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The protein encoded by Prok1 induces proliferation, migration, and fenestration (the formation of membrane discontinuities) in capillary endothelial cells derived from endocrine glands. Zusätzlich bieten wir Ihnen Prokineticin 1 Kits (68) und Prokineticin 1 Proteine (18) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal Prokineticin 1 Primary Antibody für FACS, IHC (p) - ABIN251379
Hoffmann, Feige, Alfaidy: Expression and oxygen regulation of endocrine gland-derived vascular endothelial growth factor/prokineticin-1 and its receptors in human placenta during early pregnancy. in Endocrinology 2006
important role for PK-1 in luteal function by acting as a mitogen and survival factor in corpus luteum derived endothelial cells
PROK1, acting via PROKR1 (zeige PROKR1 Antikörper), may be involved in the recruitment of monocytes to regressing CL
EG-VEGF/PK1 and Bv8/PK2 (zeige PROK2 Antikörper) thus represent new regulatory peptides acting as autocrine mitogens for endocrine cells
These findings demonstrate a novel function of primary cilia in controlling EG-VEGF-regulated trophoblast invasion and reveal the underlying molecular mechanism.
miR (zeige MLXIP Antikörper)-346 and miR (zeige MLXIP Antikörper)-582-3p regulate EG-VEGF-induced trophoblast invasion through repressing MMP 2 (zeige MMP2 Antikörper) and MMP 9 (zeige MMP9 Antikörper), and may become novel diagnostic biomarkers or therapeutic targets for EG-VEGF-related obstetric disorders. (c) 2016 BioFactors, 43(2):210-219, 2017.
Each follicular fluid (FF) was individually aspirated and FF/serum EG-VEGF, inhibin-a (zeige INHA Antikörper), and FF IGF-1 (zeige IGF1 Antikörper) levels were evaluated. The pregnant group was characterized by increased numbers of WVFs (p = 0.044), a WVE (p = 0.022), and increased levels of FF IGF-1 (zeige IGF1 Antikörper) (p = 0.001) and serum EG-VEGF (p = 0.03).
Results show the biological effects of PROK1-V67I on cell functions are similar to those of wild type, and the common variant of V67I may act as a modifier in the PROK1-PROKR system through down-regulation of PROK1 expression.
Data suggest that prokineticins (PROK1 and PROK2 (zeige PROK2 Antikörper)) and prokineticin receptors (PROKR1 (zeige PROKR1 Antikörper) and PROKR2 (zeige PROKR2 Antikörper)) act as main regulators of physiological functions of ovary, uterus, placenta, and testis. [REVIEW]
EG-VEGF and its receptor PKR1 (zeige PROKR1 Antikörper) might play a role in the pathogenesis of adrenocortical tumors and could serve as prognostic markers for this rare malignant disease.
The prognosis was poorer in colorectal cancers that expressed both PROK1 and VEGF relative to the cases that expressed only 1 protein, and the expression of both proteins was found to be an independent prognostic factor.
PROK1 levels in follicular fluid and fertilization culture media could constitute new predictive noninvasive markers of successful embryo implantation in conventional in vitro fertilization-embryo transfer
Simultaneous targeting of both angiogenic growth factors (VEGF (zeige VEGFA Antikörper)/PROK1) may prove more useful in colorectal cancer
High Prokineticin 1 protein expression is associated with sporadic colorectal cancer.
EG-VEGF is a new mediator of PPARgamma (zeige PPARG Antikörper) effects during pregnancy and bring new insights into the fine mechanism of trophoblast invasion.
Bv8 (zeige PROK2 Antikörper) and EG-VEGF, along with other factors such as VEGF-A (zeige VEGFA Antikörper), may maintain the integrity and also regulate proliferation of the blood vessels in the testis
This is the first report demonstrating that Prok-1 acts as a gut (zeige GUSB Antikörper) mucosa/mesenchyme-derived factor and maintains proliferation and differentiation of enteric neural crest cells.
EG-VEGF was mainly localised in the labyrinth in mouse placenta. It may have direct effect on both endothelial & trophoblastic cells & is likely to play an important role in mouse placentation.
Prokineticin-1 (Prok-1) works coordinately with glial cell line-derived neurotrophic factor (GDNF) to mediate proliferation and differentiation of enteric neural crest cells.
The protein encoded by this gene induces proliferation, migration, and fenestration (the formation of membrane discontinuities) in capillary endothelial cells derived from endocrine glands. It has little or no effect on a variety of other endothelial and non-endothelial cell types. Its expression is restricted to the steroidogenic glands (ovary, testis, adrenal, and placenta), is induced by hypoxia, and often complementary to the expression of vascular endothelial growth factor (VEGF), suggesting that these molecules function in a coordinated manner.
, black mamba toxin-related protein
, endocrine-gland-derived vascular endothelial growth factor