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The protein encoded by BLVRA belongs to the biliverdin reductase family, members of which catalyze the conversion of biliverdin to bilirubin in the presence of NADPH or NADH. Zusätzlich bieten wir Ihnen Biliverdin Reductase Kits (17) und Biliverdin Reductase Proteine (16) und viele weitere Produktgruppen zu diesem Protein an.
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Human Polyclonal Biliverdin Reductase Primary Antibody für IHC, IHC (p) - ABIN4284633
Gromov, Espinoza, Talman, Honma, Kroman, Timmermans Wielenga, Moreira, Gromova: FABP7 and HMGCS2 are novel protein markers for apocrine differentiation categorizing apocrine carcinoma of the breast. in PLoS ONE 2014
Suggest that Gata-1 (zeige GATA1 Antikörper) and Nrf2a (zeige NFE2L2 Antikörper) play differential roles in regulating the heme degradation enzymes hmox1a/bvra/bvrb (zeige BLVRB Antikörper) during an early developmental period of heightened cellular stress
Genetic polymorphisms of the UGT1A1 (zeige UGT1A1 Antikörper) promoter, specifically the T-3279G phenobarbital-responsive enhancer module and (TA)7 dinucleotide repeat, as well as the intron and coding region variants of the OATP2 (zeige SLCO1B1 Antikörper), HMOX1 (zeige HMOX1 Antikörper), and BLVRA genes, were significantly higher among the cases than the controls.
BLVRA mRNA levels in the liver as well as in peripheral blood leukocytes are significantly higher in hepatocellular carcinoma patients most likely as a feedback mechanism to control increased oxidative stress associated with HCC (zeige FAM126A Antikörper) progression.
Data suggest that isoenzymes BVRA and BVRB (zeige BLVRB Antikörper) play different roles in energy metabolism and in pathogenesis of abdominal obesity and hypertriglyceridemia. [REVIEW]
Interactions of HO-2 (zeige HMOX2 Antikörper) with CPR (zeige POR Antikörper) and BVR, were evaluated.
Increased biliverdin reductase expression is associated with multidrug resistance in leukemia.
Our results suggest that patients with chronic HCV infection significantly upregulate BLVRA expression in PBL.
The current study reports increased levels of both HO-1 (zeige HMOX1 Antikörper) and BVR-A in plasma from probable Alzheimer disease patients, as a result of the increased oxidative environment.
rs699512 (Thr3Ala), the only common non-synonymous SNP within BLVRA, reduced the risk of essential hypertension in Kazaks.
A significant increase of nitrated BVRA was demonstrated only in Alzheimer's disease and mild cognitive impairment hippocampi
A homozygous BLVRA inactivating mutation is associated with the appearance of green jaundice accompanying cholestasis episodes.
These findings reveal a novel BVRA-GSKbeta-PPARalpha (zeige PPARA Antikörper) axis that regulates hepatic lipid metabolism and may provide unique targets for the treatment of non-alcoholic fatty liver disease.
BVR-A integrates both oxidative/nitrosative stress- and insulin (zeige INS Antikörper)-mediated signaling, both mechanisms dysregulated in the Alzheimer disease brain.
The biliverdin reductase A positive macrophage is a source of anti-inflammatory cytokine IL-10 (zeige IL10 Antikörper) in injured kidney.
Lack of p53 (zeige TP53 Antikörper) decreases basal oxidative stress levels in the brain through upregulation of thioredoxin-1 (zeige TXN Antikörper), biliverdin reductase-A, manganese superoxide dismutase (zeige SOD2 Antikörper), and nuclear factor kappa-B.
biliverdin reductase has a role in regulating the inflammatory response to endotoxin that requires eNOS (zeige NOS3 Antikörper)-derived NO and TLR4 (zeige TLR4 Antikörper) signaling in macrophages
biliverdin reductase mediates biliverdin-induced anti-inflammatory effects via phosphatidylinositol 3-kinase and Akt (zeige AKT1 Antikörper)
Inhibition of biliverdin reductase increases ANG II (zeige AGT Antikörper)-dependent superoxide levels in cultured renal tubular epithelial cells.
biliverdin reductase A (BLVRA) was identified as a partner of ELSPBP1 (zeige ELSPBP1 Antikörper) by immunoprecipitation followed by tandem mass spectrometry.
The protein encoded by this gene belongs to the biliverdin reductase family, members of which catalyze the conversion of biliverdin to bilirubin in the presence of NADPH or NADH. Mutations in this gene are associated with hyperbiliverdinemia. Alternatively spliced transcript variants have been found for this gene.
biliverdin reductase A
, biliverdin IX-beta reductase
, biliverdin reductase
, biliverdin reductase a
, BVR A
, biliverdin-IX alpha-reductase