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Human Polyclonal BAX Primary Antibody für WB - ABIN5518641
Jiang, Li, Zhou, Wang, Zhang, Wang: Colistin-induced apoptosis in PC12 cells: involvement of the mitochondrial apoptotic and death receptor pathways. in International journal of molecular medicine 2014
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Human Polyclonal BAX Primary Antibody für FACS, IF (cc) - ABIN725390
Tian, Zhang, Wang, Dai, Shen, Yang, Huang: The protective effect of hyperbaric oxygen and Ginkgo biloba extract on A?25-35-induced oxidative stress and neuronal apoptosis in rats. in Behavioural brain research 2013
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Human Polyclonal BAX Primary Antibody für IF, ELISA - ABIN1531490
Berger, Quast, Plötz, Kammermeier, Eberle: Sensitization of melanoma cells for TRAIL-induced apoptosis by BMS-345541 correlates with altered phosphorylation and activation of Bax. in Cell death & disease 2013
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Human Polyclonal BAX Primary Antibody für IF (p), IHC (p) - ABIN682843
Pereda: [Histochemical study of the distribution of sialomucins in the oviducts and jelly coat of the oocytes of Rana pipiens. Behavior of the different layers of the coat in water]. in Journal of embryology and experimental morphology 1971
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Human Polyclonal BAX Primary Antibody für IP, WB - ABIN222887
Kim, Zhang, Hitomi, Lee, Mukherjee: Endoplasmic reticulum stress-induced caspase-4 activation mediates apoptosis and neurodegeneration in INCL. in Human molecular genetics 2006
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Human Polyclonal BAX Primary Antibody für ICC, ELISA - ABIN1001866
Lockshin, Osborne, Zakeri: Cell death in the third millennium. in Cell death and differentiation 2000
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Cow (Bovine) Monoclonal BAX Primary Antibody für ELISA, FACS - ABIN451743
Barreto-Vianna, Aguila, Mandarim-de-Lacerda: Effects of liraglutide in hypothalamic arcuate nucleus of obese mice. in Obesity (Silver Spring, Md.) 2016
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Gerbil Polyclonal BAX Primary Antibody für IHC, IHC (fro) - ABIN4283098
Krajewska, Rosenthal, Mikolajczyk, Stennicke, Wiesenthal, Mai, Naito, Salvesen, Reed, Fiskum, Krajewski: Early processing of Bid and caspase-6, -8, -10, -14 in the canine brain during cardiac arrest and resuscitation. in Experimental neurology 2004
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Human Polyclonal BAX Primary Antibody für FACS, IHC (p) - ABIN388094
Liu, Newland, Jia: Bax conformational change is a crucial step for PUMA-mediated apoptosis in human leukemia. in Biochemical and biophysical research communications 2003
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Mouse (Murine) Polyclonal BAX Primary Antibody für IHC, WB - ABIN3020683
He, Zhang, Wang, Zhong, Shao, Zhu, Shen: AURKA suppression induces DU145 apoptosis and sensitizes DU145 to docetaxel treatment. in American journal of translational research 2013
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Bid (zeige BID Antikörper) and Bax are signal transduction factors in granulosa cells and play proapoptotic roles.
The data demonstrate that severe hypocapnia results in increased Bax expression, DNA fragmentation, and membrane lipid peroxidation in mitochondria of cerebral cortical neurons of newborn piglets, and may result in apoptotic cell death.
BoHV-5 replication apparently modulates BCL-2 (zeige BCL2 Antikörper) expression and gene transcription, enhancing production of virus progeny, but does not increase Bax expression.
It was concluded that the Fas (zeige FAS Antikörper)-FasL (zeige FASL Antikörper) signaling pathway was involved in regulation of bovine oocyte apoptosis, perhaps related to B (zeige TDO2 Antikörper) cell lymphoma/leukemia-2 associated X upregulation.
apoptosis-inducing factor (zeige AIFM1 Antikörper) was expressed in luminal alveolar cells and, in concert with a change in bax protein to bcl-2 (zeige BCL2 Antikörper) protein ratio, might contribute to signalling of a change in the dynamic balance of the cell population as lactation progresses
mRNA expression of Bax, Bcl-2 (zeige BCL2 Antikörper), caspase-3 (zeige CASP3 Antikörper) and-7 cannot be used as a reliable apoptosis detection method.
The effect of culture conditions on expression of heat shock protein 70 (zeige HSP70 Antikörper) and Bax protein in bovine blastocysts is reported.
xlbax is a regulator of muscle fiber death in the regressing tail during metamorphosis.
These results suggest a role for mitochondrial p53 (zeige TP53 Antikörper) activity in promoting hair cell death due to aminoglycosides, likely upstream of Bax and Bcl2 (zeige BCL2 Antikörper).
helix alpha9 assists Bax activation via the dimer heterogeneity and interactions with specific MOM lipids, which eventually facilitate proteolipidic pore formation in apoptosis regulation
VDAC2 (zeige VDAC2 Antikörper) ensures mitochondria-specific membrane association of Bax and in the absence of VDAC2 (zeige VDAC2 Antikörper) Bax localizes towards other cell compartments. Bax retrotranslocation is also regulated by nucleotides and calcium ions, suggesting a potential role of the transport of these ions through VDAC2 (zeige VDAC2 Antikörper) in Bax retrotranslocation.
The results of the genes expression analysis revealed that indocyanine green-photodynamic therapy at concentrations 1000mug/mL, induced the significant expression of BAX in HGF (zeige HGF Antikörper) cells
Our observations point to misfolded Bax states, shedding light on the molecular mechanism of Bax mutation-elicited cancer. Most importantly, the structure of the Bax pore facilitates future study of releases cytochrome C (zeige CYCS Antikörper) in atomic detail
Data suggest that regulation of pancreatic beta-cell function and survival/apoptosis involves alternative splicing modulated by key splicing regulator (zeige PTBP2 Antikörper) SRP55 (zeige SRSF6 Antikörper); SRP55 (zeige SRSF6 Antikörper)-regulated alternative splicing includes modulation of function of pro-apoptotic proteins (BIM (zeige BCL2L11 Antikörper), BAX), JNK (zeige MAPK8 Antikörper) signaling, and endoplasmic reticulum stress. (SRP55 (zeige SRSF6 Antikörper) = pre-mRNA-splicing factor SRP55 (zeige SRSF6 Antikörper); BIM (zeige BCL2L11 Antikörper) = BCL-2 interacting protein BIM (zeige BCL2L11 Antikörper))
High expression of BAX is associated with colorectal cancer.
High mitochondrial Bax apoptosis regulator protein (BAX) levels correlate with improved acute myeloid leukemia (zeige BCL11A Antikörper) (AML (zeige RUNX1 Antikörper)) patient survival.
parkin (zeige PARK2 Antikörper)-dependent targeting of misregulated BAX on the mitochondria provides substantial protection against BAX apoptotic activity.
our data present preliminary evidence that inherited abnormalities in the intrinsic apoptosis pathway, related to BAX G(-248)A and BCL2 (zeige BCL2 Antikörper) C(-717)A SNPs, are associated with treatment response and act as independent prognostic factors in DLBCL.
SFRP5 (zeige SFRP5 Antikörper) confers protection against oxidative stress-induced (zeige SQSTM1 Antikörper) apoptosis through inhibition of beta-catenin (zeige CTNNB1 Antikörper) activation and downregulation of Bax.
Here the authors show that mouse embryonic fibroblasts deficient in Bax/Bak1 (zeige BAK1 Antikörper) are resistant to the third major form of cell death associated with autophagy through a mechanism involving lysosome permeability.
although all CR subtypes undergo cell death, septum, but not hem, CRs (zeige CARS Antikörper) die in a Bax-dependent manner. Bax-inactivated rescued septum-CRs (zeige CARS Antikörper) maintain immature electrophysiological properties
An autoinhibited dimeric form of BAX regulates the cytosolic BAX activation pathway.
Postnatal synaptic rearrangement needed for acquisition of skilled behaviors requires the activity-dependent, non-apoptotic Bax/Bak (zeige BAK1 Antikörper)-caspase (zeige CASP3 Antikörper) signaling cascade. Adult Bax/Bak (zeige BAK1 Antikörper) mutant mice exhibit aberrant co-activation of antagonistic muscle pairs and skilled grasping deficits but normal reaching and retrieval behaviors.
Study found that emphysema occurred in ku70(-/-) mice at the age of three-months old, and that Bax deficiency was able to suppress it. These results suggest that Bax-mediated apoptosis induces emphysema in ku70(-/-) mice.
The polyglutamine embedded in the ER membrane was observed at the same time as Bax insertion. These results demonstrated that the ER membrane may be a target of polyglutamine, which triggers cell death through Bax.
These results suggest that Bax mediates beta-cell apoptosis in Pdx1 (zeige PDX1 Antikörper)-deficient diabetes.
Bim (zeige BCL2L11 Antikörper) and Puma (zeige BBC3 Antikörper) overlapped apoptosis only partially during physiological apoptotic stage and they were present in non-apoptotic parts of the follicles.
This study demonstated that Bax KO mice show Hyperactivity and depression.
The influence of chronic ethanol consumption on the expression of the Bdnf (zeige BDNF Antikörper), Bax, Bcl-xL (zeige BCL2L1 Antikörper), and CASP3 (zeige CASP3 Antikörper) genes was studied in the brain structures of B6-1473C (C/C) and B6-1473G (G/G) mice that had been obtained on the base of the C57BL/6 strain.
The content of Bax protein in the cardiomyocyte cytoplasm decreased, thus indicating that the mitochondrial pathway was not involved in the realization of the apoptotic program in a model of ventricular overload.
These results indicate that RI-PostC can ameliorate myocardial ischemia-reperfusion injury and increase the Bcl-2 (zeige BCL2 Antikörper)/Bax ratio through a mechanism involving protein kinase C (zeige PKC Antikörper).
Cinobufagin can remarkably inhibit the proliferation and induce the apoptosis of lens epithelial cells by increasing expression of bax and decreasing expression of bcl2 (zeige BCL2 Antikörper).
Elevated local temperature of the testis buried in the inguinal pocket increases the apoptosis of spermatogenic cells, and the spermatogenic cell apoptosis is highly correlated with the decreased expression of Bcl-2 (zeige BCL2 Antikörper) and increased expression of Bax.
In this study evidence is provided that exogenous PGF2alpha differentially modulates luteal expression of BCL2-associated X protein (BAX) transcripts and protein depending on luteal stage.
bcl-2 (zeige BCL2 Antikörper) and bax were expressed strongly in denervated guinea-pig facial muscle. [bcl-2 (zeige BCL2 Antikörper); bax]
goat oocytes based on G6PDH (zeige G6PD Antikörper)-activity through the BCB test improves their developmental competence, increases intracellular GSH content, and affects the expression of the apoptosis-related genes9 Bax and Bcl-2 (zeige BCL2 Antikörper) ).
The protein encoded by this gene belongs to the BCL2 protein family. BCL2 family members form hetero- or homodimers and act as anti- or pro-apoptotic regulators that are involved in a wide variety of cellular activities. This protein forms a heterodimer with BCL2, and functions as an apoptotic activator. This protein is reported to interact with, and increase the opening of, the mitochondrial voltage-dependent anion channel (VDAC), which leads to the loss in membrane potential and the release of cytochrome c. The expression of this gene is regulated by the tumor suppressor P53 and has been shown to be involved in P53-mediated apoptosis. Multiple alternatively spliced transcript variants, which encode different isoforms, have been reported for this gene.
BCL2-associated X protein
, BCL2-associated protein
, apoptosis regulator BAX
, bax zeta
, BCL2-associated X protein omega
, BCL2-associated X protein transcript variant delta2
, bcl-2-like protein 4