphy, Pepperkok, Uhlén, Simpson, Lundberg: Immunofluorescence and fluorescent-protein tagging show high correlation for protein localization in mammalian cells. in Nature methods 2013
Low post translational modifications of KIF18A protein is associated with neoplasms.
KIF18a role in microtubule assembly
Cdk1 (zeige CDK1 Antikörper)-mediated inhibitory phosphorylation of Kif18A promotes chromosome oscillations in early metaphase. PP1 (zeige PPA1 Antikörper) induces metaphase plate thinning by directly dephosphorylating Kif18A.
Confocal microscopy shows that cells expressing SUMO-resistant Kif18A display a compromised dissociation of BubR1 (zeige BUB1B Antikörper) from kinetochores after anaphase onset.
that the human mitotic kinesin-8, KIF18A, directly interacts with PP1gamma through a conserved RVxF motif
the motion of yeast (Kip3) and human (Kif18A) kinesin-8s
Mechanisms controlling the temporal degradation of Nek2A (zeige NEK2 Antikörper) and Kif18A by the APC (zeige APC Antikörper)/C-Cdc20 (zeige CDC20 Antikörper) complex.
Kif18A (kinesin-8) attenuates centromere movement by directly promoting microtubule pausing in a concentration- dependent manner.
there is a mutual regulation of kinetochore MT plus-end dynamics and Kif18A accumulation, which may contribute to the highly regulated and ordered changes in kinetochore spindle microtubule dynamics during chromosome congression and oscillation
Kif18A controls spindle length independently of its role in chromosome positioning. This is mediated by an ATP-independent spindle microtubule (MT) binding site at C-terminal end of the Kif18A tail that has a strong affinity for MTs (zeige TIMM8A Antikörper) in vitro and in cells.
Kif18a is specifically required for mitotic progression during germ line development.
In conclusion, Kif18a is critical for colorectal carcinogenesis in the setting of inflammation by mechanisms of increased PI3K-AKT (zeige AKT1 Antikörper) signaling.
KIF18A is a member of the kinesin superfamily of microtubule-associated molecular motors (see MIM 148760) that use hydrolysis of ATP to produce force and movement along microtubules (Luboshits and Benayahu, 2005
kinesin family member 18A
, kinesin-like protein KIF18A
, kinesin-like protein KIF18A-like
, N-8 kinesin
, kinesin superfamily protein 18A