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IL-8 ELISA Kit

IL8 Reaktivität: Human Colorimetric Sandwich ELISA 10-1600 pg/mL
Produktnummer ABIN577068
  • Target Alle IL-8 (IL8) ELISA Kits anzeigen
    IL-8 (IL8) (Interleukin 8 (IL8))
    Reaktivität
    • 15
    • 5
    • 4
    • 4
    • 3
    • 3
    • 3
    • 2
    • 2
    • 2
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    Human
    Nachweismethode
    Colorimetric
    Methodentyp
    Sandwich ELISA
    Detektionsbereich
    10-1600 pg/mL
    Untere Nachweisgrenze
    10 pg/mL
    Applikation
    ELISA
    Verwendungszweck
    For the Quantitative Determination of Human Interleukin-8 (IL-8) Concentrations in Serum, Plasma, Cell Culture Supernatant, and Other Biological Fluids
    Analytische Methode
    Quantitative
    Sensitivität
    < 10 pg/mL
    Bestandteile
    Standards: 1 set/2 vials
    Featured
    Zu unserem meistverkauften IL8 ELISA Kit
    Top Product
    Discover our top product IL8 ELISA Kit
  • Plattentyp
    Pre-coated
    Beschränkungen
    Nur für Forschungszwecke einsetzbar
  • Konservierungsmittel
    Without preservative
  • Target Alle IL-8 (IL8) ELISA Kits anzeigen
    IL-8 (IL8) (Interleukin 8 (IL8))
    Andere Bezeichnung
    Interleukin-8 (IL-8) (IL8 Produkte)
    Synonyme
    CXCL8 ELISA Kit, GCP-1 ELISA Kit, GCP1 ELISA Kit, LECT ELISA Kit, LUCT ELISA Kit, LYNAP ELISA Kit, MDNCF ELISA Kit, MONAP ELISA Kit, NAF ELISA Kit, NAP-1 ELISA Kit, NAP1 ELISA Kit, IL-8 ELISA Kit, AMCF-I ELISA Kit, il8 ELISA Kit, IL8 ELISA Kit, si:dkey-151b16.2 ELISA Kit, cxcli2 ELISA Kit, cxcl8 ELISA Kit, gcp-1 ELISA Kit, il-8 ELISA Kit, lynap ELISA Kit, mdncf ELISA Kit, monap ELISA Kit, nap-1 ELISA Kit, C-X-C motif chemokine ligand 8 ELISA Kit, interleukin 8 ELISA Kit, chemokine (C-X-C motif) ligand 8a ELISA Kit, chemokine (C-X-C motif) ligand 1-like ELISA Kit, chemokine (C-X-C motif) ligand 8 L homeolog ELISA Kit, CXCL8 ELISA Kit, il8 ELISA Kit, cxcl8a ELISA Kit, IL8 ELISA Kit, Cxcl8 ELISA Kit, LOC422654 ELISA Kit, cxcl8.L ELISA Kit
    Hintergrund
    Hepatitis resulting from infection with viruses other than Hepatitis A Virus (HAV) and Hepatitis B (HBV) virus was previously referred to as non-A, non-B hepatitis. The first characterised non-A, non-B hepatitis agent was that responsible for parentally transmitted non-A, non-B hepatitis, or what is now called Hepatitis C Virus. This was followed by the cloning of a portion of the fecal-orally-transmitted agent, the Hepatitis E Virus (HEV). Hepatitis E Virus has been referred to as enterically transmitted non-A, non-B hepatitis. Epidemics of enterically transmitted Hepatitis E Virus have been recognised worldwide but occur principally in developing countries. They have been reported in Southeast Asia, central Asia, Africa, Mexico, and Central America. In these areas, contaminated water has been implicated as the principal vehicle of virus transmission. Although HEV and HAV are transmitted in a similar manner, there are major differences in the clinical, pathological, and epidemiological courses of these two viruses. In particular, the mortality rate for HEV infection is 1 to 2%, or approximately 1-fold greater than that seen for HAV. Infection with HEV is particularly fatal for pregnant women, for whom the mortality rate can be as high as 1 to 2%. This HEV IgM Antibody ELISA is an immunoassay, which employs synthetic and recombinant HEV antigens for the detection of IgM antibody to HEV in human serum or plasma. These antigens, which correspond to the structure regions of HEV, constitute the solid phase antigenic adsorbent. Samples with O.D. values greater than or equal to the Cut-off value are defined as initially reactive. Initially reactive specimens are to be re-tested in duplicate. Samples, which do not react in either of the duplicate, repeat tests are considered non-reactive for IgM antibodies to HEV. Samples, which are reactive in either of the duplicates tests, are considered repeatably reactive. Rev. (3/4) HEV IgM
    Pathways
    TLR Signalweg, Cellular Response to Molecule of Bacterial Origin, Regulation of G-Protein Coupled Receptor Protein Signaling, ER-Nucleus Signaling, Hepatitis C, Autophagie
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