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KLF6 encodes a member of the Kruppel-like family of transcription factors. Zusätzlich bieten wir Ihnen KLF6 Proteine (18) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 48 products:
Human Monoclonal KLF6 Primary Antibody für IHC, WB - ABIN2668584
Narla, DiFeo, Yao, Banno, Hod, Reeves, Qiao, Camacho-Vanegas, Levine, Kirschenbaum, Chan, Friedman, Martignetti: Targeted inhibition of the KLF6 splice variant, KLF6 SV1, suppresses prostate cancer cell growth and spread. in Cancer research 2005
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Human Monoclonal KLF6 Primary Antibody für ELISA, WB - ABIN560438
Lin, Jiang, Chuu: Caffeic acid phenethyl ester causes p21 induction, Akt signaling reduction, and growth inhibition in PC-3 human prostate cancer cells. in PLoS ONE 2012
Human Polyclonal KLF6 Primary Antibody für FACS, IF - ABIN390515
Sangodkar, Shi, DiFeo, Schwartz, Bromberg, Choudhri, McClinch, Hatami, Scheer, Kremer-Tal, Martignetti, Hui, Leung, Friedman, Narla: Functional role of the KLF6 tumour suppressor gene in gastric cancer. in European journal of cancer (Oxford, England : 1990) 2009
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study identifies a new mechanism by which KLF6 regulates NF-kappaB (zeige NFKB1 Antikörper) signaling, and how this mechanism is circumvented in glioblastoma through KLF6 loss.
our results established the significance of activating the KLF6-E2F1 (zeige E2F1 Antikörper) axis in aggressive primary metastatic clear cell renal cell carcinoma (zeige MOK Antikörper)
KLF6 overexpression suppressed the xenograft tumor growth.
KLF6 and KLF15 are targeted and suppressed in breast cancer cells by miR (zeige MLXIP Antikörper)-4262.
KLF6 acts as a tumor suppressor in CMM cells and miR (zeige MLXIP Antikörper)-4262 promotes the proliferation of CMM cells through KLF6-mediated EGFR (zeige EGFR Antikörper) inactivation and p21 (zeige CDKN1A Antikörper) upregulation.
These results suggest that KLF6 regulates MMP14 (zeige MMP14 Antikörper) transcription and is a critical player of the gene expression network triggered during endothelial repair.
KLF6 is a novel regulator of human villous trophoblast fusion.
KLF6 overexpression partially phenocopied chaetocin treatment in DDLPS cells and induced phenotypic changes that were consistent with adipocytic differentiation, suggesting that the effects of increased H3K9me3 may be mediated through KLF6
Study revealed that overexpression of miR (zeige MLXIP Antikörper)-101 inhibited the proliferation, cell migration, invasion, and promoted apoptosis of glioblastoma stem cells by directly targeting KLF6
Data show that MiR (zeige MLXIP Antikörper)-1301 and miR (zeige MLXIP Antikörper)-210 specifically target the tumor suppressive KLF6-FL isoform but not the oncogenic KLF6-SV1 isoform in hepatocellular carcinoma.
we identified novel transcriptional targets of KLF6 in HCC cells including VAV3, a known activator of the RAC1 small GTPase. Indeed, RAC1 activity is increased in KLF6-knockdown cells in a VAV3-dependent manner, and knockdown of either RAC1 or VAV3 impairs HCC cell migration. Together, our data demonstrate a novel function for KLF6 in constraining HCC dissemination through the regulation of a VAV3-RAC1 signaling axis.
Klf6 as required for developmental central nervous system myelination.Klf6 promotes oligodendrocyte differentiation and viability.
observations reveal that KLF6 repress BCL6 (zeige BCL6 Antikörper) to enhance macrophage inflammatory gene expression and function.
present studies on the cardiac function of KLF6 show a new mechanism whereby cardiomyocytes regulate cardiac fibrosis through transcriptional control of the extracellular matrix factor thrombospondin 4 (zeige THBS4 Antikörper)
downregulates expression and secretion of granulocyte macrophage colony-stimulating factor (zeige CSF2 Antikörper)
KLF6-dependent regulation of the cytochrome c (zeige CYCS Antikörper) oxidase assembly gene is critical for maintaining mitochondrial function and preventing podocyte apoptosis.
findings provide novel evidence highlighting KLF6 function in response to malignant transformation, suggesting the relevance of KLF6 in controlling cell proliferation and hindering tumorigenesis
KLF6 is required for optimal LPS (zeige TLR4 Antikörper)-induced pro-inflammatory gene expression, acting cooperatively with NF-kappaB (zeige NFKB1 Antikörper)
KLF6 is a novel regulator of hepatic glucose and lipid metabolism in fatty liver.
ALK1 (zeige ACVRL1 Antikörper) is upregulated in endothelial cells during vascular injury by a synergistic cooperative mechanism between KLF6 and specificity protein 1 (zeige SP1 Antikörper).
Klf6a promotes hematopoietic stem and progenitor cell maintenance through Ccl25b-Ccr7 (zeige CCR7 Antikörper) chemokine (zeige CCL1 Antikörper) signaling.
In the spinal cord, klf7 (zeige KLF7 Antikörper)- and klf6a-expressing cells are found in both the dorsal and ventral horns. Numerous sensory structures (e.g. auditory, lateral line, olfactory and visual) and several motor nuclei (e.g. oculomotor, trigeminal, and vagal motor nuclei) contain klf7 (zeige KLF7 Antikörper)- and/or klf6a-expressing cells
copeb/Klf6 is essential for the development of endoderm-derived organs
This gene encodes a member of the Kruppel-like family of transcription factors. The zinc finger protein is a transcriptional activator, and functions as a tumor suppressor. Multiple transcript variants encoding different isoforms have been found for this gene, some of which are implicated in carcinogenesis.
Kruppel-like factor 6
, core promoter element binding protein
, Krueppel-like factor 6
, krueppel-like factor 6
, B-cell-derived protein 1
, GC-rich binding factor
, GC-rich sites-binding factor GBF
, Kruppel-like zinc finger protein Zf9
, core promoter element-binding protein
, proto-oncogene BCD1
, protooncogene B-cell derived 1
, suppression of tumorigenicity 12 (prostate)
, suppressor of tumorigenicity 12 protein
, transcription factor Zf9
, immediate early response, erythropoietin 1