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ALOX5AP encodes a protein which, with 5-lipoxygenase, is required for leukotriene synthesis. Zusätzlich bieten wir Ihnen ALOX5AP Kits (8) und ALOX5AP Proteine (5) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 63 products:
Human Polyclonal ALOX5AP Primary Antibody für ELISA, ICC - ABIN268657
Finkensieper, Kieser, Bekhite, Richter, Mueller, Graebner, Figulla, Sauer, Wartenberg: The 5-lipoxygenase pathway regulates vasculogenesis in differentiating mouse embryonic stem cells. in Cardiovascular research 2010
Show all 3 Pubmed References
Mouse (Murine) Monoclonal ALOX5AP Primary Antibody für WB - ABIN968396
Liu, Yin: Identification of the binding partners for flightless I, A novel protein bridging the leucine-rich repeat and the gelsolin superfamilies. in The Journal of biological chemistry 1998
Human Polyclonal ALOX5AP Primary Antibody für IHC (p), IHC - ABIN251683
Kaaman, Rydén, Axelsson, Nordström, Sicard, Bouloumié, Langin, Arner, Dahlman: ALOX5AP expression, but not gene haplotypes, is associated with obesity and insulin resistance. in International journal of obesity (2005) 2006
Human Polyclonal ALOX5AP Primary Antibody für IHC, IHC (p) - ABIN4312085
Perisic, Hedin, Razuvaev, Lengquist, Osterholm, Folkersen, Gillgren, Paulsson-Berne, Ponten, Odeberg, Hedin: Profiling of atherosclerotic lesions by gene and tissue microarrays reveals PCSK6 as a novel protease in unstable carotid atherosclerosis. in Arteriosclerosis, thrombosis, and vascular biology 2013
ALOX5AP Overexpression in Adipose Tissue Leads to LXA4 Production and Protection Against Diet-Induced Obesity and Insulin (zeige INS Antikörper) Resistance
Data indicate that a single amino acid mutation was sufficient to reverse the speciation observed in wild type 5-Lipoxygenase activating protein (FLAP).
BRP (zeige GDF5 Antikörper)-7 potently suppresses leukotriene biosynthesis by interacting with FLAP in mouse peritonitis model.
FLAP regulates phosphorylation of tau protein via modulating GSK-3beta (zeige GSK3b Antikörper) activity.
This study demonistrated that a a novel role for FLAP in the pathogenesis of anxiety-like behavior in mice.
Deletion of myeloid cell FLAP markedly attenuates the proliferative response to vascular injury, while preserving endothelial integrity.
A FLAP inhibitor given during critical window periods may prevent oxygen induced aberration of pulmonary alveolarization in a dose- and time-dependent manner.
the 5-LO (zeige ALOX5 Antikörper) pathway emerges early during embryonic stem cell differentiation into cells of the myeloid lineage
Adipose tissue from obese mice, compared with that from lean mice, exhibited increased 5-LO (zeige ALOX5 Antikörper) activating protein
Data show that 5-lipoxygenase (zeige ALOX5 Antikörper) activity increases during senescence-like growth arrest via a p53 (zeige TP53 Antikörper)/p21 (zeige D4S234E Antikörper)-dependent pathway in both human and mouse embryo fibroblasts.
This study revealed that epistatic interaction among the ALOX5 (zeige ALOX5 Antikörper), ALOX5AP and MPO (zeige MPO Antikörper) genes played a significant role in vulnerability to ischemic stroke.
The results the study indicate that SNP genetic variants of ALOX5AP might play a role in the development of SSc (zeige CYP11A1 Antikörper)-related pulmonary fibrosis and ventilatory dysfunction.
The observation that the coexpression of FLAP with a subset of the 5-LOX (zeige ALOX5 Antikörper) mutants restores 5-LOX (zeige ALOX5 Antikörper)-wild-type (wt)-like levels of products formed in intact cells suggests a physical protein-protein interaction, beyond colocalization, of 5-LOX (zeige ALOX5 Antikörper) and FLAP.
Meta-analysis suggests that the A allele at the ALOX5AP SG13S114 polymorphism is a protective factor for the ischemic stroke in the European population
Genetic polymorphisms of T-1131C APOA5 (zeige APOA5 Antikörper) and ALOX5AP SG13S114 can be considered risk factors for the susceptibility to ischemic stroke in Morocco.
No association was found between single-nucleotide polymorphisms of ALOX5AP or PDE4D (zeige PDE4D Antikörper) and the risk of overall ischemic stroke in a southeastern Chinese population. The ALOX5AP gene might be related to ischemic stroke incidence in females.
Genetic interaction of CD80 (zeige CD80 Antikörper) and ALOX5AP was observed in systemic lupus erythematosus in Asian populations.
findings show that several enzymes known to be involved in the biosynthesis of proinflammatory LTs, such as FLAP and cPLA2alpha (zeige PLA2G4A Antikörper), also contribute to LX and Rv formation
The results of this study supported the association of the epistatic interactions of ALOX5AP, THBD (zeige THBD Antikörper), and KNG1 (zeige KNG1 Antikörper) and present novel evidence for the main effect of KNG1 (zeige KNG1 Antikörper) gene on IS susceptibility
ALOX5AP, CPNE3 (zeige CPNE3 Antikörper), IL1R2 (zeige IL1R2 Antikörper), IL6 (zeige IL6 Antikörper), TLR2 (zeige TLR2 Antikörper), TLR4 (zeige TLR4 Antikörper), and THY1 (zeige THY1 Antikörper) were upregulated in blood polymorphonuclear cells in negative energy balance versus positive energy balance cows.
This gene encodes a protein which, with 5-lipoxygenase, is required for leukotriene synthesis. Leukotrienes are arachidonic acid metabolites which have been implicated in various types of inflammatory responses, including asthma, arthritis and psoriasis. This protein localizes to the plasma membrane. Inhibitors of its function impede translocation of 5-lipoxygenase from the cytoplasm to the cell membrane and inhibit 5-lipoxygenase activation. Alternatively spliced transcript variants encoding different isoforms have been identified for this gene.
arachidonate 5-lipoxygenase-activating protein
, Arachidonate 5-lipoxygenase-activating protein
, MK-886-binding protein
, arachidonate 5 lipoxygenase activating protein
, 5-lipoxygenase-activating protein FLAP
, arachidonate 5-lipoxygenase activating protein
, 5-lipoxygenase-activating protein