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anti-Human FSHR Antikörper:
anti-Mouse (Murine) FSHR Antikörper:
anti-Rat (Rattus) FSHR Antikörper:
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Human Polyclonal FSHR Primary Antibody für IF (p), IHC (p) - ABIN729508
Luo, Yin, Zhang, Yuan, Zhao, Luan, Zhang: Role of SDF-1/CXCR4 and cytokines in the development of ovary injury in chemotherapy drug induced premature ovarian failure mice. in Life sciences 2017
Show all 2 Pubmed References
Human Polyclonal FSHR Primary Antibody für ELISA, WB - ABIN560952
Ritter, Thuering, Saint Mezard, Luong-Nguyen, Seltenmeyer, Junker, Fournier, Susa, Morvan: Follicle-stimulating hormone does not impact male bone mass in vivo or human male osteoclasts in vitro. in Calcified tissue international 2008
Human Polyclonal FSHR Primary Antibody für WB - ABIN4909704
Wang, Wang, Zhu, Zhang, Sheng, Zhang, Han, Yuan, Weng: Seasonal expression of luteinizing hormone receptor and follicle stimulating hormone receptor in testes of the wild ground squirrels (Citellus dauricus Brandt). in Acta histochemica 2018
INVESTIGATION OF STAT5A (zeige STAT5A Antikörper), FSHR AND LHR (zeige LHCGR Antikörper) GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
The expression of FSHR mRNA in granulosa cells was highest in small antral follicles, then decreased significantly as follicles increased in size, and was lowest in cysts.
transfer. We conclude that variation at these loci of the FSHR gene has no significant effect on pregnancy rates in Luxi cattle.
Specific alleles of the bovine FSHR gene are associated with variations in embryo yield and in the number of unfertilised oocytes.
This study evaluated the relationships among aromatase (zeige CYP19A1 Antikörper), IGF-1 (zeige IGF1 Antikörper), IGF2R (zeige IGF2R Antikörper), and follicle stimulating hormone (FSH) receptor levels expressed in ovarian follicles of cattle selected for twin pregnancies.
granulosa cell clustering is accompanied by marked increases in FSHr, IGF-1r (zeige IGF1R Antikörper), and p450 arom (zeige CYP19A1 Antikörper) expression, and precedes induction and subsequent peak E2 production
Dominant follicles experience a reduction in FSH (zeige BRD2 Antikörper) dependence (diminished expression of FSHR), but acquire increased LH dependence (enhanced expression of LHCGR (zeige LHCGR Antikörper)) as they grow during the low FSH (zeige BRD2 Antikörper) milieu of follicular waves.
FSHR is specifically regulated through androgen receptor (zeige AR Antikörper) in granulosa cells
Heterozygous heifers showed a higher pregnancy rate (67 and 66% for LHR (zeige LHCGR Antikörper) and FSHR genes, respectively), but no significant effects were observed for the genes studied (
Data show that double mutation of follicle-stimulating hormone receptor (fshr) and luteinizing hormone receptor (lhcgr (zeige LHCGR Antikörper)) resulted in infertile males.
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr and lhcgr (zeige LHCGR Antikörper).
Characterization of the first functional zebrafish (Danio rerio) gonadotropic hormone I receptor (follicle stimulating hormone receptor).
T cells redirected against FSHR(+) tumor cells with full-length FSH (zeige BRD2 Antikörper) represent a promising therapeutic alternative against a broad range of ovarian malignancies, with negligible toxicity even in the presence of cognate targets in tumor-free ovaries
Findings of this study suggest a significant association between FSHR gene p. Thr307Ala or p. Asn680Ser coding sequence change and PCOS. The variant homozygote genotype results in a higher risk of PCOS.
The evaluation of sperm DNA fragmentation as a surrogate marker of sperm quality, and of the FSHR SNP rs6166 (p.N680S), might be useful to predict the response to FSH (zeige BRD2 Antikörper) treatment in men with idiopathic infertility
Mouse chondrocytes and human articular cartilage express functional FSHR. Moreover, FSH (zeige BRD2 Antikörper) can act on chondrocytes and cause genetic changes.
The mutation p.R59X in FSHR is causative for primary ovarian insufficiency by means of arresting folliculogenesis.
two mutations, V(221)G and T(449)N, in the extracellular domain and transmembrane helix 3, of FSHR, respectively, are reported.
The reduced fertilisation and pregnancy rate was associated with a lower LH receptor (zeige LHCGR Antikörper) density and a lack of essential down-regulation of the FSH (zeige BRD2 Antikörper) and LH receptor (zeige LHCGR Antikörper).
This work demonstrates that the expression of FSHR and LHCGR (zeige LHCGR Antikörper) can be induced in hGL5 cells but that the FSHR-dependent cAMP/PKA pathway is constitutively silenced, possibly to protect cells from FSHR-cAMP-PKA-induced apoptosis.
The incidence of the Ser/Ser (zeige SIGLEC1 Antikörper) genotype was higher in patients with higher recombinant human follicle-stimulating hormone consumption. Based on our results, we hypothesize an association between the follicle-stimulating hormone receptor polymorphisms and a "hyporesponse" to exogenous follicle-stimulating hormone.
The data suggest novel follicle-stimulating hormone receptor expression in endometriotic lesions, qualitatively and quantitatively different from that of normal endometrium.
Study demonstrates expression of follicle-stimulating hormone receptor (FSHR) and a direct action of follicle-stimulating hormone on testicular stem/germ cells possibly mediated via alternatively spliced growth factor type 1 receptor FSHR3 in mice.
FSHR and LHR (zeige LHCGR Antikörper) proteins are significantly upregulated in CCs (zeige CCS Antikörper) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (zeige ESR1 Antikörper), LHR (zeige LHCGR Antikörper), and FSHR
Brca1 (zeige BRCA1 Antikörper)(GC-/-) models reveal that specific intra-follicular Brca1 (zeige BRCA1 Antikörper) loss alone, or combined with cancer-promoting genetic (Trp53 (zeige TP53 Antikörper) loss) and endocrine (high serum follicle-stimulating hormone) changes, was not sufficient to cause ovarian tumors.
Data (including date from knockout mice) suggest that Fshr is expressed early in pregnany in placenta and other extragonadal tissues of fetoplacental unit; expression is particularly strong at term.
Sertoli cell-specific expression of MTA2 (zeige MTA2 Antikörper) is required for transcriptional regulation of FSHR gene during spermatogenesis.
By day 20 and in adult animals total AR or FSHR ablation significantly reduced Leydig cell numbers but Sertoli cell specific AR ablation had no effect.
The results demonstrate that gain-of-function mutations of the FSHR in mice bring about distinct and clear changes in ovarian function
haploinsufficiency of the follicle-stimulating hormone receptor accelerates oocyte loss inducing early reproductive senescence and biological aging in mice
The amount of dopamine d1 receptor (zeige DRD1 Antikörper), dopamine D2 receptor (zeige DRD2 Antikörper), and follicle stimulating hormone receptor (FSHr) mRNA were quantified in ovarian tissues in anestrous and mares expressing estrus during the breeding season are reported.
Activated TGF-beta (zeige TGFB1 Antikörper) signaling rescued miR (zeige MYLIP Antikörper)-143-reduced FSHR and intracellular signaling molecules, and miR (zeige MYLIP Antikörper)-143-induced porcine granulosa cell apoptosis.
The results showed that polymorphisms in exon 10 of the FSHR gene had a significant effect on litter size traits of Wannan Black and Berkshire pigs. These results can be applied for marker-assisted selection in the 2 swine breeds
These results showed that an increased FSHR gene expression level was accompanied with an increase in histone H3K9 acetylation levels, suggesting that histone H3K9 acetylation could regulate the expression of the porcine FSHR gene.
248 F(2) animals from a Duroc and Meishan cross were genotyped for three FSHR SNPs at positions 74, 532 and 1166, and these were correlated with the phenotypes of litter size and corpus luteum number
findings suggest the FSH receptor may be involved in the early follicle formation in pigs, which begins during prenatal life.
FSHR is expressed in the hamster ovary starting from the fetal life to account for FSH (zeige BRD2 Antikörper)-induced primordial follicle formation and cyclic AMP (zeige TMPRSS5 Antikörper) production.
The protein encoded by this gene belongs to family 1 of G-protein coupled receptors. It is the receptor for follicle stimulating hormone and functions in gonad development. Mutations in this gene cause ovarian dysgenesis type 1, and also ovarian hyperstimulation syndrome. Alternative splicing results in multiple transcript variants.
follicle stimulating hormone receptor
, follicle-stimulating hormone receptor
, gonadotropic hormone I
, follicle-stimulating hormone receptor-like
, FSH receptor
, follitropin receptor