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IL32 encodes a member of the cytokine family. Zusätzlich bieten wir Ihnen IL32 Kits (39) und IL32 Proteine (10) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 153 products:
Human Polyclonal IL32 Primary Antibody für WB - ABIN2475003
Brandon, Naif, Daniel, Lavin: Early detection of bovine leukosis virus DNA in infected sheep using the polymerase chain reaction. in Research in veterinary science 1991
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Human Polyclonal IL32 Primary Antibody für IHC (fro), WB - ABIN2475004
Kim, Han, Azam, Yoon, Dinarello: Interleukin-32: a cytokine and inducer of TNFalpha. in Immunity 2005
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Human Polyclonal IL32 Primary Antibody für ELISA - ABIN4225855
Ouhara, Kawai, Silva, Fujita, Hayashida, Karimbux, Kajiya, Shiba, Kawaguchi, Kurihara: Expression levels of novel cytokine IL-32 in periodontitis and its role in the suppression of IL-8 production by human gingival fibroblasts stimulated with Porphyromonas gingivalis. in Journal of oral microbiology 2012
Human Polyclonal IL32 Primary Antibody für ICC, IF - ABIN4325180
Player, Oguamanam, Okanmelu, Burrell, Hollomon: Preliminary characterization of IL32 in basal-like/triple negative compared to other types of breast cell lines and tissues. in BMC research notes 2014
Human Polyclonal IL32 Primary Antibody für ELISA, ICC - ABIN4325179
Thomi, Yerly, Yawalkar, Simon, Schlapbach, Hunger: Interleukin-32 is highly expressed in lesions of hidradenitis suppurativa. in The British journal of dermatology 2017
Human Monoclonal IL32 Primary Antibody für ELISA, WB - ABIN2475002
Kim: Interleukin-32 in inflammatory autoimmune diseases. in Immune network 2014
Human Monoclonal IL32 Primary Antibody für FACS - ABIN4897255
Zou, Bao, Pan, Lu, Liao, Wang, Wang, Lin: NKP30-B7-H6 Interaction Aggravates Hepatocyte Damage through Up-Regulation of Interleukin-32 Expression in Hepatitis B Virus-Related Acute-On-Chronic Liver Failure. in PLoS ONE 2015
This study showed that the induction level of IL-32 was increased in chronic rhinosinusitis with nasal polyps compared to normal nasal mucosa and that LPS (zeige IRF6 Antikörper)-induced IL-32 expression in nasal polyp-derived fibroblasts was regulated via the TLR4 (zeige TLR4 Antikörper)/JNK (zeige MAPK8 Antikörper)/AKT (zeige AKT1 Antikörper)/CREB (zeige CREB1 Antikörper) signaling pathway.
findings show that IL-32alpha acts on NK cells to inhibit IL-15 (zeige IL15 Antikörper)-mediated STAT5 (zeige STAT5A Antikörper) phosphorylation and to suppress their IL-15 (zeige IL15 Antikörper)-induced effector molecule expression and cytolytic capacity. IL-32alpha also acted on DCs by downregulating IL-15 (zeige IL15 Antikörper)-induced IL-18 (zeige IL18 Antikörper) production, an important cytokine in NK cell activity.
Thus, endogenous IL-32 is a crucial cytokine involved in the host defense against Leishmania parasites.
Study indicated the overexpression of IL-32 in induced sputum of smokers and chronic obstructive pulmonary disease (COPD (zeige ARCN1 Antikörper)) patients, which was correlated with smoking exposure index and the degree of airway obstruction. It demonstrated that IL-32 might play critical role in smokers with COPD (zeige ARCN1 Antikörper).
this study shows that IL-32 might be involved in the pathogenesis of hepatic fibrosis by inducing TIMP-1 (zeige TIMP1 Antikörper) expression
serum levels of IL-32 and TNF-alpha (zeige TNF Antikörper) may be diagnostic markers, and serum IL-29 (zeige IL29 Antikörper) levels may be associated with good prognosis in patients with gastric cancer
the importance of IL-32 polymorphism and mRNA expression in susceptibility and influence of survival status in lung cancer
These findings suggested that IL-18RAP rs917997, IL-32 rs2015620, IL-22 (zeige IL22 Antikörper) rs1179251, and interactions between these polymorphisms and H. pylori infection were associated with risks of gastric lesions
Data identify interleukin-32 (IL-32) as a potential therapeutic target.
Low IL-32 expression is associated with colon cancer.
This gene encodes a member of the cytokine family. The protein contains a tyrosine sulfation site, 3 potential N-myristoylation sites, multiple putative phosphorylation sites, and an RGD cell-attachment sequence. Expression of this protein is increased after the activation of T-cells by mitogens or the activation of NK cells by IL-2. This protein induces the production of TNFalpha from macrophage cells. Alternate transcriptional splice variants, encoding different isoforms, have been characterized.
, interleukin-32 eta
, interleukin-32 small
, interleukin-32 theta
, natural killer cell transcript 4
, natural killer cells protein 4
, tumor necrosis factor alpha-inducing factor