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Human Monoclonal CCL13 Primary Antibody für CyTOF, ELISA (Capture) - ABIN4899862
Gounni Abdelilah, Wellemans, Agouli, Guenounou, Hamid, Beck, Lamkhioued: Increased expression of Th2-associated chemokines in bullous pemphigoid disease. Role of eosinophils in the production and release of these chemokines. in Clinical immunology (Orlando, Fla.) 2006
In plasma, but not CSF (zeige CSF2 Antikörper), the bivariate MCP4 (CCL13)/ MCP1(CCL2 (zeige CCL2 Antikörper)) ratio is ca. twofold elevated in PTSD patients compared with healthy controls.
CCL13 expression is significantly upregulated in human masticatory mucosa during wound healing
The reduction of circulating levels of MCP-4, eotaxin-3 (zeige CCL26 Antikörper), and MIP-1beta (zeige CCL4 Antikörper) could be one of the mechanisms by which bariatric surgery contributes to the reduction of cardiovascular risk in these patients.
CCL13 levels in serum and synovial fluid may serve as a biomarker for the progression of osteoarthritis.
identified 13 ADCC-activated genes. Six gene expression assays including 8 of the 13 genes (CCL3 (zeige CCL3 Antikörper), CCL4/CCL4L1 (zeige CCL4 Antikörper)/CCL4L2, CD160 (zeige CD160 Antikörper), IFNG (zeige IFNG Antikörper), NR4A3 (zeige NR4A3 Antikörper) and XCL1 (zeige XCL1 Antikörper)/XCL2 (zeige XCL2 Antikörper)) were analyzed in 127 kidney biopsies
CCL13 is an antimicrobial protein with bacteriocidal activity against E. coli.
Data suggest that CCL13 binds to several chemokine (zeige CCL1 Antikörper) receptors (CCR1, CCR2 (zeige CCR2 Antikörper), and CCR3 (zeige CCR3 Antikörper)), allowing CCL13 to elicit different effects on target cells of immune system. CCL13 is involved in pathology of chronic inflammatory/autoimmune diseases. [REVIEW]
MCP-4 and hsCRP may be the markers linking chronic inflammation in obesity and periodontal disease.
E(2) has adverse effects on the pathogenesis of RA as a result of unregulated cell death, increased TNF-alpha (zeige TNF Antikörper)-induced MMP-3 (zeige MMP3 Antikörper) production, and CCL13 overproduction, subsequently resulting in the disease progression of RA.
Data show that, for the small macrophages in COPD (zeige ARCN1 Antikörper), increased transcript and protein levels for CCL2 (zeige CCL2 Antikörper), CCL7 (zeige CCL7 Antikörper), CCL13 and CCL22 (zeige CCL22 Antikörper) with a more than 100-fold increase for CCL13 mRNA.
deletion of the gene encoding MMCP-4 (Mcpt4), markedly reduced late, but not early, infarct size by suppressing IGF-1 (zeige IGF1 Antikörper) degradation and, consequently, diminished cardiac dysfunction and adverse structural remodeling
Data indicate that a second-degree burn injury can initiate an immediate novel zonal degranulation of mast cell throughout all skin layers and a disruption of the epidermal tight junctions dependent on the nonredundant presence of mMCP4 and mMCP5 (zeige CMA1 Antikörper).
these results support the conclusion that mast cells can contribute to the initial lung injury induced by bleomycin through release of the MCPT4 chymase (zeige CMA1 Antikörper).
Mast cell chymase (zeige CMA1 Antikörper) degrades the alarmins heat shock protein 70 (zeige HSP70 Antikörper), biglycan (zeige BGN Antikörper), HMGB1 (zeige HMGB1 Antikörper), and interleukin-33 (IL-33 (zeige IL33 Antikörper)) and limits danger-induced inflammation.
Data from Mcp-4 (mast cell protease 4) knockout mice suggest Mcp-4 plays a pivotal role in the dynamic (in vivo and in vitro) conversion of systemic Big-endothelin-1 (zeige EDN1 Antikörper) to endothelin-1 (zeige EDN1 Antikörper) (1-31).
MCs (zeige SMCP Antikörper) exert protective functions after trauma, at least in part via mMCP-4, by suppressing exacerbated inflammation via their proteases.
The effects of interactions between mMCP-4 and TNF (zeige TNF Antikörper) in vivo by analyzing the features of a classic model of polymicrobial sepsis, were assessed.
mMCP-4 plays two different roles in the pathogenesis of experimental BP, by both activating MMP-9 (zeige MMP9 Antikörper) and by cleaving BP180 (zeige COL17A1 Antikörper), leading to injury of the hemidesmosomes and extracellular matrix of the basement membrane zone.
mMCP-4-deficient mice but not to mMCP-5 (zeige CMA1 Antikörper)-deficient mice revealing nonredundant actions for these two MC proteases in a model of innate inflammatory injury with remodeling.
MCP-4 contributes locally to the aggravation of glomerulonephritis by mediating a variety of proinflammatory effects.
This gene is one of several Cys-Cys (CC) cytokine genes clustered on the q-arm of chromosome 17. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene displays chemotactic activity for monocytes, lymphocytes, basophils and eosinophils, but not neutrophils. This chemokine plays a role in accumulation of leukocytes during inflammation. It may also be involved in the recruitment of monocytes into the arterial wall during artherosclerosis.
C-C motif chemokine 13
, monocyte chemoattractant protein 4
, monocyte chemotactic protein 4
, new CC chemokine 1
, small inducible cytokine subfamily A (Cys-Cys), member 13
, small-inducible cytokine A13
, monocyte chemotactic protein-4
, serosal mast cell protease