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AMHR2 encodes the receptor for the anti-Mullerian hormone (AMH) which, in addition to testosterone, results in male sex differentiation. Zusätzlich bieten wir Ihnen AMHR2 Antikörper (77) und AMHR2 Kits (5) und viele weitere Produktgruppen zu diesem Protein an.
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Results from these studies indicate that AMH (zeige AMH Proteine) signaling via AMHR2 plays a role in both regulating granulosa cell proliferation and preventing granulosa cells from 5- to 8-mm follicles from undergoing premature differentiation before follicle selection.
These findings indicate the followings: AMH (zeige AMH Proteine) mRNA levels decrease in both dominant and secondary follicles during follicular deviation; granulosa cells from heathy follicles express more AMH (zeige AMH Proteine) mRNA compared to subordinate follicles undergoing atresia and FSH (zeige BRD2 Proteine) stimulates AMH (zeige AMH Proteine) and AMHR2 mRNA expression in granulosa cells of co-dominant follicles.
AMH (zeige AMH Proteine), co-expressed with AMHR2, was produced heterogeneously by preantral follicles in macaques with levels correlated positively with follicle growth and oocyte maturation. AMH (zeige AMH Proteine) may serve as a biomarker for primate follicular development in vitro.
Sertoli cell maturation during puberty in the stallion was accompanied by a reduced expression of anti-Mullerian hormone (zeige AMH Proteine) and its receptor, arrest of cell proliferation, increased expression of androgen receptor (zeige AR Proteine)
In granulosa cells from women with PCOS, the regulation of AMH (zeige AMH Proteine) and AMHR2 expression is altered in a way that promotes the overexpression of the AMH (zeige AMH Proteine)/AMHR2 system, and could contribute to the follicular arrest observed in these patients.
genetic variants of AMH (zeige AMH Proteine) or AMHR2 were not found to be associated with a higher risk for polycystic ovaries syndrome.
AMHRII 1749C > T and -482A > G genetic variants are associated with the ovarian response to standard gonadotropin stimulation, affecting mainly the follicular growth in IVF (zeige SCN5A Proteine).
Genotyping of the AMH (zeige AMH Proteine) c.146G>T and AMHR2 -482A>G polymorphisms does not provide additional useful information as a predictor of ovarian reserve or ovarian response and treatment outcomes.
The aim of this study was to investigate the density and distribution of single nucleotide polymorphisms (SNPs) anti-Mullerian hormone (AMH (zeige AMH Proteine)) and AMHRII receptors in cryptorchid patients.
a result of VEGF (zeige VEGFA Proteine) misregulation, AMHR2 overexpression increases AMH (zeige AMH Proteine) binding, which may attenuate follicular or oocyte maturation.
Neither AMH (zeige AMH Proteine) nor AMHR2 polymorphisms were related to age, BMI, hormone levels or ovarian parameters in the follicular phase in women of late reproductive stage.
There is no association between single nucleotide polymorphisms in the AMH/AMHR2 signaling pathway and early ovarian hyperstimulation syndrome in Han Chinese women
A significant subset of GnRH neurons express the AMH receptor.
A significant portion of AMHRII was missing most of its extracellular domain (ECD (zeige SHFM1 Proteine)) and was unfolded and retained in the endoplasmic reticulum.
lncRNA-Amhr2 plays a role in Amhr2 gene activation in ovarian granulosa cells by enhancing promoter activity, providing insight into Amhr2 gene regulation underlying the AMH (zeige AMH Proteine) signaling in the female reproductive system.
A significant subset of GnRH neurons express the AMH receptor and AMH potently activates the GnRH neuron firing.
physiological function(s) of type 2 anti-Mullerian hormone (zeige AMH Proteine) receptor (zeige NR4A1 Proteine)
Wt1 (zeige WT1 Proteine) is an activator of Amhr2.
This gene encodes the receptor for the anti-Mullerian hormone (AMH) which, in addition to testosterone, results in male sex differentiation. AMH and testosterone are produced in the testes by different cells and have different effects. Testosterone promotes the development of male genitalia while the binding of AMH to the encoded receptor prevents the development of the mullerian ducts into uterus and Fallopian tubes. Mutations in this gene are associated with persistent Mullerian duct syndrome type II. Alternatively spliced transcript variants encoding different isoforms have been identified.
anti-Muellerian hormone type-2 receptor
, Anti-Muellerian hormone type-2 receptor
, anti-Mullerian hormone receptor, type II
, anti-Muellerian hormone type-2 receptor-like
, AMH type II receptor
, MIS type II receptor
, Muellerian inhibiting substance type II receptor
, Mullerian inhibiting substance type II receptor
, anti-Muellerian hormone type II receptor
, anti-Mullerian hormone type 2 receptor