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Cell surface receptor for SEMA4A and for class 3 semaphorins, such as SEMA3A, SEMA3C and SEMA3E. Zusätzlich bieten wir Ihnen Plexin D1 Proteine (4) und Plexin D1 Kits (1) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 72 products:
Human Polyclonal PLXND1 Primary Antibody für CyTOF, FACS - ABIN4900364
Lu, Evans, Hirano, Liu: Dual role for Islet-1 in promoting striatonigral and repressing striatopallidal genetic programs to specify striatonigral cell identity. in Proceedings of the National Academy of Sciences of the United States of America 2014
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Human Polyclonal PLXND1 Primary Antibody für ELISA, IHC - ABIN268775
Parikh, Eisner, Venugopalan, Yang, Lam, Bhattacharya: Proteomic analyses of corneal tissue subjected to alkali exposure. in Investigative ophthalmology & visual science 2011
Show all 2 Pubmed References
Plexin D1 plays a role in collagen contraction in human lung fibroblasts.
Findings suggest that Plexin-D1/class III semaphorin (Sema3E (zeige SEMA3E Antikörper)) axis is triggered in systemic sclerosis (SSc (zeige CYP11A1 Antikörper)) endothelium.
finding that PLXND1 and REV3L (zeige REV3L Antikörper) mutations are responsible for a proportion of MBS (zeige PPP1R12A Antikörper) patients suggests that de novo mutations in other genes might account for other MBS (zeige PPP1R12A Antikörper) patients
The data indicate that Plexin-D1 operates in a cell context-specific fashion, mediating different synaptogenic outcomes depending upon neuron type.
Plxnd1 is a novel regulator of VAT growth, body fat distribution, and insulin (zeige INS Antikörper) sensitivity in both zebrafish and humans
The identification and characterization of SH3BP1 (zeige SH3BP1 Antikörper) as a novel downstream effector of Sema3E (zeige SEMA3E Antikörper)-PlexinD1 provides an explanation for how extracellular signals are translated into cytoskeletal changes and unique cell behavior.
It is therefore suggested that SEMA3C (zeige SEMA3C Antikörper) signaling, propagated through the heterodimer receptor plexin-D1/neuropilin (zeige NRP1 Antikörper), is important for truncus arteriosus septation
A critical role of Sema3E (zeige SEMA3E Antikörper)/Plexin D1 interaction in tumor resistance to apoptosis.
Strong expression of plexD1 was detected in endothelial cells of cervical cancer samples, yet no expression was seen in endothelial cells of normal cervical tissues, which suggests a potential role of PlexD1 in cervical cancer-associated angiogenesis
analysis of Sema3E (zeige SEMA3E Antikörper)/plexin-D1 mediated epithelial-to-mesenchymal transition in ovarian endometrioid cancer
Our results demonstrate that sema3e (zeige SEMA3E Antikörper)/plexin D1 modulates IS formation and Ag-scanning activities of thymocytes within thymic tissues.
Sema3E (zeige SEMA3E Antikörper)/PlexinD1 signaling controls the motogenic potential of Cajal-Retzius (CR) cells in vitro and in vivo. Absence of Sema3E (zeige SEMA3E Antikörper)/PlexinD1 signalling increased the migratory properties of CR cells.
Sema3E (zeige SEMA3E Antikörper)-PlexinD1 signaling is involved in the development of CNV. Stimulation of the pathway has therapeutic potential for CNV.
Proprioceptive sensory afferents that express PlexinD1 avoid forming monosynaptic connections with neurons in Sema3E (zeige SEMA3E Antikörper)(+) motor pools yet are able to form direct connections with neurons in Sema3E (zeige SEMA3E Antikörper)(off) motor pools.
beta1 integrin adhesion is controlled by the plexinD1-sema3E (zeige SEMA3E Antikörper) axis in mice
Data an association between Plexin-B2 (zeige PLXNB2 Antikörper) and Plexin-D1 with the negative regulation of IL-12 (zeige IL12A Antikörper)/IL-23p40 in DCs.
This study demonstrated that Sema3E (zeige SEMA3E Antikörper) and Plexin-D1 specify the degree of glutamatergic connectivity between a specific source and target in the complex circuitry of the basal ganglia.
a novel role of Sema3E-Plexin-D1 function in modulating angiogenesis via a VEGF-induced feedback mechanism
Sema3E (zeige SEMA3E Antikörper)-PlexinD1 signaling selectively suppresses disoriented angiogenesis in ischemic retinopathy in mice
Plexin-D1 is expressed by B lymphocytes. PlxnD1 deficient B cells fail to participate efficiently in the GC reaction. And, recall immune responses are attenuated in the absence of plexin-D1, leading to defective Ab production.
Sema3d (zeige SEMA3D Antikörper) regulates collective endothelial cell migration in zebrafish through two separate mechanisms. Mesenchymal Sema3d (zeige SEMA3D Antikörper) guides outgrowth of the common cardinal (zeige CARD8 Antikörper) vein via repulsion and signals through PlexinD1.
Somite expression of known vascular guidance cues, efnb2, sema3a2, and plexinD1 are disrupted, suggesting that the inter-somitic vessel vascular phenotype is due to disruption of these cues.
We show that proper blood vessel pathfinding requires the endothelial receptor PlexinD1 and semaphorin signals, and we identify mutations in plexinD1 in the zebrafish vascular patterning mutant out of bounds
Loss of plxnB2 (zeige PLXNB2 Antikörper) results in delayed ISV sprouting identical to that seen in sema3e (zeige SEMA3A Antikörper) morphants, while loss of plexinD1 in out of bounds (obd) mutants results in precocious ISV sprouting.
Cell surface receptor for SEMA4A and for class 3 semaphorins, such as SEMA3A, SEMA3C and SEMA3E. Plays an important role in cell-cell signaling, and in regulating the migration of a wide spectrum of cell types. Regulates the migration of thymocytes in the medulla. Regulates endothelial cell migration. Plays an important role in ensuring the specificity of synapse formation. Mediates anti-angiogenic signaling in response to SEMA3E. Required for normal development of the heart and vasculature.
, out of bounds