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GFAP encodes one of the major intermediate filament proteins of mature astrocytes. Zusätzlich bieten wir Ihnen GFAP Kits (54) und GFAP Proteine (31) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 826 products:
Human Monoclonal GFAP Primary Antibody für IF, WB - ABIN1449159
Macaulay, Forbes: Assembly of the nuclear pore: biochemically distinct steps revealed with NEM, GTP gamma S, and BAPTA. in The Journal of cell biology 1996
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Human Monoclonal GFAP Primary Antibody für IF, WB - ABIN1449150
Kawajiri, Yasui, Goto, Tatsuka, Takahashi, Nagata, Inagaki: Functional significance of the specific sites phosphorylated in desmin at cleavage furrow: Aurora-B may phosphorylate and regulate type III intermediate filaments during cytokinesis coordinatedly with Rho-kinase. in Molecular biology of the cell 2003
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Cow (Bovine) Monoclonal GFAP Primary Antibody für IHC (fro), IF - ABIN111187
Akat, Mennel, Kremer, Gassler, Bleck, Kartenbeck: Molecular characterization of desmosomes in meningiomas and arachnoidal tissue. in Acta neuropathologica 2003
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Human Polyclonal GFAP Primary Antibody für EIA - ABIN358488
Quintanar, Franco, Salinas: Detection of glial fibrillary acidic protein and neurofilaments in the cerebrospinal fluid of patients with neurocysticercosis. in Parasitology research 2003
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Human Polyclonal GFAP Primary Antibody für EIA, IHC (p) - ABIN4620465
Shiroma, Kanazawa, Kato, Shimozawa, Imamura, Ito, Ohtani, Oka, Wakabayashi, Iai, Sugai, Sasaki, Kaga, Ohta, Tsujino: Molecular genetic study in Japanese patients with Alexander disease: a novel mutation, R79L. in Brain & development 2003
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Human Monoclonal GFAP Primary Antibody für IHC (fro), IHC (p) - ABIN1107345
Rodriguez, Gauthier, Bertini, Bugiani, Brenner, Nguyen, Goizet, Gelot, Surtees, Pedespan, Hernandorena, Troncoso, Uziel, Messing, Ponsot, Pham-Dinh, Dautigny, Boespflug-Tanguy: Infantile Alexander disease: spectrum of GFAP mutations and genotype-phenotype correlation. in American journal of human genetics 2001
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Dog (Canine) Polyclonal GFAP Primary Antibody für EIA, WB - ABIN782998
Korolainen, Auriola, Nyman, Alafuzoff, Pirttilä: Proteomic analysis of glial fibrillary acidic protein in Alzheimer's disease and aging brain. in Neurobiology of disease 2005
Human Monoclonal GFAP Primary Antibody für IHC (fro), IF - ABIN238409
Porchet, Probst, Bouras, Dráberová, Dráber, Riederer: Analysis of glial acidic fibrillary protein in the human entorhinal cortex during aging and in Alzheimer's disease. in Proteomics 2003
There was significantly more GFAP immunoreactivity in the prefrontal cortex and hippocampus of aged animals compared to adult or middle-aged animals.
serum levels of GFAP were significantly lower in autism spectrum disorders than controls
We found downregulation of GFAP mRNA and protein in the mediodorsal thalamus and caudate nucleus of depressed suicides compared with controls, whereas GFAP expression in other brain regions was similar between groups. Furthermore, a regional comparison including all samples revealed that GFAP expression in both subcortical regions was, on average, between 11- and 15-fold greater than in cerebellum and neocortex.
GFAP is upregulated following an insult or injury to the brain, additionally making it an indicator of CNS pathology.
This study demonistrated that the density of GFAP-immunoreactive astrocytes is decreased in left hippocampi in major depressive disorder
This study demonstrated that GFAP as a promising biomarker to distinguish ischemic stroke from intracerebral hemorrhage.
The levels of GFAP in Alzheimer's disease, dementia with Lewy bodies, and frontotemporal lobar degeneration patients were significantly higher than those in the healthy control subjects.
GFAP is significantly associated with outcome, but it does not add predictive power to commonly used prognostic variables in a population of patients with TBI of varying severities.
Neither duplications nor deletions of GFAP were found, suggesting that GFAP coding-region rearrangements may not be involved in Alexander disease or Alexanderrelated leukoencephalopathies.
The data suggest that human vitreous body GFAP is a protein biomarker for glial activation in response to retinal pathologies.
Studied diagnostic Value of Serum Levels of GFAP, pNF-H, and NSE (zeige ENO2 Antikörper) Compared With Clinical Findings in Severity Assessment of Human Traumatic Spinal Cord Injury.
Isolation of an evolutionary conserved novel GFAP isoform, GFAPkappa, produced by alternative splicing and polyadenylation of the 3'-region of the human GFAP pre-mRNA is described.
This study demonstrated the GFAP-ApoE4 mice exhibited motor impairments when compared to GFAP-ApoE3 and wild-type mice.
PINK1 (zeige PINK1 Antikörper) deficiency causes defects in GFAP-positive astrogliogenesis during brain development.
Gnasxl (zeige GNAS Antikörper) deficiency does not directly affect glial development in the hypothalamus, since it is expressed in neurons, and Gfap-positive astrocytes and tanycytes appear normal during early postnatal stages.
Induction of glial cytokine expression was sequential, aligned with active sickness behavior, and preceded increased Iba-1 (zeige AIF1 Antikörper) or GFAP immunoreactivity after lipopolysaccharide challenge
Study provides a mechanistic link between the GFAP mutations/overexpression and the symptoms in those affected with Type II Alexander disease
Study described GFAP-expressing non-myelinating Schwann cells in the lung, validated a transgenic mouse line that drives expression of cre under a GFAP promoter
findings thus show that the inability to produce GFAP and Vim (zeige VIM Antikörper) affects normal retinal physiology and that the effect of IF deficiency on retinal cell survival differs, depending on the underlying pathologic condition
CUL4B (zeige CUL4B Antikörper) as a negative regulator of GFAP expression during neural development.
Astrocytes deficient of GFAP and vimentin (zeige VIM Antikörper) showed decreased Notch (zeige NOTCH1 Antikörper) signal sending competence and altered expression of Notch (zeige NOTCH1 Antikörper) signaling pathway-related genes
Absence of GFAP, or both GFAP and vimentin (zeige VIM Antikörper), alters Alzheimer's disease-induced changes in gene expression profile of astrocytes, showing a compensation of the decrease of neuronal support genes and a trend for a higher inflammatory expression profile
The distribution of GFAP immunoreactivity implies that enteric glia are widespread in the fish gastrointestinal tract.
Generation of transgenic zebrafish that express green fluorescent protein (GFP) in glial cells driven by the zebrafish glial fibrillary acidic protein (GFAP) regulatory elements.
Cells expressing the two reporters display radial glial morphology, colocalize with the NSC marker Sox2 (zeige SOX2 Antikörper), undergo proliferation, and are capable of self-renewal within the matrix of distinct thickness in the telencephalon.
This gene encodes one of the major intermediate filament proteins of mature astrocytes. It is used as a marker to distinguish astrocytes from other glial cells during development. Mutations in this gene cause Alexander disease, a rare disorder of astrocytes in the central nervous system. Alternative splicing results in multiple transcript variants encoding distinct isoforms.
glial fibrillary acidic protein
, glial fibrillary acidic protein alpha
, intermediate filament
, intermediate filament protein
, zrf-1 antigen