Junctional Adhesion Molecule 3 (JAM3) ELISA Kits

Tight junctions represent one mode of cell-to-cell adhesion in epithelial or endothelial cell sheets, forming continuous seals around cells and serving as a physical barrier to prevent solutes and water from passing freely through the paracellular space. Zusätzlich bieten wir Ihnen JAM3 Antikörper (103) und JAM3 Proteine (13) und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
JAM3 83964 Q9D8B7
JAM3 83700 Q9BX67
JAM3 315509 Q68FQ2
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Top JAM3 ELISA Kits auf antikoerper-online.de

Showing 4 out of 10 products:

Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Anbieter Lieferzeit Preis Details
Human 6.2 pg/mL 15.625-1000 pg/mL 96 Tests Anmelden zum Anzeigen 15 bis 17 Tage
$983.85
Details
Ratte 1.56 pg/mL 6.25-400 pg/mL Typical standard curve 96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$910.56
Details
Maus 1.95 pg/mL 7.8-500 pg/mL Typical standard curve 96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$910.56
Details
Human 6.5 pg/mL 15.62 pg/mL - 1000 pg/mL   96 Tests Anmelden zum Anzeigen 8 bis 12 Tage
$840.25
Details

Weitere ELISA Kits für JAM3 Interaktionspartner

Zebrafish Junctional Adhesion Molecule 3 (JAM3) Interaktionspartner

  1. interaction between Jamb (zeige JAM2 ELISA Kits) and Jamc expressed by neighbouring cells is essential for fusion. jamc is ectopically expressed in prdm1a (zeige PRDM1 ELISA Kits) mutant slow muscle precursors, which inappropriately fuse with other myocytes.

Mouse (Murine) Junctional Adhesion Molecule 3 (JAM3) Interaktionspartner

  1. JAM-B (zeige JAM2 ELISA Kits)/JAM-C mediated interaction between endothelial cells and stellate cells stabilizes vessel walls and may control the sinusoidal diameter.

  2. Study provides evidence that JAM-C downregulation may contribute to acute pancreatitis-associated lung injury via reverse transendothelial migration of neutrophils.

  3. Endothelial-specific deletion of JAM-C promoted endothelial cell sprouting, and consequently vessel normalisation and revascularisation of the hypoxic retina without altering pathologic neovascularisation.

  4. JAM-C blockade can finely-tune the innate cell migration and accelerate the consequent immune response to L. major without changing the type of the T helper cell response.

  5. Function of Jam-B (zeige JAM2 ELISA Kits)/Jam-C interaction in homing and mobilization of human and mouse hematopoietic stem and progenitor cells.

  6. These findings provide evidence for a role for endothelial cell JAM-C in tumor growth and aggressiveness as well as recruitment of pericytes to newly formed blood vessels in a model of ovarian cancer.

  7. JAM-C might be involved in the final steps of trafficking and transmigration of antigen-specific autoaggressive T-cells to the islets of Langerhans.

  8. study suggests that JAM-C(-/-) C57BL/6 mice model the important role for JAM-C in brain development and CSF (zeige CSF2 ELISA Kits) homeostasis as recently observed in humans with a loss-of-function mutation in JAM-C

  9. Data show that junctional adhesion molecule-B (JAM-B (zeige JAM2 ELISA Kits)) expressed by endothelial cells contributes to murine B16 melanoma cells metastasis through its interaction with junctional adhesion molecule-C (JAM-C) on tumor cells.

  10. Results indicate that in vivo JAM-C shows enrichment at the apical surface and therefore is asymmetrically distributed during cell divisions.

Human Junctional Adhesion Molecule 3 (JAM3) Interaktionspartner

  1. The role of JAM-C in the engraftment of human lymphoma B cells in mice was investigated. The binding of anti-JAM-C antibodies inhibited the phosphorylation of ERK1/2 (zeige MAPK1/3 ELISA Kits), without affecting other signaling pathways, identifying for the first time the intracellular MAPK (zeige MAPK1 ELISA Kits) cascade as the JAM-C-driven signaling pathway in JAM-C(+) B cells.

  2. blocking JAM-C can reduce the number of atherogenic monocytes/macrophages in plaques by emigration.

  3. Palmitoylation of JAM-C promotes its localization to tight junctions and inhibits transwell migration of A549 lung cancer cells.

  4. Study provides evidence that JAM-C downregulation may contribute to acute pancreatitis-associated lung injury via reverse transendothelial migration of neutrophils.

  5. Suggest JAM3-M4 methylation as a biomarker for diagnosis of preneoplastic and neoplastic lesions of the cervix.

  6. JAM-C inactivation in endothelial cells resulted in increased spreading on fibronectin and enhanced sprouting in vitro in a manner dependent on beta1-integrin and on the activation of the small GTPase RAP1.

  7. Function of Jam-B (zeige JAM2 ELISA Kits)/Jam-C interaction in homing and mobilization of human and mouse hematopoietic stem and progenitor cells.

  8. indicate that JAM-C may be a therapeutic target for preventing and treating lymphatic metastases

  9. These findings provide evidence for a role for endothelial cell JAM-C in tumor growth and aggressiveness as well as recruitment of pericytes to newly formed blood vessels in a model of ovarian cancer.

  10. Our study confirms the importance of JAM3 as a component of the junctional complexes and its deficiency leading to a distinctive and catastrophic neonatal presentation of cataracts and hemorrhagic destruction of the brain.

JAM3 Antigen-Profil

Beschreibung des Gens

Tight junctions represent one mode of cell-to-cell adhesion in epithelial or endothelial cell sheets, forming continuous seals around cells and serving as a physical barrier to prevent solutes and water from passing freely through the paracellular space. The protein encoded by this immunoglobulin superfamily gene member is localized in the tight junctions between high endothelial cells. Unlike other proteins in this family, the this protein is unable to adhere to leukocyte cell lines and only forms weak homotypic interactions. The encoded protein is a member of the junctional adhesion molecule protein family and acts as a receptor for another member of this family. A mutation in an intron of this gene is associated with hemorrhagic destruction of the brain, subependymal calcification, and congenital cataracts. Alternative splicing results in multiple transcript variants.

Genbezeichner und Symbole assoziert mit JAM3

  • junctional adhesion molecule 3 (JAM3) Antikörper
  • junctional adhesion molecule 3b (jam3b) Antikörper
  • junctional adhesion molecule 3a (jam3a) Antikörper
  • junction adhesion molecule 3 (Jam3) Antikörper
  • junctional adhesion molecule 3 (Jam3) Antikörper
  • 1110002N23Rik Antikörper
  • JAM-2 Antikörper
  • JAM-3 Antikörper
  • JAM-C Antikörper
  • jam3 Antikörper
  • JAMC Antikörper
  • jamc.2 Antikörper
  • Jcam3 Antikörper
  • sr:nyz155 Antikörper
  • wu:fb30h11 Antikörper
  • wu:fc08c06 Antikörper
  • wu:fc13e04 Antikörper
  • wu:fc25g11 Antikörper

Bezeichner auf Proteinebene für JAM3

junctional adhesion molecule 3 , jamc , jamc.1 , si:ch211-277g22.3 , JAM-C-like protein , junction cell adhesion molecule 3 , junction cell adhesion molecule C , junctional adhesion molecule C , JAM-3 , JAM-C

GENE ID SPEZIES
419736 Gallus gallus
451678 Pan troglodytes
569217 Danio rerio
797651 Danio rerio
83964 Mus musculus
83700 Homo sapiens
489271 Canis lupus familiaris
513412 Bos taurus
315509 Rattus norvegicus
Ausgewählte Anbieter für JAM3 (JAM3) ELISA Kits
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