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Rat (Rattus) STAT5B ELISA Kit für Sandwich ELISA - ABIN1117115
Sun, Wang, Yan, Lu, Shi: Effects of Er-Zhi-Wan on microarchitecture and regulation of Wnt/?-catenin signaling pathway in alveolar bone of ovariectomized rats. in Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban 2014
This study indicates that CISH (zeige CISH ELISA Kits) functions as a conserved in vivo target and regulator of STAT5 in the control of embryonic hematopoiesis.
study demonstrates that STAT5 in basophils is activated through both the IL-3 (zeige IL-3 ELISA Kits) and the FcepsilonRI (zeige FCER1G ELISA Kits) signaling pathway
Studies demonstrate a conserved role for SOCS1 (zeige SOCS1 ELISA Kits) in T cell development and suggest a novel T cell-independent function in embryonic myelopoiesis mediated, at least in part, via its effects on receptors using the Jak2 (zeige JAK2 ELISA Kits)-Stat5 pathway.
The stat5.1 gene lies next to the stat3 (zeige STAT3 ELISA Kits) gene.
this study shows that musculin (zeige MSC ELISA Kits) inhibits human T-helper 17 cell response to interleukin 2 (zeige IL2 ELISA Kits) by controlling STAT5B activity
Molecular interactions of EphA4 (zeige EPHA4 ELISA Kits), growth hormone receptor (zeige GHR ELISA Kits), Jak2 (zeige JAK2 ELISA Kits), and STAT5B have been described.
All merkel cell carcinoma (MCC) cases with a good outcome expressed pSTAT5B, whereas all MCC cases with a poor outcome did not express pSTAT5B; hence, pSTAT5B expression may be an indicator of positive prognosis in patients with MCC.
Data show that signal transducer and activator of transcription 5 STAT5A (zeige STAT5A ELISA Kits) and STAT5B have distinctive and potentially important roles in regulation of hepatic drug response genes as well as in contributing to gender-related differences in expression of hepatic cytochrome P450s (CYPs) and transcription factors (TFs).
STAT5b confers gemcitabine chemoresistance and promotes cell adherence and invasiveness in pancreatic cancer cells.
STAT5 (zeige STAT5A ELISA Kits) activation by EGF (zeige EGF ELISA Kits) constitutes an important cascade for the regulation of cell proliferation and invasion in trophoblast cells.
MSM decreased the ability of STAT5b to bind the promoter of the HER2 (zeige ERBB2 ELISA Kits) gene.
Results confirm that STAT5B is mutated in T-PLL and underscore the potential therapeutical relevance of epigenetic regulator.
ABL (zeige ABL1 ELISA Kits)-N administration induced apoptosis of PC3 (zeige PCSK1 ELISA Kits) cells in a dose-dependent manner, along with the enhanced activity of caspases and increased Bax (zeige BAX ELISA Kits)/Bcl-2 (zeige BCL2 ELISA Kits) ratio. Expression of KLF5 (zeige KLF5 ELISA Kits), Stat5b and ICAM-1 (zeige ICAM1 ELISA Kits) was significantly downregulated in PC3 (zeige PCSK1 ELISA Kits) cells.
The two STAT5 (zeige STAT5A ELISA Kits) isoforms, STAT5a (zeige STAT5A ELISA Kits) and STAT5b.
A study of the polymorphisms of STAT5b is presented, and the use of radiation hybrid mapping to localize the gene to chromosome 12 is presented.
SNPs in JAK2 (zeige JAK2 ELISA Kits) and STAT5B are associated with mastitis susceptibility in Chinese Holstein cattle
This study suggests that growth hormone (zeige GH1 ELISA Kits) inhibits the differentiation of bovine preadipocytes to adipocytes through STAT5b-dependent inhibition of C/EBPalpha (zeige CEBPA ELISA Kits) and PPARgamma (zeige PPARG ELISA Kits) expression.
These results suggested that the SNPs in CD4 (zeige CD4 ELISA Kits) and STAT5b may be potential genetic markers for SCS (zeige TWIST1 ELISA Kits) and milk/protein (zeige CSN2 ELISA Kits) yields selecting and warrant further functional research.
These studies establish the importance of STAT5 (zeige STAT5A ELISA Kits) in macrophages during ductal morphogenesis in the mammary gland and demonstrate that altering STAT5 (zeige STAT5A ELISA Kits) function in macrophages can affect the development of tissue-specific disease.
Data indicate that 89% of signal transducer and activator of transcription 5 (zeige STAT5A ELISA Kits) (Stat5b)-CA mice in which the T2/Onc transposon had been mobilized died of progenitor B-cell acute lymphoblastic leukemia (B-ALL) by 3 months of age.
STAT5B and MOF (zeige KAT8 ELISA Kits) work as negative regulators in adipogenesis.
Strong selective advantage for leukemic transformation in the background of Stat5 (zeige STAT5A ELISA Kits) deficient hematopoiesis was permissive for faster initiation of Myc (zeige MYC ELISA Kits)-induced transformation to B (zeige TDO2 ELISA Kits)-ALL.
liver STAT5b activation/masculinization occurred at puberty and suppression/feminization occurred during aging and in mutant mice with defects in growth hormone signaling.
Loss of STAT5b had no effect on either sex.
These data provide the first definitive evidence for a contribution of STAT5a (zeige STAT5A ELISA Kits)/b to the sex bias in pulmonary hypertension in the hypoxic mouse and implicate reduced STAT5 (zeige STAT5A ELISA Kits) in the pathogenesis of the human disease.
Data show that three hypotheses of dimer formation between STAT5 (zeige STAT5A ELISA Kits) transcription factors STAT5A (zeige STAT5A ELISA Kits) and STAT5B were used to explain the analytical results by a static mathematical model.
Increased expression of STAT5b + PDGFB (zeige PDGFB ELISA Kits) led to increased expression of downstream STAT5b targets.
Expression of miR (zeige MLXIP ELISA Kits)-21 is under prolactin (zeige PRL ELISA Kits) control through the transcription factors STAT5A (zeige STAT5A ELISA Kits)/B.
The protein encoded by this gene is a member of the STAT family of transcription factors. In response to cytokines and growth factors, STAT family members are phosphorylated by the receptor associated kinases, and then form homo- or heterodimers that translocate to the cell nucleus where they act as transcription activators. This protein mediates the signal transduction triggered by various cell ligands, such as IL2, IL4, CSF1, and different growth hormones. It has been shown to be involved in diverse biological processes, such as TCR signaling, apoptosis, adult mammary gland development, and sexual dimorphism of liver gene expression. This gene was found to fuse to retinoic acid receptor-alpha (RARA) gene in a small subset of acute promyelocytic leukemias (APLL). The dysregulation of the signaling pathways mediated by this protein may be the cause of the APLL.
signal transducer and activator of transcription 5A
, Signal transducer and activator of transcription 5B
, signal transducer and activator of transcription 5B
, transcription factor STAT5B
, mammary gland factor STAT5B
, Signal transducer and activator of transcription 5a