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anti-Human NDUFS3 Antikörper:
anti-Mouse (Murine) NDUFS3 Antikörper:
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Human Monoclonal NDUFS3 Primary Antibody für IHC (p), IP - ABIN561943
Cheng, Kuo, Fan, Fang, Jiang, Lo, Wang, Kao, Lee: Overexpression of Lon contributes to survival and aggressive phenotype of cancer cells through mitochondrial complex I-mediated generation of reactive oxygen species. in Cell death & disease 2013
Human Polyclonal NDUFS3 Primary Antibody für ELISA, WB - ABIN542450
Martinvalet, Dykxhoorn, Ferrini, Lieberman: Granzyme A cleaves a mitochondrial complex I protein to initiate caspase-independent cell death. in Cell 2008
We observed loss of NDUFB8 immunoreactivity in all patients with mutations affecting nuclear-encoding structural subunits and assembly factors, whilst only 3 of the 10 patients with mutations affecting mtDNA-encoded structural subunits showed loss of NDUFB8, confirmed by BN-PAGE analysis of CI assembly and IHC using an alternative, commercially-available CI (NDUFS3) antibody.
identified a novel Leigh syndrome causing NDUFS3 mutation and expanded the clinical spectrum caused by NDUFS3 mutations in this study.
The results suggest that the impaired mitochondrial activity could be due to the broken interaction between DJ-1 and NDUFS3 and that downregulation of DJ-1 in sperm and testes contributes to AS pathogenesis.
The NDUFS3 expressed in the temporal cortex in patient with late-onset Alzheimer's disease.
High NDUFS3 expression is associated with IgA nephropathy.
pH stability of w-t and Leigh syndrome mutant varied at extreme acidic pH. the molten globule like structure of w-t at pH1 was absent in case of the mutant protein. Both the w-t and mutant proteins showed multi-step thermal and Gdn-HCl induced unfolding
NDUFS3 is down-regulated in serous ovarian adenocarcinoma
Expressions of GRIM-19, NDUFS3, and extracellular matrix elements are correlated with invasive capabilities of breast cancer cell lines.
a novel, biomarker potential of a mitochondrial complex I subunit protein, NDUFS3 - as a robust indicator of breast cancer progression and invasiveness as well as of hypoxia/necrosis in clinical specimens of invasive ductal carcinoma, was uncovered.
Decreased protein levels of complex I 30-kDa subunit in fetal Down syndrome brains.
This protein has been found differentially expressed in the temporal lobe from patients with schizophrenia.
subunits of complex I and two subcomplexes (Ialpha and Ibeta) were resolved on one- and two-dimensional gels and by reverse-phase high performance liquid chromatography
This gene encodes one of the iron-sulfur protein (IP) components of mitochondrial NADH:ubiquinone oxidoreductase (complex I). Mutations in this gene are associated with Leigh syndrome resulting from mitochondrial complex I deficiency.
, NADH dehydrogenase [ubiquinone] iron-sulfur protein 3, mitochondrial
, NADH dehydrogenase-ubiquinone 30 kDa subunit
, NADH-ubiquinone oxidoreductase 30 kDa subunit
, complex I 30kDa subunit
, complex I-30kD
, NADH dehydrogenase (ubiquinone) Fe-S protein 3
, mitochondrial complex I subunit NDUFS3
, NADH dehydrogenase (ubiquinone) Fe-S protein 3, 30kDa (NADH-coenzyme Q reductase)
, OXPHOS complex I 30 kDa subunit