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Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. Zusätzlich bieten wir Ihnen APOH Kits (79) und APOH Proteine (35) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 151 products:
Human Polyclonal APOH Primary Antibody für IHC, IHC (p) - ABIN4281228
Häggmark, Neiman, Drobin, Zwahlen, Uhlén, Nilsson, Schwenk: Classification of protein profiles from antibody microarrays using heat and detergent treatment. in New biotechnology 2012
Show all 2 Pubmed References
Human Polyclonal APOH Primary Antibody für IHC, IHC (p) - ABIN4281227
Kato, Nicholson, Neiman, Rantalainen, Holmes, Barrett, Uhlén, Nilsson, Spector, Schwenk: Variance decomposition of protein profiles from antibody arrays using a longitudinal twin model. in Proteome science 2011
Human Polyclonal APOH Primary Antibody für ELISA, WB - ABIN4281229
Hirose, Williams, Alberts, Furie, Chartash, Jain, Sison, Lahita, Merrill, Cucurull, Gharavi, Sammaritano, Salmon, Hashimoto, Sawada, Chu, Gregersen, Chiorazzi: A role for the polymorphism at position 247 of the beta2-glycoprotein I gene in the generation of anti-beta2-glycoprotein I antibodies in the antiphospholipid syndrome. in Arthritis and rheumatism 1999
Human Polyclonal APOH Primary Antibody für WB - ABIN513421
Mulla, Brosens, Chamley, Giles, Pericleous, Rahman, Joyce, Panda, Paidas, Abrahams: Antiphospholipid antibodies induce a pro-inflammatory response in first trimester trophoblast via the TLR4/MyD88 pathway. in American journal of reproductive immunology (New York, N.Y. : 1989) 2009
These results suggested that CRP (zeige CRP Antikörper)/oxidized low density lipoprotein/beta 2-Glycoprotein I complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (zeige MAPK14 Antikörper) and NFkappaB signaling pathways.
TLR2 (zeige TLR2 Antikörper) increase the stimulating effect of beta2GP1/anti-beta2GP1 complex on the expression of TNF-alpha (zeige TNF Antikörper) in mouse peritoneal macrophages
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury.
These results indicate that both NF-kappaB (zeige NFKB1 Antikörper) and c-Jun/AP-1 (zeige JUN Antikörper) are involved in regulating anti-beta2GPI-induced expression of prothrombotic and proinflammatory molecules in vivo.
Phospholipid scramblase 1 regulates phosphatidylserine exposure in response to oxygen stress, leading to beta2-glycoprotein I and IgM binding and lipid-mediated, inflammatory responses.
beta2-GPI-D-I-tDCs may offer a novel approach for developing therapy for APS (zeige SH2B2 Antikörper) patients
Deletion of beta2 GPI accelerates and potentiates the autoimmune phenotype in male BXSB.Yaa mice
Interaction of beta2-glycoprotein I with lipopolysaccharide leads to Toll-like receptor 4 (TLR4 (zeige TLR4 Antikörper))-dependent activation of macrophages
Data are consistent with pathogenic mechanisms in antiphospholipid syndrome involving both neutralization of beta(2)-glycoprotein I (beta(2)GPI) function and beta(2)GPI-immunoglobulin complex formation.
beta-2-glycoprotein 1 plays an important physiologic role in the recognition of dying cells
we have demonstrated a pathogenic role for aPL (zeige FASL Antikörper) containing samples, mediated via aPL (zeige FASL Antikörper)-2bGPI interactions, resulting in activation of the pro-apoptotic p38 MAPK (zeige MAPK14 Antikörper) pathway.
Pretransplant IgA-aB2GP1 was the main risk factor for graft thrombosis and early graft loss after renal transplantation.
These results suggest that antibodies directed against domain 1 of beta-2-glycoprotein 1 may contribute to antiphospholipid syndrome risk assessment, due to their association with the diagnosis of systemic autoimmunity and with more aggressive triple antiphospholipid antibody profiles, such as triple positivity and lupus anticoagulant activity.
Circulating Immune Complexes of IgA Bound to Beta 2 Glycoprotein are Strongly Associated with the Occurrence of Acute Thrombotic Events.
Genome-wide significant results in or near the APOH gene on chromosome 17 have been found for plasma apolipoprotein H levels in middle-aged and older adults.
Prevalence and pathogenicity of IgA aB2GPI antibodies may vary from a population of ESRD patients to another.
The association of IL28B (zeige IL28B Antikörper) and APOH single nucleotide polymorphisms (SNPs) with sustained virological response and of ITPA (zeige ITPA Antikörper) SNPs with anemia related phenotypes.
We identified APOH as an obesity-resistance gene that interacts with FTO (zeige FTO Antikörper) rs9939609 to more than double the occurrence of thinness.
Trp316Ser variant (rs1801690) near the apolipoprotein H (beta2-glycoprotein-I) gene was associated with serum lipid parameters in Chinese volunteers. Suggest that there may be have racial/ethnic-and/or gender-specificity.
Plasma apolipoprotein H limits HCV replication and associates with response to NS3 protease inhibitor-based therapy.
Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.
, apolipoprotein H (beta-2-glycoprotein I)
, APC inhibitor
, activated protein C-binding protein
, beta-2-glycoprotein 1
, beta-2-glycoprotein I
, anticardiolipin cofactor