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BNIP3L is a member of the BCL2/adenovirus E1B 19 kd-interacting protein (BNIP) family. Zusätzlich bieten wir Ihnen BCL2/adenovirus E1B 19kDa Interacting Protein 3-Like Proteine (4) und BCL2/adenovirus E1B 19kDa Interacting Protein 3-Like Kits (1) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 113 products:
Human Polyclonal BNIP3L Primary Antibody für WB - ABIN264993
Matsushima, Fujiwara, Takahashi, Minaguchi, Eguchi, Tsujimoto, Suzumori, Nakamura: Isolation, mapping, and functional analysis of a novel human cDNA (BNIP3L) encoding a protein homologous to human NIP3. in Genes, chromosomes & cancer 1998
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Human Polyclonal BNIP3L Primary Antibody für EIA, FACS - ABIN950671
Novak, Kirkin, McEwan, Zhang, Wild, Rozenknop, Rogov, Löhr, Popovic, Occhipinti, Reichert, Terzic, Dötsch, Ney, Dikic: Nix is a selective autophagy receptor for mitochondrial clearance. in EMBO reports 2009
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Human Monoclonal BNIP3L Primary Antibody für IF, ELISA - ABIN560088
Nakamura, Kitamura, Shinogi, Yoshida, Goda, Murai, Kamino, Arakawa: BNIP3 and NIX mediate Mieap-induced accumulation of lysosomal proteins within mitochondria. in PLoS ONE 2012
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Human Polyclonal BNIP3L Primary Antibody für IHC, IHC (p) - ABIN4285004
Li, Zhang, Zhuang, Chen, Chen, Tian, Wu, Li, Wang, Zhang, Chen, Li, Zhao, Sui, Hu, Feng: MicroRNA-137 is a novel hypoxia-responsive microRNA that inhibits mitophagy via regulation of two mitophagy receptors FUNDC1 and NIX. in The Journal of biological chemistry 2014
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Human Polyclonal BNIP3L Primary Antibody für IHC (p), IHC - ABIN269357
Passer, Nancy-Portebois, Amzallag, Prieur, Cans, Roborel de Climens, Fiucci, Bouvard, Tuynder, Susini, Morchoisne, Crible, Lespagnol, Dausset, Oren, Amson, Telerman: The p53-inducible TSAP6 gene product regulates apoptosis and the cell cycle and interacts with Nix and the Myt1 kinase. in Proceedings of the National Academy of Sciences of the United States of America 2003
Human Polyclonal BNIP3L Primary Antibody für FACS, IF - ABIN655731
Wang, McDonnell, Hebbring, Cunningham, St Sauver, Cerhan, Isaya, Schaid, Thibodeau: Polymorphisms in mitochondrial genes and prostate cancer risk. in Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2008
p75(NTR (zeige NGFR Antikörper)) and NIX may play critical roles in intracerebral hemorrhage-induced neuronal apoptosis in vitro and in vivo.
Data suggest that targeting BNIP3L protein in wild-type p53 tumor suppressor (zeige TP53 Antikörper) colon cancer cells is an anticancer strategy activating iron depletion signaling and the mitophagy-related cell death pathway.
regulates mitophagy during hypoxia, whereas NIX is required for mitophagy during development of the erythroid lineage.
Nix protein positively regulates NF-kappaB (zeige NFKB1 Antikörper) activation in gliomas
The physical interaction of Mieap (zeige SPATA18 Antikörper), BNIP3 (zeige BNIP3 Antikörper) and NIX at the mitochondrial outer membrane may play a critical role in the translocation of lysosomal proteins from the cytoplasm to the mitochondrial matrix.
mitochondrial ROS (zeige ROS1 Antikörper) and NIX are essential factors for Mieap (zeige SPATA18 Antikörper)-induced accumulation of lysosome-like organelles within mitochondria
Nix functions as an autophagy receptor, which mediates mitochondrial clearance after mitochondrial damage and during erythrocyte differentiation
NIX binds to TSAP6 (zeige STEAP3 Antikörper) in tumor cells and has a role in apoptosis
The proapoptotic factor Nix is a highly regulated effector of growth during terminal erythroid maturation.
Bnip3L, capable of attenuating tumorigenicity, mediates p53 (zeige TP53 Antikörper)-dependent apoptosis under hypoxia.
studies highlight a new hypoxia-induced pathway in which NANOG (zeige NANOG Antikörper) activates BNIP3L expression, contributing to autophagy induction in hypoxic tumor cells and their resistance to killing by CTL
miR (zeige MLXIP Antikörper)-30e protects mitochondria and podocytes from aldosterone induced apoptosis by targeting BNIP3L expression.
Bnip3L and caspase-12 (zeige CASP12 Antikörper) identified by the PCR arrays at both catagen stages were additionally localized using immunofluorescence and were reported in physiological hair development for the first time.
our study demonstrates that BNIP3L, as a substrate of PARK2 (zeige PARK2 Antikörper), promotes mitophagy in the PINK1 (zeige PINK1 Antikörper)/PARK2 (zeige PARK2 Antikörper) pathway associated with PD pathogenesis.
Suggest Nix is a novel mediator of norepinephrine-induced fibrosis.
MicroRNA-137 has a role in inhibiting mitophagy via regulation of two mitophagy receptors FUNDC1 (zeige FUNDC1 Antikörper) and NIX
-stimulated cytoplasmic-nuclear shuttling of the alternately spliced non-mitochondrial Nix isoform and uncover a role for sNix as a modulator of TNFalpha/NFkappaB-stimulated cardiac gene expression
BNIP3L activity localizes to a small region in its cytoplasmic domain, the minimal essential region (MER (zeige ERH Antikörper)).
These results establish Nix as a critical mediator of beta cell apoptosis and programmed necrosis in Pdx1 (zeige PDX1 Antikörper)-deficient diabetes.
molecular events responsible for the different phases of mitophagy and placed Nix upstream of the events
This gene is a member of the BCL2/adenovirus E1B 19 kd-interacting protein (BNIP) family. It interacts with the E1B 19 kDa protein, which protects cells from virally-induced cell death. The encoded protein also interacts with E1B 19 kDa-like sequences of BCL2, another apoptotic protector. This protein counteracts the apoptotic inducer BNIP3 and may play a role in tumor suppression.
BCL2/adenovirus E1B 19 kDa protein-interacting protein 3-like
, BCL2/adenovirus E1B 19 kDa protein-interacting protein 3A
, BCL2/adenovirus E1B 19-kd protein-interacting protein 3a
, NIP3-like protein X
, adenovirus E1B19k-binding protein B5
, BCL2/adenovirus E1B 19kD-interacting protein 3-like
, BCL2/adenovirus E1B 19 kDa-interacting protein 3-like
, BCL2/adenovirus E1B 19kDa-interacting protein 3-like