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CFHR1 encodes a secreted protein belonging to the complement factor H protein family. Zusätzlich bieten wir Ihnen CFHR1 Kits (14) und CFHR1 Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
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To our knowledge, this is the first evaluation of the involvement of the CFHR3 (zeige CFHR3 Antikörper)/CFHR1 deletion and age-related macular degeneration in CFH (zeige CFH Antikörper) Y402H polymorphism Brazilian patients.
These results identify C-reactive protein (zeige CRP Antikörper) as a ligand for FHR-1 and suggest that FHR-1 enhances, rather than inhibits, complement activation, which may explain the protective effect of FHR-1 deficiency in age-related macular degeneration.
The CFHR1 level in plasma of T2DM patients were significantly higher than that of the healthy controls.
CFHL-1 is a CFH (zeige CFH Antikörper) gene splice variant.
We investigated whether the causal sequence variant resides in the CFH (zeige CFH Antikörper) gene or the neighboring complement factor H-related 1 (CFHR1) gene, and fine mapping Implicates a deletion of CFHR1 in protection from IgA nephropathy in Han Chinese
Next-generation sequencing of the CFH (zeige CFH Antikörper) region identified putatively functional variants (missense, splice site and indel) on the four common haplotypes. We found no expression of any of the five CFH (zeige CFH Antikörper)-related genes in the retina or RPE (zeige RPE Antikörper)/Choroid/Sclera, in contrast to the liver, which is the main source of the circulating proteins. [CFHR1]
Studies indicate that complement factor H (zeige CFH Antikörper)-related proteins (FHR1-5) may enhance complement activation, with important implications for the role of these proteins in disease.
These results suggest that the combination of quantitative and qualitative variations in the complement proteins encoded by CFH (zeige CFH Antikörper), CFHR3 (zeige CFHR3 Antikörper) and CFHR1 genes is key for the association of these haplotypes with disease.
Genetic variants in CFH, CFHR3, and CFHR1 affect complement activation and thereby predispose patients to develop IgA nephropathy.
An average of 15.2% of factor H (zeige CFH Antikörper)-autoantibody positive individuals with rheumatic diseases or hemolytic uremic syndrome had homozygous deficiency of CFHR1.
This gene encodes a secreted protein belonging to the complement factor H protein family. It binds to Pseudomonas aeruginosa elongation factor Tuf together with plasminogen, which is proteolytically activated. It is proposed that Tuf acts as a virulence factor by acquiring host proteins to the pathogen surface, controlling complement, and facilitating tissue invasion. Mutations in this gene are associated with an increased risk of atypical hemolytic-uremic syndrome.
complement component factor h-like 1
, complement factor H-related protein
, complement factor H-related protein 1
, complement factor H-related 1
, H factor (complement)-like 1
, H factor (complement)-like 2
, H-factor-like 1
, complement factor H-related 1 pseudogene
, h factor-like protein 1