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MAOA is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Zusätzlich bieten wir Ihnen Monoamine Oxidase A Antikörper (91) und Monoamine Oxidase A Proteine (12) und viele weitere Produktgruppen zu diesem Protein an.
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Rat (Rattus) Monoamine Oxidase A ELISA Kit für Sandwich ELISA - ABIN432431
Baluchnejadmojarad, Rabiee, Zabihnejad, Roghani: Ellagic acid exerts protective effect in intrastriatal 6-hydroxydopamine rat model of Parkinson's disease: Possible involvement of ERβ/Nrf2/HO-1 signaling. in Brain research 2017
Social behavior was facilitated by chronic fluoxetine treatment in juvenile rhesus monkeys interacting with a familiar peer. The type of social behavior affected depended on the MAOA genotype of the monkey and its partner.
Results provide novel evidence that blood MAOA expression predicts alcohol consumption and that heavy alcohol use is linked to low MAOA expression in both the blood and NAc (zeige NLRP1 ELISA Kits) core.
study found the ability to consolidate sleep during the dark cycle was disrupted by prenatal iron deprivation, specifically in monkeys with the low-MAOA genotype
The effects of iron deficiency are dependent on MAOA genotype in terms of both direction and size of the effect.
rhMAOA-LPR (zeige FAS ELISA Kits) allele modifies the effects of maternal temperament on offspring's personality development.
brain protein levels of MAOB (zeige MAOB ELISA Kits) are normal or elevated in the three parkinsonian conditions-with MAOB (zeige MAOB ELISA Kits) increase generally associated with elevations in levels of astrocyte markers. Brain MAOA concentrations were not decreased in Parkinson's disease, progressive supranuclear palsy , or multiple system atroph with the exception of the atrophic putamen in MSA (zeige TPO ELISA Kits), despite loss of dopamine neurons that presumably contain this enzyme
MAOA and MAOB (zeige MAOB ELISA Kits) variants may contribute to the etiology of Attention deficit hyperactivity disorder (ADHD) in the Indo (zeige IDO1 ELISA Kits)-Caucasoid population and could be responsible for higher occurrence of ADHD in the boys.
Importance of MAOA and 5-HTTLPR (zeige SLC6A4 ELISA Kits) polymorphisms in the treatment of spousal violence and drinking in men in batterer intervention programs.
Polymorphisms of COMT (zeige COMT ELISA Kits) (c.649G>A), MAO-A (c.1460C>T), NET (c.1287G>A) Genes and the Level of Catecholamines, Serotonin in Patients with Parkinson's Disease
The influence of MAOA failed to reach statistical significance within any of the regression equations in predicting adult criminal behavior.
MAOA provides tumor cell growth advantages in the bone microenvironment by stimulating interleukin-6 (IL6 (zeige IL6 ELISA Kits)) release from osteoblasts, and triggers skeletal colonization by activating osteoclastogenesis through osteoblast production of RANKL (zeige TNFSF11 ELISA Kits) and IL6 (zeige IL6 ELISA Kits).
results highlight an association of ventral striatum MAO-A level with the functional connectivity of striatal regions linked to impulsive behavior in antisocial personality disorder
No significant association was found between severe agitation and MAOA uVNTR or MAOB (zeige MAOB ELISA Kits) rs1799836 polymorphism, revealing that these individual polymorphisms in MAO (zeige MAO ELISA Kits) genes are not related to severe agitation in subjects with schizophrenia and conduct disorder
The effects of parenting on self-control and offending are most pronounced for those who carry plasticity alleles for both MAOA and DAT1. Thus, MAOA and DAT1 may be implicated in offending because they increase the negative effects of parenting on self-control. Implications for theory are discussed.
These data suggest a close association between MAO-A-dependent reactive oxygen species generation, actin oxidation, and ventricular dysfunction.
cloning and characterization of monoamine oxidase A and B genes in pig
Two pools of ten bovine embryos, comprised of the 4-, 8- to 16-cell, morula, blastocyst, and expanded blastocyst stages, were collected. Total RNA was isolated, and the RT-PCR-RFLP technique was used to observe expression of the MAOA gene.
MAOA was seriously demethylated or showed aberrant methylation patterns in four aborted clones.
This study unravels a new link between MAO (zeige MAO ELISA Kits)-dependent H2O2 production and lysosomal dysfunction. Altogether, our findings demonstrate that the MAO-A/H2O2 axis has a negative impact on the elimination and recycling of mitochondria through the autophagy-lysosome pathway, which participates in cardiomyocyte death and heart failure
Results demonstrate that C17H11IOSe, a selective inhibitor of cortical MAO-A, elicited an antidepressant-like action in mice by interacting with the serotonergic system
data lead us to conclude that elevation of AP-1 (zeige JUN ELISA Kits) or NF-kB indirectly decreases MAO-A protein levels which, in turn, diminishes MAO-A ability in the VTA of the mesolimbic dopaminergic pathway
Huntington disease (zeige HTT ELISA Kits) neural cells exhibit increased Monoamine oxidase-A and Monoamine oxidases-B expression and activity
acute stress induces anxiety-like responses by affecting rapid dendritic remodeling in the pyramidal cells of orbitofrontal cortex and basolateral amygdala; furthermore, our data show that MAO-A and monoamine metabolism are required for these phenomena.
These findings demonstrate that regulation of monoamine levels by Mao (zeige MAO ELISA Kits) activity in beta cells is pivotal for physiological insulin (zeige INS ELISA Kits) secretion and that loss of MaoB (zeige MAOB ELISA Kits) expression may contribute to the beta cell dysfunction in type 2 diabetes.
Results suggest a role for KLF11 (zeige KLF11 ELISA Kits) in upregulating MAO-A in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The results of this study suggest that heightened dACC and amygdala activation and their connectivity are neuroaffective mechanisms underlying anger control in participants with the low-functioning allele of the MAOA gene.
These results suggest that early developmental enhancements in 5-HT (zeige DDC ELISA Kits) levels have long-term effects on the modulation of behavioral flexibility associated with MAO-A deficiency.
Under conditions of chronic hemodynamic stress, enhanced MAO-B (zeige MAOB ELISA Kits) activity is a major determinant of cardiac structural and functional disarrangement.
This gene is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Mutation of this gene results in Brunner syndrome. This gene has also been associated with a variety of other psychiatric disorders, including antisocial behavior. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
monoamine oxidase A
, amine oxidase [flavin-containing] A-like
, amine oxidase [flavin-containing] A
, monoamine oxidase type A
, putative monoamine oxidase A