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Human Polyclonal SLC25A4 Primary Antibody für ELISA - ABIN559869
Loers, Makhina, Bork, Dörner, Schachner, Kleene et al.: The interaction between cell adhesion molecule L1, matrix metalloproteinase 14, and adenine nucleotide translocator at the plasma membrane regulates L1-mediated neurite outgrowth of murine cerebellar ... in The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
ANT1 can induce autoimmune myocarditis in A/J mice by generating autoreactive T cells.
miR (zeige MLXIP Antikörper)-2861 and ANT1 are regulators of cardiomyocyte necrosis and myocardial infarction.
These results suggest that follicular helper T cells cells and IL-21 (zeige IL21 Antikörper) might be involved in the pathogenesis of viral myocarditis and play an important role in anti-ANT autoantibody production.
Data indicate that palmitoyl-carnitine (PC) increases the type 2 ryanodine receptor (RyR2 (zeige RYR2 Antikörper)) oxidation and reduces the ATP/ADP translocase (ANT) activity.
Downregulation of adenine nucleotide translocator 1 exacerbates tumor necrosis factor-alpha (zeige TNF Antikörper)-mediated cardiac inflammatory responses.
Plasma membrane-localized ANT1 and ANT2 (zeige SLC25A5 Antikörper) regulate L1-mediated neurite outgrowth in conjunction with MMP14 (zeige MMP14 Antikörper).
Although the molecular mechanism linking ANT1-Cys (zeige DNAJC5 Antikörper)(57) nitroalkylation and uncoupling is not yet known, these data suggest that ANT1-mediated uncoupling may be a mechanism for nitroalkene-induced cardioprotection.
The attenuation of ANT-1 in the presence of PGC-1alpha (zeige PPARGC1A Antikörper) overexpression preserves the mitochondrial membrane potential in response to hydrogen-peroxide stress.
Data demonstrate that ANT1 protein level (zeige YY1 Antikörper)s are upregulated in Mecp2-null mice.
uncoupling protein-3 (zeige UCP3 Antikörper) can minimize the induction of the adenine nucleotide translocase (zeige SLC25A5 Antikörper)-mediated 'energy-wasting' process during Calorie restriction (CR).
Yeast aac2 (zeige SLC25A5 Antikörper) R96H and aac2 (zeige SLC25A5 Antikörper) R252G mutations are equivalent to R80H and R235G human ANT1 pathological mutations. mtDNA instability induced by aac2R96H and aac2R252G is rescued by N-acetylcysteine.
identification by whole-exome sequencing of seven probands harboring dominant, de novo SLC25A4 mutations; all affected individuals presented at birth, were ventilator dependent and, where tested, revealed severe combined mitochondrial respiratory chain deficiencies associated with a marked loss of mitochondrial DNA copy number in skeletal muscle
A directed proteomic approach discovered the novel interaction of BKCa (zeige KCNMA1 Antikörper) with Tom22 (zeige TOMM22 Antikörper), a component of the mitochondrion outer membrane import system, and the adenine nucleotide translocator (ANT).
Identification of ZNF555 (zeige ZNF555 Antikörper) as a putative transcriptional factor that impacts ANT1 promoter activity in facioscapulohumeral dystrophy myoblasts.
elevated ANT1 expression supports EV infection and is associated with EV persistence, a condition with adverse prognosis.
Data suggest acetylation of ANT1 at lysines 10/23/92 has dramatic physiological effects on ADP-ATP exchange; extent of acetylation of lysine 23 decreases following physical activity; this change is highly dependent on insulin (zeige INS Antikörper) sensitivity/resistance.
Expression of ANT1 were lower in inclusion body myositis samples versus both polymyositis and controls
Compares and contrasts all the known human SLC25A (zeige SLC25A25 Antikörper)* genes and includes functional information.
A 13-generation Mennonite pedigree with autosomal recessive myopathy and cardiomyopathy due to an SLC25A4 frameshift null mutation (c.523delC, p.Q175RfsX38), which codes for the heart-muscle isoform of the adenine nucleotide translocator-1, was studied.
This report expands the clinical spectrum of ANT1-related human diseases, and emphasises the crucial role of the mitochondrial ADP/ATP carriers in muscle function and pathophysiology of human myopathies.
Data show that the binding sites on adenine nucleotide translocator 1 (ANT1) have an affinity for cardiolipin (CL).
This gene is a member of the mitochondrial carrier subfamily of solute carrier protein genes. The product of this gene functions as a gated pore that translocates ADP from the mitochondrial matrix into the cytoplasm. The protein forms a homodimer embedded in the inner mitochondria membrane. Mutations in this gene have been shown to result in autosomal dominant progressive external opthalmoplegia and familial hypertrophic cardiomyopathy.
adenine nucleotide translocator 1
, ADP,ATP carrier protein 1, mitochondrial
, ANT 1
, ADP/ATP translocase 1
, Adenine nucleotide translocator 1
, ADP,ATP carrier protein 1
, ADP,ATP carrier protein, heart/skeletal muscle
, adenine nucleotide translocase-1
, adenine nucleotide translocator 1, skeletal muscle
, solute carrier family 25 member 4
, mitochondrial adenine nucleotide translocator
, solute carrier family 25 (mitochondrial adenine nucleotide translocator) member 4
, adenine nucleotide translocator 1 (skeletal muscle)
, heart/skeletal muscle ATP/ADP translocator
, ADP,ATP carrier protein, heart
, ADP/ATP carrier
, ADP/ATP translocase T1
, 30 kDa CSQ-binding protein
, 30 kDa calsequestrin-binding protein
, CSQ-binding 30 kDa protein
, solute carrier family 25 (mitochondrial carrier; adenine nucleotide translocator), member 5