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anti-Rat (Rattus) PTPRJ Antikörper:
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Human Monoclonal PTPRJ Primary Antibody für FACS - ABIN4898524
Llinàs, Lázaro, de Salort, Matesanz-Isabel, Sintes, Engel: Expression profiles of novel cell surface molecules on B-cell subsets and plasma cells as analyzed by flow cytometry. in Immunology letters 2010
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Human Monoclonal PTPRJ Primary Antibody für CyTOF, FACS - ABIN4900751
Cabezón, Sintes, Llinàs, Benitez-Ribas: Analysis of HLDA9 mAbs on plasmacytoid dendritic cells. in Immunology letters 2010
Show all 2 Pubmed References
Human Monoclonal PTPRJ Primary Antibody für FACS - ABIN4898523
Rollin, Pouplard, Gratacap, Leroux, May, Aupart, Gouilleux-Gruart, Payrastre, Gruel: Polymorphisms of protein tyrosine phosphatase CD148 influence FcγRIIA-dependent platelet activation and the risk of heparin-induced thrombocytopenia. in Blood 2012
study reveals the crucial role of miR (zeige MLXIP Antikörper)-155/PTPRJ/AKT (zeige AKT1 Antikörper) axis in proliferation and migration of colorectal cancer cells and suggests a therapeutic potential of PTPRJ.
Authors demonstrate that mtp53 prevents the COP1 (zeige CARD16 Antikörper)/DET1 complex from ubiquitinating ETS2 (zeige ETS2 Antikörper) and thereby marking it for destruction. Authors show that mtp53 destabilizes DET1 and also disrupts the DET1/ETS2 (zeige ETS2 Antikörper) complex thereby preventing ETS2 (zeige ETS2 Antikörper) degradation.
These data support that PTPN22 (zeige PTPN22 Antikörper) 1858C/T, PTPRJ 2965C/G and 1176 A/C polymorphisms and ACP1 A (zeige ACP1 Antikörper), B and C alleles are not associated with a higher risk of immune thrombocytopenia P in adults.
Loss of PTPRJ expression may predict an aggressive clinical course in ESCC patients.
The strongest association with frailty was observed in the Protein Tyrosine Phosphatase (zeige ACP1 Antikörper), Receptor type, J (PTPRJ) (rs1566729, P = 0.001372, beta = 0.09397) gene.
the combination of CD200 (zeige CD200 Antikörper) and CD148 may have a potential differential diagnostic value in leukemic B-CLPDs, especially between CLL and MCL (zeige FH Antikörper).
These results demonstrated Ptprj as a physiological enzyme that attenuates insulin (zeige INS Antikörper) signalling in vivo, and indicate that an inhibitor of Ptprj may be an insulin (zeige INS Antikörper)-sensitizing agent.
CD148 tyrosine phosphatase promotes e-cadherin (zeige CDH1 Antikörper) cell adhesion.
The studies suggest induction of MMP-9 (zeige MMP9 Antikörper) expression promoted by DEP-1 deficiency.
Moderate expression of DEP-1 was associated with the increased relapse.
This study demonstrated that the protein-tyrosine phosphatase (zeige ACP1 Antikörper) DEP-1 promotes migration and phagocytic activity of microglial cells in part through negative regulation of fyn (zeige FYN Antikörper) tyrosine kinase (zeige TYRO3 Antikörper).
our results established DEP-1 as an essential driver of VEGF-dependent permeability, angiogenesis, and metastasis
DEP-1 acts as an endogenous antagonist of the insulin receptor (zeige INSR Antikörper), and downregulation of DEP-1 results in an improvement of insulin (zeige INS Antikörper) sensitivity.
Data indicate that CD148 mRNA is upregulated in diseased joints of with collagen-induced arthritis.
The large ectodomains of CD45 and CD148 modulate their inhibitory effect by enabling their passive, size-based segregation from ligated TCR, supporting the kinetic-segregation model of TCR triggering.
phosphatase CD148 promotes airway hyperresponsiveness through SRC (zeige SRC Antikörper) family kinases
These data indicate that PTPRJ may regulate differentiation of normal mammary epithelia and that dysregulation of protein localisation may be associated with tumorigenesis.
CD148 plays a dominant role in activating Src (zeige SRC Antikörper) family kinases in platelets relative to PTP-1B (zeige PTPN1 Antikörper). Both PTPs (zeige PTS Antikörper) are required for optimal platelet activation and aggregate formation under high arterial shear rates.
CD45 and CD148 preferentially target different SFK members (Hck and Fgr versus Lyn, respectively) to positively and negatively regulate GPCR pathways.
The protein encoded by this gene is a member of the protein tyrosine phosphatase (PTP) family. PTPs are known to be signaling molecules that regulate a variety of cellular processes, including cell growth, differentiation, mitotic cycle, and oncogenic transformation. This PTP possesses an extracellular region containing five fibronectin type III repeats, a single transmembrane region, and a single intracytoplasmic catalytic domain, and thus represents a receptor-type PTP. This protein is present in all hematopoietic lineages, and was shown to negatively regulate T cell receptor signaling possibly through interfering with the phosphorylation of Phospholipase C Gamma 1 and Linker for Activation of T Cells. This protein can also dephosphorylate the PDGF beta receptor, and may be involved in UV-induced signal transduction. Multiple transcript variants encoding different isoforms have been found for this gene.
receptor-type tyrosine-protein phosphatase eta
, density enhanced phosphatase-1
, protein tyrosine phosphatase, receptor type, J
, receptor-type tyrosine-protein phosphatase eta-like
, CD148 antigen
, HPTP eta
, density-enhanced phosphatase 1
, human density enhanced phosphatase-1
, protein tyrosine phosphatase, receptor type, J polypeptide
, protein-tyrosine phosphatase eta
, protein-tyrosine phosphatase receptor type J
, susceptibility to colon cancer 1, mouse, homolog of
, HPTP beta-like tyrosine phosphatase
, colon tumor susceptibility 1
, susceptibility to colon cancer 1
, supporting-cell antigen