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anti-Human WNT9B Antikörper:
anti-Rat (Rattus) WNT9B Antikörper:
anti-Mouse (Murine) WNT9B Antikörper:
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Human Polyclonal WNT9B Primary Antibody für ELISA, WB - ABIN4366209
Chiquet, Blanton, Burt, Ma, Stal, Mulliken, Hecht: Variation in WNT genes is associated with non-syndromic cleft lip with or without cleft palate. in Human molecular genetics 2008
Our study further supports the involvement of WNT9B as a cleft susceptibility gene in Brazilian families experiencing NSCL (zeige NHLH1 Antikörper)+/-P. Although additional studies are still necessary to unveil the exact mechanism by which WNT (zeige WNT2 Antikörper) genes would contribute to NSCL (zeige NHLH1 Antikörper)+/-P, allelic polymorphisms in these genes and their interactions may partly explain the variance of individual susceptibility to NSCL (zeige NHLH1 Antikörper)+/-P.
Multi-layered mutation in hedgehog (zeige SHH Antikörper)-related genes in Gorlin syndrome may affect the phenotype
This association study successfully identified two susceptibility Single Nucleotide Polymorphisms, (WNT9B and PBX1 (zeige PBX1 Antikörper)) associated with Mayer-Rokitansky-Kuster-Hauser syndrome risk, both separately and interactively.
Genome association study shows a highly conserved 32 kb intergenic region containing regulatory elements between WNT3 (zeige WNT3 Antikörper) and WNT9B in patients with classic bladder exstrophy.
results indicate that mutations in the coding sequence of WNT9B are not responsible for Mullerian duct abnormalities in the Chinese population
Two novel mutations (a missense mutation in exon 1, and one in the 3-UTR (zeige UTS2R Antikörper)) may be pathogenic variants in Mayer-Rokitansky-Kuster-Hauser syndrome patients and warrant further functional study.
secretion of WNT2B (zeige WNT2B Antikörper) and WNT9B and stabilization of beta-catenin (CTNNB1 (zeige CTNNB1 Antikörper)) upon virus infection negatively regulate expression of representative inducible genes IFNB1 (zeige IFNB1 Antikörper), IFIT1 (zeige IFIT1 Antikörper) and TNF (zeige TNF Antikörper) in a CTNNB1 (zeige CTNNB1 Antikörper)-dependent effector mechanism
The signals from the stromal fibroblasts cooperate with Wnt9b to promote differentiation of the progenitor cells.
Mutations in the coding sequence of WNT4 (zeige WNT4 Antikörper), WNT5A (zeige WNT5A Antikörper), WNT7A (zeige WNT7A Antikörper), and WNT9B are not responsible for the Mayer-Rokitansky-Kuster-Hauser syndrome.
These analyses suggest that in zebrafish, as in humans, wnt9b plays distinct roles in directing morphogenetic movements of developing branchial arch elements.
Data indicate that mutations in wingless-type MMTV integration site family, member 9B protein (WNT9B) were frequently associated with Mayer-Rokitansky-Kuster-Hauser syndrome (MRKHS).
Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain.
Expression of Wnt9b in Six2 (zeige SIX2 Antikörper)-positive cells disrupts cell fate decisions in the kidney and the gastrointestinal tract.
Epithelial Wnt7b (zeige WNT7B Antikörper) and Wnt9b as possible ligands of Fzd1 (zeige Fzd1 Antikörper)-mediated beta-catenin (Ctnnb1 (zeige CTNNB1 Antikörper))-dependent (canonical) Wnt (zeige WNT2 Antikörper) signaling in the undifferentiated ureteric mesenchyme
Study has identified a previously unknown regulatory link between WNT9B and FGF signaling during lip and upper jaw development.
Notch (zeige NOTCH1 Antikörper) pathway activation can replace the requirement for Wnt4 (zeige WNT4 Antikörper) and Wnt9b in mesenchymal-to-epithelial transition of nephron stem cells
wnt9b signaling regulates stem cell proliferation and differention in the developing kidney depending on the activity of the transcription factor Six2 (zeige SIX2 Antikörper) in the responding cell.
transgenic overexpression of Wnt9b in the ureteric bud causes reduced branching in multiple founder lines.
expression in most but not all tissues of the 16.5-day embryo; no significant elevation of Wnt9b expression was detected in 29 mouse mammary tumor virus-induced tumors
Wnt9b is a common organizing signal regulating diverse components of the mammalian urogenital system.
Ligand for members of the frizzled family of seven transmembrane receptors. Probable developmental protein. May be a signaling molecule which affects the development of discrete regions of tissues. Is likely to signal over only few cell diameters (By similarity).
, protein Wnt-9b
, wingless-type MMTV integration site family, member 15
, wnt9b protein
, wingless related MMTV integration site 9B
, wingless-type MMTV integration site 9B
, wingless-type MMTV integration site family, member 9B
, wingless-type MMTV integration site 15